His work on Cell biology is being expanded to include thematically relevant topics such as Signal transducing adaptor protein. Shoji Yamaoka merges Signal transducing adaptor protein with Signal transduction in his research. Shoji Yamaoka integrates many fields in his works, including Signal transduction and Apoptosis. As part of his studies on Apoptosis, Shoji Yamaoka often connects relevant areas like Biochemistry. He frequently studies issues relating to NFKB1 and Biochemistry. He combines Gene and Ubiquitin ligase in his research. His work blends Ubiquitin ligase and Ubiquitin studies together. He undertakes multidisciplinary investigations into Ubiquitin and Proteasome in his work. Proteasome and Cell biology are commonly linked in his work.
His research on Cell biology often connects related topics like Phosphorylation. His studies link Cell biology with Phosphorylation. In his works, he conducts interdisciplinary research on Gene and Gene expression. Borrowing concepts from Molecular biology, Shoji Yamaoka weaves in ideas under Gene expression. Shoji Yamaoka conducted interdisciplinary study in his works that combined Molecular biology and Gene. He combines Genetics and Immunology in his studies. He combines Immunology and Genetics in his studies. His In vitro research extends to the thematically linked field of Biochemistry. He frequently studies issues relating to Biochemistry and In vitro.
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Complementation Cloning of NEMO, a Component of the IκB Kinase Complex Essential for NF-κB Activation
Shoji Yamaoka;Gilles Courtois;Christine Bessia;Simon T Whiteside.
Role of the Toll-like Receptor 4/NF-κB Pathway in Saturated Fatty Acid-induced Inflammatory Changes in the Interaction Between Adipocytes and Macrophages
Takayoshi Suganami;Kanami Tanimoto-Koyama;Junko Nishida;Michiko Itoh.
Arteriosclerosis, Thrombosis, and Vascular Biology (2007)
Involvement of linear polyubiquitylation of NEMO in NF-kappaB activation.
Fuminori Tokunaga;Shin-ichi Sakata;Shin-ichi Sakata;Yasushi Saeki;Yoshinori Satomi.
Nature Cell Biology (2009)
Genomic rearrangement in NEMO impairs NF-kappaB activation and is a cause of incontinentia pigmenti. The International Incontinentia Pigmenti (IP) Consortium.
Asmae Smahi;G. Courtois;P. Vabres;S. Yamaoka.
Regulation of NF-κB Signaling by Pin1-Dependent Prolyl Isomerization and Ubiquitin-Mediated Proteolysis of p65/RelA
Akihide Ryo;Futoshi Suizu;Yasuhiro Yoshida;Kilian Perrem.
Molecular Cell (2003)
Neutrophil extracellular traps mediate a host defense response to human immunodeficiency virus-1.
Tatsuya Saitoh;Jun Komano;Yasunori Saitoh;Takuma Misawa.
Cell Host & Microbe (2012)
Regulation of Toll/IL-1-receptor-mediated gene expression by the inducible nuclear protein IκBζ
Masahiro Yamamoto;Soh Yamazaki;Satoshi Uematsu;Shintaro Sato.
Oncogenic transformation by the tax gene of human T-cell leukemia virus type I in vitro
Atsushi Tanaka;Chiaki Takahashi;Shoji Yamaoka;Tetsuya Nosaka.
Proceedings of the National Academy of Sciences of the United States of America (1990)
Bcl10 and MALT1, Independent Targets of Chromosomal Translocation in MALT Lymphoma, Cooperate in a Novel NF-κB Signaling Pathway
Peter C. Lucas;Masakatsu Yonezumi;Naohiro Inohara;Linda M. McAllister-Lucas.
Journal of Biological Chemistry (2001)
Negative regulation of interferon-regulatory factor 3-dependent innate antiviral response by the prolyl isomerase Pin1
Tatsuya Saitoh;Adrian Tun-Kyi;Akihide Ryo;Masahiro Yamamoto.
Nature Immunology (2006)
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