Muniswamy Madesh mostly deals with Cell biology, Mitochondrion, Calcium, Apoptosis and Endoplasmic reticulum. His Cell biology study integrates concerns from other disciplines, such as Programmed cell death and Voltage-dependent calcium channel. His Mitochondrion research integrates issues from Superoxide, Homeostasis and Mitochondrial permeability transition pore.
His research integrates issues of Endocrinology and Receptor antagonist in his study of Calcium. His Apoptosis research includes themes of Necrosis, Pathology and Cytosol. Muniswamy Madesh combines subjects such as Stromal cell, Cell calcium and Membrane with his study of Endoplasmic reticulum.
Muniswamy Madesh mainly investigates Cell biology, Mitochondrion, Biochemistry, Internal medicine and Calcium. The study incorporates disciplines such as Apoptosis, Programmed cell death and Mitochondrial permeability transition pore in addition to Cell biology. His Mitochondrion research is multidisciplinary, relying on both Uniporter and Reactive oxygen species.
His research investigates the connection between Reactive oxygen species and topics such as Superoxide that intersect with problems in Superoxide dismutase. His work carried out in the field of Internal medicine brings together such families of science as Endocrinology and Cardiology. In his study, which falls under the umbrella issue of Calcium signaling, Cytosol is strongly linked to Endoplasmic reticulum.
Muniswamy Madesh mainly focuses on Cell biology, Mitochondrion, Calcium, Internal medicine and Biochemistry. Cell biology connects with themes related to Membrane potential in his study. His Mitochondrion research is multidisciplinary, incorporating elements of Homeostasis, Uniporter, Programmed cell death, Mitochondrial permeability transition pore and Contractility.
As part of the same scientific family, Muniswamy Madesh usually focuses on Calcium, concentrating on Intracellular and intersecting with Mitochondrial ROS and Inner mitochondrial membrane. Muniswamy Madesh works mostly in the field of Internal medicine, limiting it down to topics relating to Endocrinology and, in certain cases, Receptor, Agonist, Rosiglitazone and Mitochondrial biogenesis. His studies deal with areas such as Endoplasmic reticulum and Cytosol as well as Calcium signaling.
His main research concerns Mitochondrion, Cell biology, Programmed cell death, Calcium and Mitochondrial permeability transition pore. He studied Mitochondrion and Cytosol that intersect with Mitochondrial fission, Biophysics, EF hand and Mitophagy. His Cell biology research incorporates elements of Uniporter and Oxidative phosphorylation, Biochemistry.
His Uniporter study incorporates themes from Reactive oxygen species and Bioenergetics. His study in the fields of PPIF under the domain of Programmed cell death overlaps with other disciplines such as Voltage-dependent anion channel. His Mitochondrial permeability transition pore research incorporates themes from Homeostasis and Voltage-dependent calcium channel.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
MICU1 is an essential gatekeeper for MCU-mediated mitochondrial Ca(2+) uptake that regulates cell survival.
Karthik Mallilankaraman;Patrick Doonan;César Cárdenas;César Cárdenas;Harish C. Chandramoorthy.
STIM proteins: dynamic calcium signal transducers
Jonathan Soboloff;Brad S. Rothberg;Muniswamy Madesh;Donald L. Gill.
Nature Reviews Molecular Cell Biology (2012)
Calcium signaling and apoptosis.
György Hajnóczky;Erika Davies;Muniswamy Madesh.
Biochemical and Biophysical Research Communications (2003)
VDAC-dependent permeabilization of the outer mitochondrial membrane by superoxide induces rapid and massive cytochrome c release
Muniswamy Madesh;György Hajnóczky.
Journal of Cell Biology (2001)
Microtiter plate assay for superoxide dismutase using MTT reduction by superoxide.
M Madesh;K A Balasubramanian.
Indian Journal of Biochemistry & Biophysics (1998)
MCUR1 is an essential component of mitochondrial Ca2+ uptake that regulates cellular metabolism
Karthik Mallilankaraman;César Cárdenas;Patrick J. Doonan;Harish C. Chandramoorthy.
Nature Cell Biology (2013)
The endoplasmic reticulum gateway to apoptosis by Bcl-XL modulation of the InsP3R
Carl White;Chi Li;Jun Yang;Nataliya B. Petrenko.
Nature Cell Biology (2005)
Simultaneous detection of apoptosis and mitochondrial superoxide production in live cells by flow cytometry and confocal microscopy.
Partha Mukhopadhyay;Mohanraj Rajesh;György Haskó;Brian J Hawkins.
Nature Protocols (2007)
The Mitochondrial Calcium Uniporter Matches Energetic Supply with Cardiac Workload during Stress and Modulates Permeability Transition
Timothy S. Luongo;Jonathan P. Lambert;Ancai Yuan;Xueqian Zhang.
Cell Reports (2015)
The mitochondrial Na+/Ca2+ exchanger is essential for Ca2+ homeostasis and viability.
Timothy S. Luongo;Jonathan P. Lambert;Polina Gross;Mary Nwokedi.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: