World's Best Scientists 2026 revealed!

D-Index & Metrics

Genetics

D-Index
81
Citations
25659
World Ranking
1521
National Ranking
193

Simon J. Boulton publication distribution in Genetics in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Genetics in 2026. The highlighted bar marks where Simon J. Boulton sits on this spectrum.

45–54 publications: 6 scientists 55–64 publications: 10 scientists 65–74 publications: 35 scientists 75–84 publications: 84 scientists 85–94 publications: 102 scientists 95–104 publications: 151 scientists 105–114 publications: 175 scientists 115–124 publications: 203 scientists 125–134 publications: 217 scientists 135–144 publications: 205 scientists 145–154 publications: 193 scientists 155–164 publications: 188 scientists 165–174 publications: 170 scientists 175–184 publications: 178 scientists 185–194 publications: 164 scientists 195–204 publications: 173 scientists 205–214 publications: 159 scientists 215–224 publications: 134 scientists 225–234 publications: 143 scientists 235–244 publications: 105 scientists 245–254 publications: 114 scientists 255–264 publications: 92 scientists 265–274 publications: 88 scientists 275–284 publications: 87 scientists 285–294 publications: 80 scientists 295–304 publications: 62 scientists 305–314 publications: 75 scientists 315–324 publications: 67 scientists 325–334 publications: 60 scientists 335–344 publications: 52 scientists 345–354 publications: 40 scientists 355–364 publications: 48 scientists 365–374 publications: 47 scientists 375–384 publications: 46 scientists 385–394 publications: 31 scientists 395–404 publications: 27 scientists 405–414 publications: 40 scientists 415–424 publications: 30 scientists 425–434 publications: 43 scientists 435–444 publications: 29 scientists 445–454 publications: 14 scientists 455–464 publications: 28 scientists 465–474 publications: 21 scientists 475–484 publications: 21 scientists 485–494 publications: 22 scientists 495–504 publications: 17 scientists 505–514 publications: 12 scientists 515–524 publications: 11 scientists 525–534 publications: 8 scientists 535–544 publications: 8 scientists 545–554 publications: 14 scientists 555–564 publications: 4 scientists 565–574 publications: 11 scientists 575–584 publications: 5 scientists 585–594 publications: 11 scientists 595–604 publications: 12 scientists 605–614 publications: 7 scientists 615–624 publications: 6 scientists 625–634 publications: 10 scientists 635–644 publications: 9 scientists 645–654 publications: 10 scientists 655–664 publications: 6 scientists 665–674 publications: 6 scientists 675–684 publications: 6 scientists 685–694 publications: 4 scientists 695–702 publications: 6 scientists 703+ publications: 100 scientists
45 publications 703+

This scientist: 180 publications — 43rd percentile

43% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 703 publications or more.

Simon J. Boulton D-index placement in Genetics in 2026

The chart shows the D-index (discipline H-index) distribution of Genetics scientists ranked by Research.com in 2026. The highlighted bar marks where Simon J. Boulton sits on this spectrum.

