What is he best known for?
The fields of study he is best known for:
- Enzyme
- Apoptosis
- Internal medicine
Muniswamy Madesh mostly deals with Cell biology, Mitochondrion, Calcium, Apoptosis and Endoplasmic reticulum.
His Cell biology study integrates concerns from other disciplines, such as Programmed cell death and Voltage-dependent calcium channel.
His Mitochondrion research integrates issues from Superoxide, Homeostasis and Mitochondrial permeability transition pore.
His research integrates issues of Endocrinology and Receptor antagonist in his study of Calcium.
His Apoptosis research includes themes of Necrosis, Pathology and Cytosol.
Muniswamy Madesh combines subjects such as Stromal cell, Cell calcium and Membrane with his study of Endoplasmic reticulum.
His most cited work include:
- STIM proteins: dynamic calcium signal transducers (454 citations)
- VDAC-dependent permeabilization of the outer mitochondrial membrane by superoxide induces rapid and massive cytochrome c release (438 citations)
- MICU1 is an essential gatekeeper for MCU-mediated mitochondrial Ca(2+) uptake that regulates cell survival. (415 citations)
What are the main themes of his work throughout his whole career to date?
Muniswamy Madesh mainly investigates Cell biology, Mitochondrion, Biochemistry, Internal medicine and Calcium.
The study incorporates disciplines such as Apoptosis, Programmed cell death and Mitochondrial permeability transition pore in addition to Cell biology.
His Mitochondrion research is multidisciplinary, relying on both Uniporter and Reactive oxygen species.
His research investigates the connection between Reactive oxygen species and topics such as Superoxide that intersect with problems in Superoxide dismutase.
His work carried out in the field of Internal medicine brings together such families of science as Endocrinology and Cardiology.
In his study, which falls under the umbrella issue of Calcium signaling, Cytosol is strongly linked to Endoplasmic reticulum.
He most often published in these fields:
- Cell biology (59.26%)
- Mitochondrion (33.33%)
- Biochemistry (20.74%)
What were the highlights of his more recent work (between 2014-2020)?
- Cell biology (59.26%)
- Mitochondrion (33.33%)
- Calcium (20.00%)
In recent papers he was focusing on the following fields of study:
Muniswamy Madesh mainly focuses on Cell biology, Mitochondrion, Calcium, Internal medicine and Biochemistry.
Cell biology connects with themes related to Membrane potential in his study.
His Mitochondrion research is multidisciplinary, incorporating elements of Homeostasis, Uniporter, Programmed cell death, Mitochondrial permeability transition pore and Contractility.
As part of the same scientific family, Muniswamy Madesh usually focuses on Calcium, concentrating on Intracellular and intersecting with Mitochondrial ROS and Inner mitochondrial membrane.
Muniswamy Madesh works mostly in the field of Internal medicine, limiting it down to topics relating to Endocrinology and, in certain cases, Receptor, Agonist, Rosiglitazone and Mitochondrial biogenesis.
His studies deal with areas such as Endoplasmic reticulum and Cytosol as well as Calcium signaling.
Between 2014 and 2020, his most popular works were:
- The Mitochondrial Calcium Uniporter Matches Energetic Supply with Cardiac Workload during Stress and Modulates Permeability Transition (198 citations)
- The mitochondrial Na+/Ca2+ exchanger is essential for Ca2+ homeostasis and viability. (166 citations)
- SPG7 Is an Essential and Conserved Component of the Mitochondrial Permeability Transition Pore. (114 citations)
In his most recent research, the most cited papers focused on:
- Enzyme
- Apoptosis
- Internal medicine
His main research concerns Mitochondrion, Cell biology, Programmed cell death, Calcium and Mitochondrial permeability transition pore.
He studied Mitochondrion and Cytosol that intersect with Mitochondrial fission, Biophysics, EF hand and Mitophagy.
His Cell biology research incorporates elements of Uniporter and Oxidative phosphorylation, Biochemistry.
His Uniporter study incorporates themes from Reactive oxygen species and Bioenergetics.
His study in the fields of PPIF under the domain of Programmed cell death overlaps with other disciplines such as Voltage-dependent anion channel.
His Mitochondrial permeability transition pore research incorporates themes from Homeostasis and Voltage-dependent calcium channel.
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