What is he best known for?
The fields of study he is best known for:
- Gene
- DNA
- Internal medicine
His primary areas of study are Internal medicine, Endocrinology, Cell biology, Signal transduction and Muscle hypertrophy.
His Internal medicine research incorporates themes from Endothelial stem cell, Gene expression and Myosin.
His Endocrinology study integrates concerns from other disciplines, such as Downregulation and upregulation and Growth factor.
His Cell biology research is multidisciplinary, incorporating elements of Phenotype, Molecular biology and Programmed cell death.
As part of the same scientific family, Seigo Izumo usually focuses on Signal transduction, concentrating on Autocrine signalling and intersecting with Mediator.
In his study, Heat shock protein, Cardiac Myosins and Tropomyosin is inextricably linked to Skeletal muscle, which falls within the broad field of Muscle hypertrophy.
His most cited work include:
- Hemodynamic shear stress and its role in atherosclerosis. (2544 citations)
- Endothelial-to-mesenchymal transition contributes to cardiac fibrosis (1490 citations)
- Molecular characterization of angiotensin II--induced hypertrophy of cardiac myocytes and hyperplasia of cardiac fibroblasts. Critical role of the AT1 receptor subtype. (1273 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Internal medicine, Endocrinology, Cell biology, Molecular biology and Myocyte.
His Internal medicine research integrates issues from PI3K/AKT/mTOR pathway and Cardiology.
The various areas that Seigo Izumo examines in his Endocrinology study include Cardiac function curve, Genetically modified mouse, Signal transduction and Gene expression.
His Cell biology research is multidisciplinary, incorporating perspectives in Regulation of gene expression, Myofibril, Cellular differentiation and Microfilament.
The Molecular biology study combines topics in areas such as Homeobox, Transcription factor, Gene, Gene isoform and Binding site.
The concepts of his Muscle hypertrophy study are interwoven with issues in Cardiac muscle, Left ventricular hypertrophy, Downregulation and upregulation, Circulatory system and Diastole.
He most often published in these fields:
- Internal medicine (48.02%)
- Endocrinology (42.08%)
- Cell biology (28.22%)
What were the highlights of his more recent work (between 2006-2021)?
- Internal medicine (48.02%)
- Endocrinology (42.08%)
- PI3K/AKT/mTOR pathway (12.87%)
In recent papers he was focusing on the following fields of study:
Seigo Izumo mainly focuses on Internal medicine, Endocrinology, PI3K/AKT/mTOR pathway, Cell biology and Protein kinase B.
While the research belongs to areas of Internal medicine, Seigo Izumo spends his time largely on the problem of Cardiology, intersecting his research to questions surrounding P110α.
The study incorporates disciplines such as Genetically modified mouse, Signal transduction and Transcription factor in addition to Endocrinology.
His Mesoderm formation and Stem cell study in the realm of Cell biology connects with subjects such as Electrical conduction system of the heart and Embryoid body.
His Protein kinase B study incorporates themes from Anesthesia and Protein kinase C, Kinase.
His Cardiac function curve study combines topics in areas such as Fibrosis and Dilated cardiomyopathy.
Between 2006 and 2021, his most popular works were:
- Endothelial-to-mesenchymal transition contributes to cardiac fibrosis (1490 citations)
- Protective effects of exercise and phosphoinositide 3-kinase(p110α) signaling in dilated and hypertrophic cardiomyopathy (224 citations)
- Protective effects of exercise and phosphoinositide 3-kinase(p110α) signaling in dilated and hypertrophic cardiomyopathy (224 citations)
In his most recent research, the most cited papers focused on:
Seigo Izumo mainly investigates Fibrosis, Pressure overload, Muscle hypertrophy, Internal medicine and Dilated cardiomyopathy.
His research in Fibrosis intersects with topics in Mesenchymal stem cell, Extracellular matrix and Bone morphogenetic protein 7, Bone morphogenetic protein.
His Pressure overload research is multidisciplinary, relying on both Embryonic heart and Cardiac fibrosis.
His research integrates issues of Circulatory system, Everolimus and Sirolimus in his study of Muscle hypertrophy.
His PI3K/AKT/mTOR pathway research extends to Internal medicine, which is thematically connected.
His work deals with themes such as Cardiac function curve, Hypertrophic cardiomyopathy, Heart disease and Endocrinology, which intersect with Dilated cardiomyopathy.
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