What is he best known for?
The fields of study he is best known for:
His scientific interests lie mostly in DNA repair, Homologous recombination, Molecular biology, DNA and Non-homologous end joining.
His study with DNA repair involves better knowledge in Genetics.
The Homologous recombination study which covers Cell biology that intersects with DNA replication.
His Molecular biology research integrates issues from RAD51, Poly ADP ribose polymerase and Homology directed repair.
His research in Homology directed repair intersects with topics in Double Strand Break Repair and Microhomology-mediated end joining.
His research investigates the connection between DNA and topics such as Ubiquitin that intersect with issues in Antephase Checkpoint.
His most cited work include:
- Playing the End Game: DNA Double-Strand Break Repair Pathway Choice (1027 citations)
- Double-strand break repair: 53BP1 comes into focus (587 citations)
- Components of the Ku‐dependent non‐homologous end‐joining pathway are involved in telomeric length maintenance and telomeric silencing (578 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are DNA repair, Cell biology, Genetics, DNA damage and Homologous recombination.
His DNA repair study combines topics in areas such as Molecular biology and Caenorhabditis elegans.
His Molecular biology research includes themes of Replication protein A, Homology directed repair, Poly ADP ribose polymerase and Proliferating cell nuclear antigen.
His Cell biology research integrates issues from CHEK1, Suppressor, Genome instability, Telomere and DNA replication.
His DNA damage research is multidisciplinary, incorporating elements of Chromatin, G2-M DNA damage checkpoint, Synthetic lethality and Phosphorylation.
His Homologous recombination study frequently intersects with other fields, such as Homologous chromosome.
He most often published in these fields:
- DNA repair (42.96%)
- Cell biology (40.74%)
- Genetics (36.30%)
What were the highlights of his more recent work (between 2018-2021)?
- DNA damage (31.11%)
- Cell biology (40.74%)
- Telomere (17.04%)
In recent papers he was focusing on the following fields of study:
Simon J. Boulton mainly investigates DNA damage, Cell biology, Telomere, DNA repair and DNA replication.
In his research, Non-homologous end joining and Protein subunit is intimately related to G2-M DNA damage checkpoint, which falls under the overarching field of DNA damage.
The study incorporates disciplines such as Chromatin, Chromosome and Synthetic lethality in addition to Cell biology.
His DNA repair research incorporates elements of PARP inhibitor and Homologous recombination.
Genetics covers Simon J. Boulton research in Homologous recombination.
His DNA replication research is multidisciplinary, incorporating perspectives in Cellular differentiation, Cancer research, Genome instability and Helicase.
Between 2018 and 2021, his most popular works were:
- Moving From Poly (ADP-Ribose) Polymerase Inhibition to Targeting DNA Repair and DNA Damage Response in Cancer Therapy (47 citations)
- CDK phosphorylation of TRF2 controls t-loop dynamics during the cell cycle (28 citations)
- SLX4IP Antagonizes Promiscuous BLM Activity during ALT Maintenance. (24 citations)
In his most recent research, the most cited papers focused on:
Simon J. Boulton focuses on DNA damage, Cell biology, DNA repair, Telomere and Cancer research.
His research integrates issues of Meiosis and Synthetic lethality in his study of DNA damage.
The concepts of his Cell biology study are interwoven with issues in Sister chromatids, Chromosome, Synaptonemal complex, Germline and Meiotic chromosome segregation.
His studies deal with areas such as Shelterin, Cyclin-dependent kinase, DNA replication and Helicase as well as DNA repair.
His study on Cancer research also encompasses disciplines like
- Kinase which connect with DNA,
- Cell signaling and related RAD51.
As part of one scientific family, Simon J. Boulton deals mainly with the area of Telomerase, narrowing it down to issues related to the Telomere Recombination, and often Homologous recombination.
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