D-Index & Metrics Best Publications
Masakazu Hatanaka

Masakazu Hatanaka

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Molecular Biology D-index 64 Citations 12,698 214 World Ranking 1136 National Ranking 103

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

The scientist’s investigation covers issues in Molecular biology, Virus, Virology, Human T-lymphotropic virus 1 and Cell culture. His Molecular biology research is multidisciplinary, relying on both Cell, Nucleolus and Complementary DNA, Peptide sequence, Gene. Masakazu Hatanaka interconnects CATS, Immunological tests, Antigen-antibody reactions, Rhabdomyosarcoma and Epitope in the investigation of issues within Virus.

His work in Virology addresses subjects such as Provirus, which are connected to disciplines such as Transactivation. Masakazu Hatanaka has included themes like Transfection and Cell growth in his Human T-lymphotropic virus 1 study. His work deals with themes such as Viral culture, Clone and Antigen, which intersect with Cell culture.

His most cited work include:

  • Oncogenic transformation by the tax gene of human T-cell leukemia virus type I in vitro (364 citations)
  • Induction of inflammatory arthropathy resembling rheumatoid arthritis in mice transgenic for HTLV-I. (321 citations)
  • Transport of sugars in tumor cell membranes (296 citations)

What are the main themes of his work throughout his whole career to date?

Masakazu Hatanaka spends much of his time researching Molecular biology, Virology, Virus, Cell culture and Gene. His Molecular biology research includes elements of Gene expression, RNA, Nucleolus and Biochemistry, DNA. His Nucleolus research incorporates themes from Cell nucleus and Peptide sequence, Signal peptide.

Masakazu Hatanaka usually deals with Virology and limits it to topics linked to Leukemia and Lymphoma and Carcinogenesis. As a member of one scientific family, he mostly works in the field of Virus, focusing on In vitro and, on occasion, In vivo. His research on Cell culture also deals with topics like

  • Cell growth which connect with Cancer research,
  • Cell biology and related Internal medicine and Endocrinology.

He most often published in these fields:

  • Molecular biology (55.16%)
  • Virology (39.01%)
  • Virus (29.60%)

What were the highlights of his more recent work (between 1993-2008)?

  • Molecular biology (55.16%)
  • Virology (39.01%)
  • Gene (19.28%)

In recent papers he was focusing on the following fields of study:

Masakazu Hatanaka focuses on Molecular biology, Virology, Gene, Long terminal repeat and Virus. His study in Molecular biology is interdisciplinary in nature, drawing from both NFKB1, Genetic enhancement, Viral replication, Provirus and Transcription. His Virology research is multidisciplinary, incorporating perspectives in Regulation of gene expression, Leukemia, Transfection and Cell biology.

His research in Long terminal repeat intersects with topics in Enhancer, Inflammation, DNA-binding protein and Arthritis. The Virus study combines topics in areas such as Mutation, Cell culture, Reverse transcriptase and Protease. His biological study spans a wide range of topics, including Cell, Mutant and Cell growth.

Between 1993 and 2008, his most popular works were:

  • Constitutive activation of NF-kappa B is essential for transformation of rat fibroblasts by the human T-cell leukemia virus type I Tax protein. (177 citations)
  • Tumor necrosis factor alpha-induced phosphorylation of I kappa B alpha is a signal for its degradation but not dissociation from NF-kappa B (176 citations)
  • Crystal structure of calcium bound domain VI of calpain at 1.9 Å resolution and its role in enzyme assembly, regulation, and inhibitor binding (163 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Enzyme

His scientific interests lie mostly in Calpain, Molecular biology, Biochemistry, Mutant and Calpastatin. The various areas that he examines in his Molecular biology study include NFKB1, DNA, Poly ADP ribose polymerase, Poly Polymerase Inhibitor and Protein kinase R. His NFKB1 study also includes

  • Cell, which have a strong connection to Cell culture,
  • In vivo and related Phosphorylation.

His Cell culture course of study focuses on Apoptosis and Virus and Virology. His Mutant research is multidisciplinary, relying on both Cancer research, Recombinant DNA, Inhibitor protein, Tumor progression and Regulation of gene expression. His Calpastatin research includes themes of Protein subunit, Peptide sequence, Protease inhibitor, Calmodulin and Oligopeptide.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Oncogenic transformation by the tax gene of human T-cell leukemia virus type I in vitro

Atsushi Tanaka;Chiaki Takahashi;Shoji Yamaoka;Tetsuya Nosaka.
Proceedings of the National Academy of Sciences of the United States of America (1990)

517 Citations

Induction of inflammatory arthropathy resembling rheumatoid arthritis in mice transgenic for HTLV-I.

Yoichiro Iwakura;Mariko Tosu;Emi Yoshida;Masafumi Takiguchi.
Science (1991)

496 Citations

Transport of sugars in tumor cell membranes

Masakazu Hatanaka.
Biochimica et Biophysica Acta (1974)

467 Citations

C-type virus released from cultured human rhabdomyosarcoma cells

R. M. McALLISTER;M. Nicolson;M. B. Gardner;R. W. Rongey.
Nature (1972)

364 Citations

Sequence requirements for nucleolar localization of human T cell leukemia virus type I pX protein, which regulates viral RNA processing

Haruhiko Siomi;Hisatoshi Shida;Seok Hyun Nam;Tetsuya Nosaka.
Cell (1988)

342 Citations

Differential susceptibility to the acquired immunodeficiency syndrome retrovirus in cloned cells of human leukemic T-cell line Molt-4.

R Kikukawa;Y Koyanagi;S Harada;N Kobayashi.
Journal of Virology (1986)

330 Citations

Gene expression of fibroblast growth factors in human gliomas and meningiomas: demonstration of cellular source of basic fibroblast growth factor mRNA and peptide in tumor tissues.

Jun A. Takahashi;Hirotaka Mori;Manabu Fukumoto;Koichi Igarashi.
Proceedings of the National Academy of Sciences of the United States of America (1990)

307 Citations

Tumor necrosis factor alpha-induced phosphorylation of I kappa B alpha is a signal for its degradation but not dissociation from NF-kappa B

Shigeki Miyamoto;Masatoshi Maki;Mark J. Schmitt;Masakazu Hatanaka.
Proceedings of the National Academy of Sciences of the United States of America (1994)

284 Citations

Constitutive activation of NF-kappa B is essential for transformation of rat fibroblasts by the human T-cell leukemia virus type I Tax protein.

S. Yamaoka;H. Inoue;M. Sakurai;T. Sugiyama.
The EMBO Journal (1996)

269 Citations

Ha-ras oncogenes are activated by somatic alterations in human urinary tract tumours

Jun Fujita;Osamu Yoshida;Yasuhito Yuasa;Johng S. Rhim.
Nature (1984)

246 Citations

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