2008 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary scientific interests are in Cell biology, Receptor, G protein, Molecular biology and Biochemistry. His work on Cell biology deals in particular with G protein-coupled receptor, Frizzled, Cell signaling, G protein-coupled receptor kinase and Protein kinase A. His Receptor study combines topics in areas such as Endocrinology and Effector.
His biological study spans a wide range of topics, including Alpha, Adenylyl cyclase and Cellular differentiation. The Hamster research Craig C. Malbon does as part of his general Molecular biology study is frequently linked to other disciplines of science, such as Solution hybridization, therefore creating a link between diverse domains of science. His research in Heterotrimeric G protein intersects with topics in Wnt signaling pathway, GTPase-activating protein and Intracellular.
His primary areas of investigation include Cell biology, Receptor, Molecular biology, Biochemistry and Internal medicine. His study in Cell biology concentrates on G protein-coupled receptor, Signal transduction, Wnt signaling pathway, Frizzled and G protein. His studies in G protein integrate themes in fields like Alpha and Cellular differentiation.
He has researched Receptor in several fields, including Membrane and Effector. As a member of one scientific family, Craig C. Malbon mostly works in the field of Molecular biology, focusing on Antisense RNA and, on occasion, Sense. His studies link Endocrinology with Internal medicine.
His main research concerns Cell biology, Wnt signaling pathway, Dishevelled, Frizzled and G protein-coupled receptor. Cell signaling, Signal transduction, Heterotrimeric G protein, Protein kinase A and Phosphorylation are among the areas of Cell biology where he concentrates his study. His study brings together the fields of Molecular biology and Wnt signaling pathway.
Craig C. Malbon interconnects Methylation, AXIN1 and MAPK/ERK pathway in the investigation of issues within Frizzled. His G protein-coupled receptor study is focused on Receptor and Biochemistry. His study in Receptor is interdisciplinary in nature, drawing from both Kinase and Intracellular.
Craig C. Malbon mainly focuses on Cell biology, Frizzled, Wnt signaling pathway, Signal transduction and Dishevelled. His studies in Cell biology integrate themes in fields like Receptor and Receptor recycling. His Receptor research includes themes of Intracellular and Effector.
His Dishevelled research integrates issues from MAPK/ERK pathway, LRP5, Molecular biology, LRP6 and AXIN1. His Cell signaling study incorporates themes from Heterotrimeric G protein, G protein and G protein-coupled receptor. His G protein research entails a greater understanding of Biochemistry.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Protein kinase C is differentially stimulated by Wnt and Frizzled homologs in aG-protein-dependent manner
Laird C. Sheldahl;Maiyon Park;Craig C. Malbon;Randall T. Moon.
Current Biology (1999)
Physiological regulation of G protein-linked signaling.
Andrew J. Morris;Craig C. Malbon.
Physiological Reviews (1999)
Agonist-induced Destabilization of β-Adrenergic Receptor mRNA: Attenuation of glucocorticoid-induced up-regulation of β-adrenergic receptors
J. R. Hadcock;Hsien-Yu Wang;C. C. Malbon.
Journal of Biological Chemistry (1989)
G Protein Signaling from Activated Rat Frizzled-1 to the β-Catenin-Lef-Tcf Pathway
Tong Liu;Anthony J. DeCostanzo;Xunxian Liu;Hsien-yu Wang.
Science (2001)
Down-regulation of beta-adrenergic receptors: agonist-induced reduction in receptor mRNA levels
John R. Hadcock;Craig C. Malbon.
Proceedings of the National Academy of Sciences of the United States of America (1988)
Dynamic Complexes of β2-Adrenergic Receptors with Protein Kinases and Phosphatases and the Role of Gravin
Meiling Shih;Fubao Lin;John D. Scott;Hsien Yu Wang.
Journal of Biological Chemistry (1999)
Insulin action impaired by deficiency of the G-protein subunit G iα2
Christopher M. Moxham;Craig C. Malbon.
Nature (1996)
Regulation of beta-adrenergic receptors by "permissive" hormones: glucocorticoids increase steady-state levels of receptor mRNA.
John R. Hadcock;Craig C. Malbon.
Proceedings of the National Academy of Sciences of the United States of America (1988)
Overexpression of mitogen-activated protein kinase phosphatases MKP1, MKP2 in human breast cancer.
Hsien-yu Wang;Zhiyong Cheng;Craig C. Malbon.
Cancer Letters (2003)
G proteins in development
Craig C. Malbon.
Nature Reviews Molecular Cell Biology (2005)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Washington
University of Wisconsin–Madison
Kindai University
Nara Medical University
Osaka University
University of Kentucky
University of North Carolina at Chapel Hill
University of Massachusetts Medical School
Stony Brook University
University of Massachusetts Medical School
University of Auckland
Université Paris Cité
University of Angers
Texas Tech University
University of Hull
University of Electronic Science and Technology of China
Roskilde University
CAB International
Kiel University
National Institutes of Health
University of Connecticut Health Center
Florida International University
Pennsylvania State University
University of Manchester
University of Basel
University of Central Florida