His primary areas of study are Immunology, Mycobacterium tuberculosis, Tuberculosis, Latent tuberculosis and Granuloma. His Immunology research includes themes of Pneumococcal conjugate vaccine, Streptococcus pneumoniae and Pneumococcal infections. His research ties Cytokine and Mycobacterium tuberculosis together.
His Tuberculosis study combines topics in areas such as Antigen and Immunity. His research integrates issues of Macrophage, Isoniazid, Disease and Virology in his study of Latent tuberculosis. His work carried out in the field of Granuloma brings together such families of science as Tumor necrosis factor alpha and Interferon gamma.
His scientific interests lie mostly in Tuberculosis, Immunology, Mycobacterium tuberculosis, Virology and Immune system. His Tuberculosis study integrates concerns from other disciplines, such as T cell, Immunity, Lung, Disease and Granuloma. In his research on the topic of Immunology, Spleen is strongly related with Bronchoalveolar lavage.
His research in Mycobacterium tuberculosis is mostly concerned with Latent tuberculosis. Philana Ling Lin interconnects Acquired immune system and Lymph in the investigation of issues within Virology. His Immune system research integrates issues from Antibody, Avidity and Lymph node.
Philana Ling Lin focuses on Tuberculosis, Mycobacterium tuberculosis, Immunology, Immune system and Cytotoxic T cell. His research in Tuberculosis intersects with topics in Multiple pathologies, Disease, Antibiotic exposure, Molecular imaging and Human pathogen. He focuses mostly in the field of Mycobacterium tuberculosis, narrowing it down to matters related to CD8 and, in some cases, Heterologous, Adjuvant, Cytokine and Peripheral blood mononuclear cell.
His Immunology research focuses on Immunity in particular. He studied Immune system and Granuloma that intersect with Interleukin 4. His T cell research incorporates elements of Latent tuberculosis, Simian immunodeficiency virus and Virology.
Philana Ling Lin spends much of his time researching Lung injury, Cell biology, Interferon-stimulated gene, Downregulation and upregulation and Interferon. His Lung injury research includes elements of Proteases, Cell culture and Single-cell analysis. His research on Cell biology often connects related topics like Receptor.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
SARS-CoV-2 Receptor ACE2 Is an Interferon-Stimulated Gene in Human Airway Epithelial Cells and Is Detected in Specific Cell Subsets across Tissues.
Carly G.K. Ziegler;Samuel J. Allon;Sarah K. Nyquist;Ian M. Mbano.
SARS-CoV-2 Receptor ACE2 is an Interferon-Stimulated Gene in Human Airway Epithelial Cells and Is Enriched in Specific Cell Subsets Across Tissues
Carly Ziegler;Samuel J. Allon;Sarah K. Nyquist;Ian Mbano.
Social Science Research Network (2020)
Understanding Latent Tuberculosis: A Moving Target
Philana Ling Lin;JoAnne L. Flynn.
Journal of Immunology (2010)
Experimental Mycobacterium tuberculosis infection of cynomolgus macaques closely resembles the various manifestations of human M. tuberculosis infection.
Saverio V. Capuano;Denise A. Croix;Santosh Pawar;Angelica Zinovik.
Infection and Immunity (2003)
Use of whole genome sequencing to estimate the mutation rate of Mycobacterium tuberculosis during latent infection
Christopher B Ford;Philana Ling Lin;Michael R Chase;Rupal R Shah.
Nature Genetics (2011)
Sterilization of granulomas is common in active and latent tuberculosis despite within-host variability in bacterial killing
Philana Ling Lin;Christopher B Ford;Christopher B Ford;M Teresa Coleman;Amy J Myers.
Nature Medicine (2014)
Quantitative Comparison of Active and Latent Tuberculosis in the Cynomolgus Macaque Model
Philana Ling Lin;Mark Rodgers;Le'kneitah Smith;Matthew Bigbee.
Infection and Immunity (2009)
Early trends for invasive pneumococcal infections in children after the introduction of the 13-valent pneumococcal conjugate vaccine
Sheldon L. Kaplan;William J. Barson;Philana Ling Lin;José R. Romero.
Pediatric Infectious Disease Journal (2013)
Tumor necrosis factor and chemokine interactions in the formation and maintenance of granulomas in tuberculosis.
Holly M. Scott Algood;Philana Ling Lin;JoAnne L. Flynn.
Clinical Infectious Diseases (2005)
Tumor necrosis factor neutralization results in disseminated disease in acute and latent Mycobacterium tuberculosis infection with normal granuloma structure in a cynomolgus macaque model
Philana Ling Lin;Amy Myers;Le'Kneitah Smith;Carolyn Bigbee.
Arthritis & Rheumatism (2010)
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