What is he best known for?
The fields of study he is best known for:
His primary scientific interests are in DNA damage, Genetics, DNA repair, Molecular biology and Cell cycle checkpoint.
Anton Gartner has researched DNA damage in several fields, including Transcription factor and Gene.
The Genetics study combines topics in areas such as NAD+ kinase and Sirtuin.
His DNA repair research includes elements of Phenotype, Model organism and Genome instability.
The study incorporates disciplines such as Synapsis, Prophase and Synaptonemal complex in addition to Molecular biology.
His Cell cycle checkpoint research incorporates themes from Origin of replication and Cell biology.
His most cited work include:
- A conserved checkpoint pathway mediates DNA damage--induced apoptosis and cell cycle arrest in C. elegans. (420 citations)
- FAR1 links the signal transduction pathway to the cell cycle machinery in yeast (297 citations)
- Excess Mcm2–7 license dormant origins of replication that can be used under conditions of replicative stress (295 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Cell biology, Genetics, DNA damage, Caenorhabditis elegans and DNA repair.
His work deals with themes such as Chromatin and Apoptosis, Cell cycle, Cell cycle checkpoint, which intersect with Cell biology.
His DNA damage research is multidisciplinary, relying on both G2-M DNA damage checkpoint, Transcription factor, Germline, Molecular biology and Germ cell.
His research in Molecular biology intersects with topics in Regulation of gene expression and DNA ligase.
His Caenorhabditis elegans study combines topics in areas such as Oxidative stress, Dopaminergic, Proteomics and Programmed cell death.
His DNA repair research is multidisciplinary, incorporating elements of Mutation, Mutagenesis and Homologous recombination.
He most often published in these fields:
- Cell biology (54.17%)
- Genetics (40.83%)
- DNA damage (35.00%)
What were the highlights of his more recent work (between 2017-2021)?
- Genetics (40.83%)
- DNA repair (33.33%)
- Cell biology (54.17%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Genetics, DNA repair, Cell biology, DNA and DNA damage.
His DNA repair study frequently links to other fields, such as Homologous recombination.
His biological study spans a wide range of topics, including Chromatin, Caenorhabditis elegans, Chromosome segregation and Enzyme.
His Caenorhabditis elegans study integrates concerns from other disciplines, such as Oxidative stress and Dopaminergic.
His work focuses on many connections between DNA and other disciplines, such as Mutation rate, that overlap with his field of interest in Point mutation and Whole genome sequencing.
In his work, Hsp70 and Carcinogenesis is strongly intertwined with APAF1, which is a subfield of DNA damage.
Between 2017 and 2021, his most popular works were:
- Mutational signatures of DNA mismatch repair deficiency in C. elegans and human cancers. (50 citations)
- Mutational signatures are jointly shaped by DNA damage and repair (36 citations)
- Mutational signatures are jointly shaped by DNA damage and repair (36 citations)
In his most recent research, the most cited papers focused on:
His main research concerns Mutation, Cell biology, DNA repair, DNA damage and Genetics.
His research integrates issues of Genome and Methylation in his study of Mutation.
His Cell biology research integrates issues from Ubiquitin, Homologous recombination, Chromosome segregation, Chromatin and APAF1.
His DNA repair research incorporates elements of Whole genome sequencing, Mutation rate and Point mutation.
His DNA damage study incorporates themes from Apoptosis, NEDD8, Chaperone, Enzyme and Hsp70.
His DNA sequencing, Gene knockout and DNA mismatch repair study in the realm of Genetics interacts with subjects such as CpG site and Somatic hypermutation.
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