His primary areas of study are Molecular biology, Cancer, Genetics, Cancer research and DNA. His Molecular biology study incorporates themes from Allosteric regulation, Homologous recombination and DNA replication. His work in the fields of Non-homologous end joining overlaps with other areas such as PARP inhibitor.
His Cancer research incorporates elements of Downregulation and upregulation, Oncology and Pathology. His research in Cancer research intersects with topics in Gene expression profiling, Transitional cell carcinoma, Microsatellite instability, DNA mismatch repair and Carcinoma. His studies deal with areas such as Wild type and Gene, Locus, Nucleotide as well as DNA.
Molecular biology, Genetics, Gene, Cancer research and DNA are his primary areas of study. His study in Molecular biology is interdisciplinary in nature, drawing from both Chinese hamster ovary cell, Mutant, DNA repair, Adenine phosphoribosyltransferase and DNA replication. While the research belongs to areas of Chinese hamster ovary cell, Mark Meuth spends his time largely on the problem of Biochemistry, intersecting his research to questions surrounding Fibroblast.
His research on DNA repair frequently links to adjacent areas such as Homologous recombination. The study incorporates disciplines such as Cell cycle checkpoint, Transitional cell carcinoma, Pathology and Colorectal cancer, DNA mismatch repair in addition to Cancer research. His DNA study combines topics in areas such as Mutation, Frameshift mutation, Gene rearrangement and Hamster.
Mark Meuth mostly deals with Cancer research, DNA replication, Cell biology, Pathology and Molecular biology. His DNA replication research incorporates themes from Apoptosis and DNA damage. His research integrates issues of CHEK1, Embryonic stem cell, Aurora Kinase A and Control of chromosome duplication in his study of Cell biology.
His Pathology research includes elements of Prostate, DNA methylation, Microsatellite instability, Urinary system and Epigenetics. Mark Meuth has researched Molecular biology in several fields, including Homologous recombination, Sequence analysis, Repeated sequence and DNA repair. His work on RAD51, DNA Repair Inhibition and Homologous Recombination Pathway as part of general DNA repair study is frequently connected to Poly Polymerase Inhibitor and PARP1, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
His primary areas of study are DNA methylation, Pathology, DNA re-replication, Cell biology and DNA replication. His DNA methylation research is multidisciplinary, relying on both Bladder cancer, Transitional cell carcinoma, Cancer research, Carcinoma and Tumor progression. The various areas that Mark Meuth examines in his Cancer research study include Carcinoma in situ, Methylation and Cytokeratin.
The Pathology study combines topics in areas such as Internal medicine and Urinary system. His DNA re-replication research is multidisciplinary, incorporating elements of CHEK1, DNA replication factor CDT1 and DNA Replication Fork. As part of one scientific family, Mark Meuth deals mainly with the area of Control of chromosome duplication, narrowing it down to issues related to the Molecular biology, and often DNA repair.
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Specific killing of BRCA2-deficient tumours with inhibitors of poly(ADP-ribose) polymerase
Helen E. Bryant;Nilklas Schultz;Huw D. Thomas;Kayan M. Parker.
An established pre-adipose cell line and its differentiation in culture
Howard Green;Mark Meuth.
Mutator phenotypes in human colorectal carcinoma cell lines
Nitai P. Bhattacharyya;Adonis Skandalis;Anil Ganesh;Joanna Groden.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Distinct MicroRNA Alterations Characterize High- and Low-Grade Bladder Cancer
James W.F. Catto;Saiful Miah;Helen C. Owen;Helen Bryant.
Cancer Research (2009)
Different roles for nonhomologous end joining and homologous recombination following replication arrest in mammalian cells.
Cecilia Lundin;Klaus Erixon;Catherine Arnaudeau;Niklas Schultz.
Molecular and Cellular Biology (2002)
Promoter Hypermethylation Is Associated With Tumor Location, Stage, and Subsequent Progression in Transitional Cell Carcinoma
James W.F. Catto;Abdel-Rahmene Azzouzi;Ishtiaq Rehman;Kenneth M. Feeley.
Journal of Clinical Oncology (2005)
The molecular basis of mutations induced by deoxyribonucleoside triphosphate pool imbalances in mammalian cells.
Experimental Cell Research (1989)
Mapping replication units in animal cells
Shlomo Handeli;Avihu Klar;Mark Meuth;Howard Cedar.
Induction of a deoxycytidineless state in cultured mammalian cells by bromodeoxyuridine
Mark Meuth;Howard Green.
Apoptosis Induced by Overexpression of hMSH2 or hMLH1
Hong Zhang;Burt Richards;Teresa Wilson;Michael Lloyd.
Cancer Research (1999)
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