D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Genetics and Molecular Biology D-index 48 Citations 12,336 78 World Ranking 4015 National Ranking 428

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Mutation

His primary areas of study are Molecular biology, Cancer, Genetics, Cancer research and DNA. His Molecular biology study incorporates themes from Allosteric regulation, Homologous recombination and DNA replication. His work in the fields of Non-homologous end joining overlaps with other areas such as PARP inhibitor.

His Cancer research incorporates elements of Downregulation and upregulation, Oncology and Pathology. His research in Cancer research intersects with topics in Gene expression profiling, Transitional cell carcinoma, Microsatellite instability, DNA mismatch repair and Carcinoma. His studies deal with areas such as Wild type and Gene, Locus, Nucleotide as well as DNA.

His most cited work include:

  • Specific killing of BRCA2-deficient tumours with inhibitors of poly(ADP-ribose) polymerase (3289 citations)
  • An established pre-adipose cell line and its differentiation in culture (913 citations)
  • Mutator phenotypes in human colorectal carcinoma cell lines (382 citations)

What are the main themes of his work throughout his whole career to date?

Molecular biology, Genetics, Gene, Cancer research and DNA are his primary areas of study. His study in Molecular biology is interdisciplinary in nature, drawing from both Chinese hamster ovary cell, Mutant, DNA repair, Adenine phosphoribosyltransferase and DNA replication. While the research belongs to areas of Chinese hamster ovary cell, Mark Meuth spends his time largely on the problem of Biochemistry, intersecting his research to questions surrounding Fibroblast.

His research on DNA repair frequently links to adjacent areas such as Homologous recombination. The study incorporates disciplines such as Cell cycle checkpoint, Transitional cell carcinoma, Pathology and Colorectal cancer, DNA mismatch repair in addition to Cancer research. His DNA study combines topics in areas such as Mutation, Frameshift mutation, Gene rearrangement and Hamster.

He most often published in these fields:

  • Molecular biology (46.85%)
  • Genetics (41.44%)
  • Gene (32.43%)

What were the highlights of his more recent work (between 2004-2016)?

  • Cancer research (22.52%)
  • DNA replication (19.82%)
  • Cell biology (16.22%)

In recent papers he was focusing on the following fields of study:

Mark Meuth mostly deals with Cancer research, DNA replication, Cell biology, Pathology and Molecular biology. His DNA replication research incorporates themes from Apoptosis and DNA damage. His research integrates issues of CHEK1, Embryonic stem cell, Aurora Kinase A and Control of chromosome duplication in his study of Cell biology.

His Pathology research includes elements of Prostate, DNA methylation, Microsatellite instability, Urinary system and Epigenetics. Mark Meuth has researched Molecular biology in several fields, including Homologous recombination, Sequence analysis, Repeated sequence and DNA repair. His work on RAD51, DNA Repair Inhibition and Homologous Recombination Pathway as part of general DNA repair study is frequently connected to Poly Polymerase Inhibitor and PARP1, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.

Between 2004 and 2016, his most popular works were:

  • Specific killing of BRCA2-deficient tumours with inhibitors of poly(ADP-ribose) polymerase (3289 citations)
  • Distinct MicroRNA Alterations Characterize High- and Low-Grade Bladder Cancer (263 citations)
  • Promoter Hypermethylation Is Associated With Tumor Location, Stage, and Subsequent Progression in Transitional Cell Carcinoma (256 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Mutation

His primary areas of study are DNA methylation, Pathology, DNA re-replication, Cell biology and DNA replication. His DNA methylation research is multidisciplinary, relying on both Bladder cancer, Transitional cell carcinoma, Cancer research, Carcinoma and Tumor progression. The various areas that Mark Meuth examines in his Cancer research study include Carcinoma in situ, Methylation and Cytokeratin.

The Pathology study combines topics in areas such as Internal medicine and Urinary system. His DNA re-replication research is multidisciplinary, incorporating elements of CHEK1, DNA replication factor CDT1 and DNA Replication Fork. As part of one scientific family, Mark Meuth deals mainly with the area of Control of chromosome duplication, narrowing it down to issues related to the Molecular biology, and often DNA repair.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Specific killing of BRCA2-deficient tumours with inhibitors of poly(ADP-ribose) polymerase

Helen E. Bryant;Nilklas Schultz;Huw D. Thomas;Kayan M. Parker.
Nature (2005)

4295 Citations

An established pre-adipose cell line and its differentiation in culture

Howard Green;Mark Meuth.
Cell (1974)

1380 Citations

Mutator phenotypes in human colorectal carcinoma cell lines

Nitai P. Bhattacharyya;Adonis Skandalis;Anil Ganesh;Joanna Groden.
Proceedings of the National Academy of Sciences of the United States of America (1994)

555 Citations

Distinct MicroRNA Alterations Characterize High- and Low-Grade Bladder Cancer

James W.F. Catto;Saiful Miah;Helen C. Owen;Helen Bryant.
Cancer Research (2009)

378 Citations

Different roles for nonhomologous end joining and homologous recombination following replication arrest in mammalian cells.

Cecilia Lundin;Klaus Erixon;Catherine Arnaudeau;Niklas Schultz.
Molecular and Cellular Biology (2002)

326 Citations

Promoter Hypermethylation Is Associated With Tumor Location, Stage, and Subsequent Progression in Transitional Cell Carcinoma

James W.F. Catto;Abdel-Rahmene Azzouzi;Ishtiaq Rehman;Kenneth M. Feeley.
Journal of Clinical Oncology (2005)

315 Citations

The molecular basis of mutations induced by deoxyribonucleoside triphosphate pool imbalances in mammalian cells.

Mark Meuth.
Experimental Cell Research (1989)

276 Citations

Mapping replication units in animal cells

Shlomo Handeli;Avihu Klar;Mark Meuth;Howard Cedar.
Cell (1989)

245 Citations

Induction of a deoxycytidineless state in cultured mammalian cells by bromodeoxyuridine

Mark Meuth;Howard Green.
Cell (1974)

242 Citations

Apoptosis Induced by Overexpression of hMSH2 or hMLH1

Hong Zhang;Burt Richards;Teresa Wilson;Michael Lloyd.
Cancer Research (1999)

229 Citations

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