Takenobu Kamada focuses on Internal medicine, Cardiology, Ischemia, Hepatitis C virus and Pathology. His studies deal with areas such as Gastroenterology and Endocrinology as well as Internal medicine. His Cardiology research incorporates themes from Doppler echocardiography, Surgery, Vascular disease and Ultrasound.
His Ischemia study combines topics from a wide range of disciplines, such as Anesthesia, Hippocampus, Blood flow and Programmed cell death. His Hepatitis C virus research integrates issues from Chronic liver disease, Titer and Hepatitis C. His research in Pathology intersects with topics in Neuroscience and Blood volume.
Takenobu Kamada mostly deals with Internal medicine, Cardiology, Endocrinology, Ischemia and Pathology. His study on Internal medicine is mostly dedicated to connecting different topics, such as Gastroenterology. He studies Stomach which is a part of Gastroenterology.
His Cardiology study frequently draws connections to adjacent fields such as Diastole. His works in Diabetes mellitus and Insulin are all subjects of inquiry into Endocrinology. Takenobu Kamada interconnects Anesthesia, Adenosine and Pharmacology in the investigation of issues within Ischemia.
His primary areas of investigation include Internal medicine, Cardiology, Endocrinology, Immunology and Gastroenterology. His Internal medicine research focuses on Diabetes mellitus and how it relates to Insulin. His research links Diastole with Cardiology.
He combines subjects such as Cotransporter, Cell culture and Molecular biology with his study of Endocrinology. He has researched Hepatitis C virus in several fields, including Viral disease, Hepatocellular carcinoma and Hepatitis C. The various areas that Takenobu Kamada examines in his Ischemia study include Anesthesia and Adenosine.
His primary scientific interests are in Internal medicine, Endocrinology, Cardiology, Immunology and Hepatitis C virus. In his research on the topic of Internal medicine, Pathology and Hepatocellular carcinoma is strongly related with Gastroenterology. His study explores the link between Endocrinology and topics such as Symporter that cross with problems in Tonicity, Macula densa and Medulla.
Takenobu Kamada has included themes like Vascular disease and Diastole in his Cardiology study. His work in Hepatitis C virus addresses issues such as Interferon alfa, which are connected to fields such as Chemotherapy and Hepatitis C. His studies in Ischemia integrate themes in fields like Hippocampus and Contractility.
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'Ischemic tolerance' phenomenon found in the brain
Kazuo Kitagawa;Masayasu Matsumoto;Masafumi Tagaya;Ryuji Hata.
Brain Research (1990)
Vagally mediated heart rate recovery after exercise is accelerated in athletes but blunted in patients with chronic heart failure
Katsuji Imai;Hideyuki Sato;Masatsugu Hori;Hideo Kusuoka.
Journal of the American College of Cardiology (1994)
Delayed effects of sublethal ischemia on the acquisition of tolerance to ischemia.
T Kuzuya;S Hoshida;N Yamashita;H Fuji.
Circulation Research (1993)
Glomerulosclerosis induced by in vivo transfection of transforming growth factor-beta or platelet-derived growth factor gene into the rat kidney.
Yoshitaka Isaka;Yoshihiro Fujiwara;Naohiko Ueda;Yasufumi Kaneda.
Journal of Clinical Investigation (1993)
Quantitative analysis of hepatitis C virus RNA in serum during interferon alfa therapy
Hideki Hagiwara;Norio Hayashi;Eiji Mita;Tetsuo Takehara.
Estrogen regulation of the insulin-like growth factor I gene transcription involves an AP-1 enhancer.
Y Umayahara;R Kawamori;H Watada;E Imano.
Journal of Biological Chemistry (1994)
Ultrasonic evaluation of early carotid atherosclerosis.
Nobuo Handa;Masayasu Matsumoto;Hiroaki Maeda;Hidetaka Hougaku.
Immunohistochemical detection of Fas antigen in liver tissue of patients with chronic hepatitis C
Naoki Hiramatsu;Norio Hayashi;Kazuhiro Katayama;Kiyoshi Mochizuki.
‘Ischemic tolerance’ phenomenon detected in various brain regions
Kazuo Kitagawa;Masayasu Matsumoto;Keisuke Kuwabara;Masafumi Tagaya.
Brain Research (1991)
Glycation-dependent, reactive oxygen species-mediated suppression of the insulin gene promoter activity in HIT cells.
T.-A. Matsuoka;Y. Kajimoto;H. Watada;H. Kaneto.
Journal of Clinical Investigation (1997)
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