Her primary areas of investigation include Cell biology, Signal transduction, Anti-apoptotic Ras signalling cascade, Kinase and Cell surface receptor. She interconnects Ras superfamily and Genetics in the investigation of issues within Cell biology. The study incorporates disciplines such as Apoptosis, Programmed cell death, Tissue homeostasis, Intrinsic apoptosis and Effector in addition to Signal transduction.
Roya Khosravi-Far has included themes like Cancer cell, Cell type and Mitochondrion in her Programmed cell death study. Roya Khosravi-Far works in the field of Kinase, namely MAPK/ERK pathway. The MAPK/ERK pathway study which covers Molecular biology that intersects with Mitogen-activated protein kinase kinase and Mitogen-activated protein kinase.
Roya Khosravi-Far mainly investigates Cell biology, Apoptosis, Cancer research, Programmed cell death and Signal transduction. Her biological study spans a wide range of topics, including Transcription factor and Biochemistry. Her work on Caspase is typically connected to c-Fos as part of general Apoptosis study, connecting several disciplines of science.
Her work deals with themes such as Tumor necrosis factor alpha, Immunology, Proteasome inhibitor, Carcinogenesis and PI3K/AKT/mTOR pathway, which intersect with Cancer research. The Programmed cell death study combines topics in areas such as Cancer cell, Cancer, Tissue homeostasis and Mitochondrion. Her Signal transduction research is multidisciplinary, incorporating elements of Molecular biology and Cell surface receptor.
Roya Khosravi-Far focuses on Cancer research, Apoptosis, Cancer cell, Programmed cell death and PI3K/AKT/mTOR pathway. Her research investigates the connection with Cancer research and areas like Cancer which intersect with concerns in FOXO3 and Transcription factor. The various areas that Roya Khosravi-Far examines in her Apoptosis study include Fibrosarcoma, Immunology, Immune system and Systemic administration.
Her research on PI3K/AKT/mTOR pathway also deals with topics like
Her main research concerns Cancer research, Imatinib, Tyrosine-kinase inhibitor, Chronic myelogenous leukemia and Imatinib mesylate. The Cancer research study combines topics in areas such as Cancer cell, Cancer, Thyroid cancer and Apoptosis, Protein kinase B. Her biological study spans a wide range of topics, including Kinase and MAPK/ERK pathway.
Her study in Proteasome inhibitor extends to Imatinib with its themes.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Increasing complexity of Ras signaling
Sharon L Campbell;Sharon L Campbell;Roya Khosravi-Far;Kent L Rossman;Geoffrey J Clark.
Development of Human Protein Reference Database as an Initial Platform for Approaching Systems Biology in Humans
Suraj Peri;Suraj Peri;J. Daniel Navarro;J. Daniel Navarro;Ramars Amanchy;Troels Z. Kristiansen;Troels Z. Kristiansen.
Genome Research (2003)
Daxx, a novel fas-binding protein that activates jnk and apoptosis
Xiaolu Yang;Roya Khosravi-Far;Howard Y. Chang;David Baltimore.
Activation of Rac1, RhoA, and mitogen-activated protein kinases is required for Ras transformation.
Roya Khosravi-Far;Patricia A. Solski;Geoffrey J. Clark;Michael S. Kinch.
Molecular and Cellular Biology (1995)
GTP-binding mutants of rab1 and rab2 are potent inhibitors of vesicular transport from the endoplasmic reticulum to the Golgi complex.
Ellen J. Tisdale;Jeffrey R. Bourne;Roya Khosravi-Far;Channing J. Der.
Journal of Cell Biology (1992)
The Mitogen-activated Protein Kinase Phosphatases PAC1, MKP-1, and MKP-2 Have Unique Substrate Specificities and Reduced Activity in Vivo toward the ERK2 sevenmaker Mutation
Yanfang Chu;Patricia A. Solski;Roya Khosravi-Far;Channing J. Der.
Journal of Biological Chemistry (1996)
The Ras signal transduction pathway
Roya Khosravi-Far;Channing J. Der.
Cancer and Metastasis Reviews (1994)
Rab1b regulates vesicular transport between the endoplasmic reticulum and successive Golgi compartments.
H Plutner;A D Cox;S Pind;R Khosravi-Far.
Journal of Cell Biology (1991)
Rho family proteins and Ras transformation: the RHOad less traveled gets congested
Irene M Zohn;Sharon L Campbell;Roya Khosravi-Far;Kent L Rossman.
Oncogenic Ras activation of Raf/mitogen-activated protein kinase-independent pathways is sufficient to cause tumorigenic transformation.
R Khosravi-Far;M A White;J K Westwick;P A Solski.
Molecular and Cellular Biology (1996)
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