1998 - Distinguished Fellowship Award, American College of Cardiology (ACC)
1933 - Fellow of the American Association for the Advancement of Science (AAAS)
Internal medicine, Cardiology, Cardiomyopathy, Hypertrophic cardiomyopathy and Muscle hypertrophy are his primary areas of study. The Internal medicine study combines topics in areas such as Endocrinology and Pathology. His work in Cardiology addresses issues such as Anesthesia, which are connected to fields such as Occlusion.
His research integrates issues of Penetrance, Sudden death, Gene mapping and Bioinformatics in his study of Cardiomyopathy. His Hypertrophic cardiomyopathy study combines topics from a wide range of disciplines, such as Phenotype, Molecular biology, Sudden cardiac death and Gene expression profiling. His Muscle hypertrophy study combines topics in areas such as Left ventricular hypertrophy, Pathogenesis, Doppler echocardiography, Wild type and Myocyte.
His main research concerns Internal medicine, Cardiology, Myocardial infarction, Molecular biology and Hypertrophic cardiomyopathy. Internal medicine is often connected to Endocrinology in his work. His studies in Cardiology integrate themes in fields like Anesthesia and Surgery.
His Myocardial infarction study integrates concerns from other disciplines, such as Tissue plasminogen activator and Chest pain. As a part of the same scientific family, he mostly works in the field of Molecular biology, focusing on Gene and, on occasion, Bioinformatics. The various areas that Robert Roberts examines in his Hypertrophic cardiomyopathy study include Sudden death, Mutation, Troponin complex, Sudden cardiac death and Myosin.
The scientist’s investigation covers issues in Internal medicine, Cardiology, Hypertrophic cardiomyopathy, Cardiomyopathy and Genetics. His work in Muscle hypertrophy, Sudden cardiac death, Heart disease, Troponin T and Creatine kinase are all subfields of Internal medicine research. His Muscle hypertrophy research integrates issues from Doppler echocardiography and Left ventricular hypertrophy.
His study looks at the relationship between Troponin T and topics such as Troponin I, which overlap with Unstable angina, Chest pain and Infarction. In his research on the topic of Hypertrophic cardiomyopathy, Fibrosis, Genotype and Allele is strongly related with Endocrinology. He combines subjects such as Mitral valve, Penetrance, Pathogenesis and Ventricular outflow tract with his study of Cardiomyopathy.
Robert Roberts focuses on Internal medicine, Cardiology, Cardiomyopathy, Hypertrophic cardiomyopathy and Muscle hypertrophy. His work in Cardiac hypertrophy, Brugada syndrome, Conduction system disease, Ventricular tachycardia and Cardiac rhythm disturbances is related to Internal medicine. His Cardiology research is multidisciplinary, incorporating perspectives in Scientific evidence and Consensus conference.
His work carried out in the field of Hypertrophic cardiomyopathy brings together such families of science as Penetrance, Endocrinology, Sudden cardiac death and Troponin T. The concepts of his Endocrinology study are interwoven with issues in Fibrosis and Heart failure. He has researched Muscle hypertrophy in several fields, including Left ventricular hypertrophy, Bioinformatics, Doppler echocardiography, Mitral valve and Age of onset.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Biological, clinical and population relevance of 95 loci for blood lipids
Tanya M. Teslovich;Kiran Musunuru;Albert V. Smith;Andrew C. Edmondson.
Myocardial infarction redefined - A consensus document of the Joint European Society of Cardiology/American College of Cardiology committee for the redefinition of myocardial infarction
J. S. Alpert;E. Antman;F. Apple;P. W. Armstrong.
European Heart Journal (2000)
Plasma HDL cholesterol and risk of myocardial infarction: A mendelian randomisation study
Benjamin F. Voight;Benjamin F. Voight;Benjamin F. Voight;Gina M. Peloso;Gina M. Peloso;Marju Orho-Melander;Ruth Frikke-Schmidt.
web science (2012)
Large-scale association analysis identifies 13 new susceptibility loci for coronary artery disease
Heribert Schunkert;Inke R. König;Sekar Kathiresan;Muredach P. Reilly.
Nature Genetics (2011)
A comprehensive 1000 Genomes–based genome-wide association meta-analysis of coronary artery disease
M Nikpay;A Goel;Won H-H.;L M Hall.
Nature Genetics (2015)
Large-scale association analysis identifies new risk loci for coronary artery disease
Panos Deloukas;Stavroula Kanoni;Christina Willenborg;Martin Farrall.
Nature Genetics (2013)
Impairment of endothelium-dependent arterial relaxation by lysolecithin in modified low-density lipoproteins.
Kiyotaka Kugiyama;Scott A. Kerns;Joel D. Morrisett;Robert Roberts.
Thrombolysis inMyocardial Infarction (TIMI) Trial, Phase I:acomparison between intravenous tissue plasminogen activator andintravenous streptokinase*
J. H. Chesebro;G. Knatterud;R. Roberts;J. Borer.
It’s Time for a Change to a Troponin Standard
Allan S. Jaffe;Jan Ravkilde;Robert Roberts;Ulf Naslund.
A common allele on chromosome 9 associated with coronary heart disease
Ruth Mcpherson;Alexander Pertsemlidis;Nihan Kavaslar;Alexandre Stewart.
Obstetrical & Gynecological Survey (2007)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: