1982 - Fellow of the American Association for the Advancement of Science (AAAS)
Fellow of The Academy of Medical Sciences, United Kingdom
Mervyn Maze mainly investigates Anesthesia, Dexmedetomidine, Pharmacology, Anesthetic and Sedative. Mervyn Maze has researched Anesthesia in several fields, including Randomized controlled trial, Postoperative cognitive dysfunction and Delirium. His studies deal with areas such as Agonist, Endocrinology, Medetomidine and Internal medicine as well as Dexmedetomidine.
His Pharmacology study integrates concerns from other disciplines, such as Adrenergic receptor, Mechanism of action, Glutamate receptor, Antagonist and Pathology. His research integrates issues of Nitrous oxide and Neurocognitive Dysfunction in his study of Anesthetic. His work deals with themes such as Hypnotic, Environmental air flow and Sympatholytic, which intersect with Sedative.
Mervyn Maze mostly deals with Anesthesia, Pharmacology, Dexmedetomidine, Internal medicine and Endocrinology. His research in Anesthesia intersects with topics in Randomized controlled trial, Neuroprotection and Nociception. His work in Pharmacology addresses issues such as Adrenergic receptor, which are connected to fields such as Alpha.
His research on Dexmedetomidine also deals with topics like
Anesthesia, Cognitive decline, Perioperative, Intensive care medicine and Randomized controlled trial are his primary areas of study. His study in Dexmedetomidine and Anesthetic falls under the purview of Anesthesia. His Dexmedetomidine study is related to the wider topic of Pharmacology.
His studies in Cognitive decline integrate themes in fields like Inflammation, Proinflammatory cytokine, Neuroinflammation and Metabolic syndrome. His Inflammation study incorporates themes from Hippocampal formation, Endocrinology and Pathology. His work in Randomized controlled trial tackles topics such as Neuroprotection which are related to areas like Neurologic injury.
Mervyn Maze focuses on Anesthesia, Cognitive decline, Randomized controlled trial, Postoperative cognitive dysfunction and Surgery. His Anesthesia research incorporates elements of Analysis of variance, Clinical trial and Metabolic syndrome. His study in Cognitive decline is interdisciplinary in nature, drawing from both Hippocampal formation, Endocrinology, Inflammation and Innate immune system.
His Randomized controlled trial research is multidisciplinary, incorporating perspectives in Inhalation, Dexmedetomidine, Intensive care and Out of hospital cardiac arrest. His work in Dexmedetomidine covers topics such as Neurologic injury which are related to areas like Neuroprotection. His work on Methyllycaconitine, Oxidative stress, Neuroinflammation and Agonist is typically connected to Nicotinamide adenine dinucleotide phosphate as part of general Internal medicine study, connecting several disciplines of science.
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Effect of Sedation With Dexmedetomidine vs Lorazepam on Acute Brain Dysfunction in Mechanically Ventilated Patients: The MENDS Randomized Controlled Trial
Pratik P. Pandharipande;Brenda T. Pun;Daniel L. Herr;Mervyn Maze.
The alpha2-adrenoceptor agonist dexmedetomidine converges on an endogenous sleep-promoting pathway to exert its sedative effects.
Laura E. Nelson;Jun Lu;Tianzhi Guo;Clifford B. Saper.
Alpha-2 adrenoceptor agonists: defining the role in clinical anesthesia.
Mervyn Maze;William Tranquilli.
Effects of intravenous dexmedetomidine in humans. II. Hemodynamic changes.
Byron C. Bloor;Denham S. Ward;Jon P. Belleville;Mervyn Maze.
Clinical Uses of α2-Adrenergic Agonists
Takahiko Kamibayashi;Mervyn Maze.
Postoperative cognitive dysfunction after noncardiac surgery: a systematic review.
Stanton Newman;Jan Stygall;Shashivadan Hirani;Shahzad Shaefi.
Role of interleukin-1beta in postoperative cognitive dysfunction.
Mario Cibelli;Antonio Rei Fidalgo;Niccolò Terrando;Niccolò Terrando;Daqing Ma.
Annals of Neurology (2010)
Effects of intravenous dexmedetomidine in humans. I. Sedation, ventilation, and metabolic rate.
Jon P. Belleville;Denham S. Ward;Byron C. Bloor;Mervyn Maze.
Tumor necrosis factor-alpha triggers a cytokine cascade yielding postoperative cognitive decline.
Niccolò Terrando;Claudia Monaco;Daqing Ma;Brian M. J. Foxwell.
Proceedings of the National Academy of Sciences of the United States of America (2010)
The sedative component of anesthesia is mediated by GABA(A) receptors in an endogenous sleep pathway.
L. E. Nelson;T. Z. Guo;J. Lu;C. B. Saper.
Nature Neuroscience (2002)
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