40–41 D-Index: 24 scientists 42–43 D-Index: 52 scientists 44–45 D-Index: 84 scientists 46–47 D-Index: 112 scientists 48–49 D-Index: 118 scientists 50–51 D-Index: 141 scientists 52–53 D-Index: 143 scientists 54–55 D-Index: 145 scientists 56–57 D-Index: 179 scientists 58–59 D-Index: 162 scientists 60–61 D-Index: 175 scientists 62–63 D-Index: 191 scientists 64–65 D-Index: 172 scientists 66–67 D-Index: 184 scientists 68–69 D-Index: 164 scientists 70–71 D-Index: 158 scientists 72–73 D-Index: 150 scientists 74–75 D-Index: 136 scientists 76–77 D-Index: 127 scientists 78–79 D-Index: 127 scientists 80–81 D-Index: 111 scientists 82–83 D-Index: 110 scientists 84–85 D-Index: 110 scientists 86–87 D-Index: 84 scientists 88–89 D-Index: 102 scientists 90–91 D-Index: 66 scientists 92–93 D-Index: 72 scientists 94–95 D-Index: 70 scientists 96–97 D-Index: 54 scientists 98–99 D-Index: 60 scientists 100–101 D-Index: 49 scientists 102–103 D-Index: 55 scientists 104–105 D-Index: 45 scientists 106–107 D-Index: 42 scientists 108–109 D-Index: 28 scientists 110–111 D-Index: 39 scientists 112–113 D-Index: 25 scientists 114–115 D-Index: 31 scientists 116–117 D-Index: 29 scientists 118–119 D-Index: 34 scientists 120–121 D-Index: 29 scientists 122–123 D-Index: 29 scientists 124–125 D-Index: 18 scientists 126–127 D-Index: 27 scientists 128–129 D-Index: 22 scientists 130–131 D-Index: 16 scientists 132–133 D-Index: 11 scientists 134–135 D-Index: 17 scientists 136–137 D-Index: 12 scientists 138–139 D-Index: 21 scientists 140–141 D-Index: 4 scientists 142–143 D-Index: 9 scientists 144–145 D-Index: 14 scientists 146–147 D-Index: 6 scientists 148–149 D-Index: 10 scientists 150–151 D-Index: 7 scientists 152–153 D-Index: 9 scientists 154–155 D-Index: 8 scientists 156–157 D-Index: 8 scientists 158–159 D-Index: 9 scientists 160+ D-Index: 96 scientists
40 D-Index 160+

This scientist: 81 D-Index — 66th percentile

66% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 160 D-Index or more.

Overview

Simon J. Boulton is affiliated with The Francis Crick Institute in the United Kingdom, where their research focuses on molecular biology and genetics related to DNA repair and cancer therapy. Their work spans several main fields including Biochemistry, Genetics and Molecular Biology, and Medicine, with a strong emphasis on subfields such as Molecular Biology, Oncology, Genetics, Physiology, and Aging.

Research topics explored by Simon J. Boulton include:

  • DNA Repair Mechanisms
  • CRISPR and Genetic Engineering
  • PARP inhibition in cancer therapy
  • Telomeres, Telomerase, and Senescence
  • Genetics, Aging, and Longevity in Model Organisms
  • Advanced biosensing and bioanalysis techniques
  • Genomics and Chromatin Dynamics

Simon J. Boulton has been published extensively in scientific journals, with frequent publication venues comprising:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Molecular Cell
  • Nature Communications
  • Cell Reports
  • Nature

Notable recent papers include:

  • "Polθ inhibitors elicit BRCA-gene synthetic lethality and target PARP inhibitor resistance," 2021, Nature Communications
  • "Defective ALC1 nucleosome remodeling confers PARPi sensitization and synthetic lethality with HRD," 2020, Molecular Cell
  • "Induction of APOBEC3 Exacerbates DNA Replication Stress and Chromosomal Instability in Early Breast and Lung Cancer Evolution," 2021, Cancer Discovery
  • "Targeting the nucleotide salvage factor DNPH1 sensitizes BRCA -deficient cells to PARP inhibitors," 2021, Science
  • "POLQ seals post-replicative ssDNA gaps to maintain genome stability in BRCA-deficient cancer cells," 2022, Molecular Cell

The scientist has collaborated frequently with other researchers in their field. Frequent co-authors include:

  • Graeme Hewitt
  • Ondrej Beláň
  • David Rueda
  • Matthew D. Newton
  • Roopesh Anand

Best Publications

  • Playing the End Game: DNA Double-Strand Break Repair Pathway Choice

    J. Ross Chapman;Martin R.G. Taylor;Simon J. Boulton

  • Double-strand break repair: 53BP1 comes into focus

    Stephanie Panier;Simon J. Boulton

  • Components of the Ku‐dependent non‐homologous end‐joining pathway are involved in telomeric length maintenance and telomeric silencing

    Simon J. Boulton;Stephen P. Jackson

  • Homologous-recombination-deficient tumours are dependent on Polθ-mediated repair

    Raphael Ceccaldi;Jessica C. Liu;Ravindra Amunugama;Ildiko Hajdu

  • RIF1 Is Essential for 53BP1-Dependent Nonhomologous End Joining and Suppression of DNA Double-Strand Break Resection

    J. Ross Chapman;Patricia Barral;Jean-Baptiste Vannier;Valérie Borel

  • Poly(ADP-ribose)-dependent regulation of DNA repair by the chromatin remodeling enzyme ALC1.

