His scientific interests lie mostly in Immunology, Molecular biology, Cytotoxic T cell, Cytokine and CD8. His Immunology study combines topics from a wide range of disciplines, such as Cancer research and Myofibroblast. His Molecular biology research is multidisciplinary, incorporating perspectives in Apoptosis, Ex vivo and Jurkat cells.
His Cytotoxic T cell research is multidisciplinary, relying on both T cell and T lymphocyte. Peter D. Katsikis has researched T cell in several fields, including Fas receptor and Cell biology. His research in Cytokine intersects with topics in Tumor necrosis factor alpha and Synovial membrane.
His primary areas of investigation include Immunology, Cytotoxic T cell, CD8, T cell and Virology. His study in Cytokine, Immune system, Rheumatoid arthritis, Inflammation and Tumor necrosis factor alpha falls under the purview of Immunology. His work deals with themes such as Cancer research and Cell biology, which intersect with Cytotoxic T cell.
He combines subjects such as Molecular biology, T-cell receptor and Fas receptor with his study of CD8. His Molecular biology study which covers Programmed cell death that intersects with Annexin and In vivo. His Interleukin 21 research includes elements of CD40 and Antigen-presenting cell.
Peter D. Katsikis mainly focuses on Cell biology, Immune system, Immunology, Cytotoxic T cell and CD8. In his work, Intracellular, Apoptosis, Programmed cell death, Cell culture and Downregulation and upregulation is strongly intertwined with RNA, which is a subfield of Cell biology. The concepts of his Immunology study are interwoven with issues in Lung and Respiratory system.
His Cytotoxic T cell research incorporates themes from T cell, Regulation of gene expression, microRNA and Chronic infection. His T cell study also includes fields such as
Peter D. Katsikis focuses on Cell biology, Stem cell, Influenza A virus, Virus and A549 cell. His research integrates issues of microRNA, CD8, Lymphocytic choriomeningitis and Cellular differentiation in his study of Cell biology. His study in Stem cell is interdisciplinary in nature, drawing from both Interferon, Transcriptome and Wnt signaling pathway.
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Treatment of rheumatoid arthritis with chimeric monoclonal antibodies to tumor necrosis factor alpha
M J Elliott;R N Maini;M Feldmann;A Long-Fox.
Arthritis & Rheumatism (1993)
In vivo detection and imaging of phosphatidylserine expression during programmed cell death
F G Blankenberg;P D Katsikis;J F Tait;R E Davis.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Immunoregulatory role of interleukin 10 in rheumatoid arthritis.
Peter D. Katsikis;Cong Qiu Chu;Fionula M. Brennan;Ravinder N. Maini.
Journal of Experimental Medicine (1994)
Fas antigen stimulation induces marked apoptosis of T lymphocytes in human immunodeficiency virus-infected individuals.
P D Katsikis;E S Wunderlich;C A Smith;L A Herzenberg.
Journal of Experimental Medicine (1995)
Interleukin-10 inhibition of the progression of established collagen-induced arthritis.
Marita Walmsley;Peter D. Katsikis;Erika Abney;Sarah Parry.
Arthritis & Rheumatism (1996)
The microRNA miR-155 controls CD8 + T cell responses by regulating interferon signaling
Donald T Gracias;Erietta Stelekati;Jennifer L Hope;Alina C Boesteanu.
Nature Immunology (2013)
Imaging of Apoptosis (Programmed Cell Death) with 99mTc Annexin V
Francis G. Blankenberg;Peter D. Katsikis;Jonathan F. Tait;Richard Eric Davis.
The Journal of Nuclear Medicine (1999)
Interleukin-1β Converting Enzyme–like Protease Involvement in Fas-induced and Activation-induced Peripheral Blood T Cell Apoptosis in HIV Infection. TNF-related Apoptosis-inducing Ligand Can Mediate Activation-induced T Cell Death in HIV Infection
P D Katsikis;M E Garcia-Ojeda;J F Torres-Roca;I M Tijoe.
Journal of Experimental Medicine (1997)
Surface expression patterns of negative regulatory molecules identify determinants of virus-specific CD8+ T-cell exhaustion in HIV infection
Takuya Yamamoto;David A. Price;David A. Price;Joseph P. Casazza;Guido Ferrari.
Induction of the interleukin 1 receptor antagonist protein by transforming growth factor-beta.
M Turner;D Chantry;P Katsikis;A Berger.
European Journal of Immunology (1991)
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