What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Mutation
Peter M. Andersen mainly investigates Amyotrophic lateral sclerosis, Genetics, Pathology, Superoxide dismutase and SOD1.
Amyotrophic lateral sclerosis is a subfield of Internal medicine that Peter M. Andersen explores.
Ischemia and Anatomy is closely connected to Endocrinology in his research, which is encompassed under the umbrella topic of Internal medicine.
He focuses mostly in the field of Pathology, narrowing it down to topics relating to Neurology and, in certain cases, Differential diagnosis, Gastroenterology and Predictive value of tests.
His Superoxide dismutase study combines topics from a wide range of disciplines, such as Mutant and Superoxide.
The concepts of his Genome-wide association study study are interwoven with issues in Genetic association and Locus.
His most cited work include:
- VEGF is a modifier of amyotrophic lateral sclerosis in mice and humans and protects motoneurons against ischemic death (768 citations)
- EFNS guidelines on the Clinical Management of Amyotrophic Lateral Sclerosis (MALS) : revised report of an EFNS task force (584 citations)
- ANG mutations segregate with familial and 'sporadic' amyotrophic lateral sclerosis (539 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Amyotrophic lateral sclerosis, Genetics, SOD1, Pathology and Gene.
His biological study spans a wide range of topics, including Mutation, Superoxide dismutase and Neurology.
Peter M. Andersen interconnects Immunology and Genetically modified mouse, Transgene in the investigation of issues within SOD1.
He has researched Pathology in several fields, including Biomarker and Spinal cord.
His Disease research includes themes of Asymptomatic and Bioinformatics.
Peter M. Andersen combines subjects such as Genetic association and Locus with his study of Genome-wide association study.
He most often published in these fields:
- Amyotrophic lateral sclerosis (83.16%)
- Genetics (45.61%)
- SOD1 (29.47%)
What were the highlights of his more recent work (between 2016-2021)?
- Amyotrophic lateral sclerosis (83.16%)
- Genetics (45.61%)
- SOD1 (29.47%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Amyotrophic lateral sclerosis, Genetics, SOD1, Neurology and Disease.
His study on Amyotrophic lateral sclerosis is covered under Pathology.
When carried out as part of a general Pathology research project, his work on Neurofilament light is frequently linked to work in In patient, therefore connecting diverse disciplines of study.
His SOD1 research integrates issues from Mutation and Pharmacology.
His Disease research is multidisciplinary, incorporating elements of Epidemiology and Bioinformatics.
His Superoxide dismutase research is multidisciplinary, relying on both Molecular biology, Genetically modified mouse, Astrocyte and Cell biology.
Between 2016 and 2021, his most popular works were:
- Genome-wide Analyses Identify KIF5A as a Novel ALS Gene (215 citations)
- Genome-Wide Analyses Identify KIF5A as a Novel ALS Gene (83 citations)
- Hot-spot KIF5A mutations cause familial ALS (81 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Mutation
Peter M. Andersen spends much of his time researching Amyotrophic lateral sclerosis, Neurology, Pathology, Gene and SOD1.
His Amyotrophic lateral sclerosis study is related to the wider topic of Disease.
The various areas that Peter M. Andersen examines in his Disease study include Prospective cohort study and Physical medicine and rehabilitation.
His study focuses on the intersection of Neurology and fields such as C9orf72 with connections in the field of Neurodegeneration, Medical genetics, DNA damage, Sarcoma and Cell biology.
His Gene study is concerned with the field of Genetics as a whole.
Peter M. Andersen has included themes like Molecular biology, Tolerability and Spinal cord in his SOD1 study.
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