    Dragana Ahel;Zuzana Hořejší;Nicola Wiechens;Sophie E. Polo

  • Identification of a Saccharomyces Cerevisiae Ku80 Homologue: Roles in DNA Double Strand Break Rejoining and in Telomeric Maintenance

    Simon J. Boulton;Stephen P. Jackson

  • Saccharomyces cerevisiae Ku70 potentiates illegitimate DNA double-strand break repair and serves as a barrier to error-prone DNA repair pathways.

    S. J. Boulton;S. P. Jackson

  • REV7 counteracts DNA double-strand break resection and affects PARP inhibition

    Guotai Xu;J. Ross Chapman;Inger Brandsma;Jingsong Yuan

  • RTEL1 Dismantles T Loops and Counteracts Telomeric G4-DNA to Maintain Telomere Integrity

    Jean-Baptiste Vannier;Visnja Pavicic-Kaltenbrunner;Mark I.R. Petalcorin;Hao Ding

  • Poly(ADP-ribose)-binding zinc finger motifs in DNA repair/checkpoint proteins

    Ivan Ahel;Dragana Ahel;Takahiro Matsusaka;Allison J. Clark

  • RTEL1 maintains genomic stability by suppressing homologous recombination.

    Louise J. Barber;Jillian L. Youds;Jillian L. Youds;Jordan D. Ward;Michael J. McIlwraith

  • C. elegans ORFeome version 1.1: experimental verification of the genome annotation and resource for proteome-scale protein expression.

    Jérôme Reboul;Philippe Vaglio;Jean François Rual;Jean François Rual;Philippe Lamesch;Philippe Lamesch

  • The C. elegans homolog of the p53 tumor suppressor is required for DNA damage-induced apoptosis

    Björn Schumacher;Kay Hofmann;Simon Boulton;Anton Gartner

  • Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place.

    Panagiotis Kotsantis;Eva Petermann;Simon J. Boulton

  • Cellular functions of the BRCA tumour-suppressor proteins

    Simon Boulton

  • BRCA1-associated exclusion of 53BP1 from DNA damage sites underlies temporal control of DNA repair.

    J. Ross Chapman;J. Ross Chapman;Alex J. Sossick;Simon J. Boulton;Stephen P. Jackson

  • Preventing nonhomologous end joining suppresses DNA repair defects of Fanconi anemia.

    Adele Adamo;Spencer J. Collis;Spencer J. Collis;Carrie A. Adelman;Nicola Silva;Nicola Silva

  • Combined Functional Genomic Maps of the C. elegans DNA Damage Response

    Simon J. Boulton;Anton Gartner;Jérôme Reboul;Philippe Vaglio

  • Caenorhabditis elegans HUS-1 is a DNA damage checkpoint protein required for genome stability and EGL-1-mediated apoptosis

    E. R. Hofmann;E. R. Hofmann;S. Milstein;S. Milstein;S. Milstein;S. J. Boulton;M. J. Ye

Frequent Co-Authors

J. Mark Skehel
J. Mark Skehel MRC Laboratory of Molecular Biology
Stephen C. West
Stephen C. West The Francis Crick Institute
Anton Gartner
Anton Gartner University of Dundee
Charles Swanton
Charles Swanton The Francis Crick Institute
Michael Howell
Michael Howell The Francis Crick Institute
Stephen P. Jackson
Stephen P. Jackson University of Cambridge
Paolo Plevani
Paolo Plevani University of Milan
Nicholas M. Luscombe
Nicholas M. Luscombe The Francis Crick Institute
Peter J. Campbell
Peter J. Campbell Wellcome Sanger Institute
Charlotte M. Niemeyer
Charlotte M. Niemeyer University of Freiburg

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