The scientist’s investigation covers issues in Molecular biology, Virus, Virology, Human T-lymphotropic virus 1 and Cell culture. His Molecular biology research is multidisciplinary, relying on both Cell, Nucleolus and Complementary DNA, Peptide sequence, Gene. Masakazu Hatanaka interconnects CATS, Immunological tests, Antigen-antibody reactions, Rhabdomyosarcoma and Epitope in the investigation of issues within Virus.
His work in Virology addresses subjects such as Provirus, which are connected to disciplines such as Transactivation. Masakazu Hatanaka has included themes like Transfection and Cell growth in his Human T-lymphotropic virus 1 study. His work deals with themes such as Viral culture, Clone and Antigen, which intersect with Cell culture.
Masakazu Hatanaka spends much of his time researching Molecular biology, Virology, Virus, Cell culture and Gene. His Molecular biology research includes elements of Gene expression, RNA, Nucleolus and Biochemistry, DNA. His Nucleolus research incorporates themes from Cell nucleus and Peptide sequence, Signal peptide.
Masakazu Hatanaka usually deals with Virology and limits it to topics linked to Leukemia and Lymphoma and Carcinogenesis. As a member of one scientific family, he mostly works in the field of Virus, focusing on In vitro and, on occasion, In vivo. His research on Cell culture also deals with topics like
Masakazu Hatanaka focuses on Molecular biology, Virology, Gene, Long terminal repeat and Virus. His study in Molecular biology is interdisciplinary in nature, drawing from both NFKB1, Genetic enhancement, Viral replication, Provirus and Transcription. His Virology research is multidisciplinary, incorporating perspectives in Regulation of gene expression, Leukemia, Transfection and Cell biology.
His research in Long terminal repeat intersects with topics in Enhancer, Inflammation, DNA-binding protein and Arthritis. The Virus study combines topics in areas such as Mutation, Cell culture, Reverse transcriptase and Protease. His biological study spans a wide range of topics, including Cell, Mutant and Cell growth.
His scientific interests lie mostly in Calpain, Molecular biology, Biochemistry, Mutant and Calpastatin. The various areas that he examines in his Molecular biology study include NFKB1, DNA, Poly ADP ribose polymerase, Poly Polymerase Inhibitor and Protein kinase R. His NFKB1 study also includes
His Cell culture course of study focuses on Apoptosis and Virus and Virology. His Mutant research is multidisciplinary, relying on both Cancer research, Recombinant DNA, Inhibitor protein, Tumor progression and Regulation of gene expression. His Calpastatin research includes themes of Protein subunit, Peptide sequence, Protease inhibitor, Calmodulin and Oligopeptide.
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Oncogenic transformation by the tax gene of human T-cell leukemia virus type I in vitro
Atsushi Tanaka;Chiaki Takahashi;Shoji Yamaoka;Tetsuya Nosaka.
Proceedings of the National Academy of Sciences of the United States of America (1990)
Induction of inflammatory arthropathy resembling rheumatoid arthritis in mice transgenic for HTLV-I.
Yoichiro Iwakura;Mariko Tosu;Emi Yoshida;Masafumi Takiguchi.
Science (1991)
Transport of sugars in tumor cell membranes
Masakazu Hatanaka.
Biochimica et Biophysica Acta (1974)
C-type virus released from cultured human rhabdomyosarcoma cells
R. M. McALLISTER;M. Nicolson;M. B. Gardner;R. W. Rongey.
Nature (1972)
Sequence requirements for nucleolar localization of human T cell leukemia virus type I pX protein, which regulates viral RNA processing
Haruhiko Siomi;Hisatoshi Shida;Seok Hyun Nam;Tetsuya Nosaka.
Cell (1988)
Differential susceptibility to the acquired immunodeficiency syndrome retrovirus in cloned cells of human leukemic T-cell line Molt-4.
R Kikukawa;Y Koyanagi;S Harada;N Kobayashi.
Journal of Virology (1986)
Gene expression of fibroblast growth factors in human gliomas and meningiomas: demonstration of cellular source of basic fibroblast growth factor mRNA and peptide in tumor tissues.
Jun A. Takahashi;Hirotaka Mori;Manabu Fukumoto;Koichi Igarashi.
Proceedings of the National Academy of Sciences of the United States of America (1990)
Tumor necrosis factor alpha-induced phosphorylation of I kappa B alpha is a signal for its degradation but not dissociation from NF-kappa B
Shigeki Miyamoto;Masatoshi Maki;Mark J. Schmitt;Masakazu Hatanaka.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Constitutive activation of NF-kappa B is essential for transformation of rat fibroblasts by the human T-cell leukemia virus type I Tax protein.
S. Yamaoka;H. Inoue;M. Sakurai;T. Sugiyama.
The EMBO Journal (1996)
Ha-ras oncogenes are activated by somatic alterations in human urinary tract tumours
Jun Fujita;Osamu Yoshida;Yasuhito Yuasa;Johng S. Rhim.
Nature (1984)
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