D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 51 Citations 7,869 131 World Ranking 9975 National Ranking 795

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • DNA

The scientist’s investigation covers issues in Virology, Hepatitis C virus, NS5A, Viral replication and Virus. Mark Harris interconnects Mutation, Myristoylation and Biochemistry in the investigation of issues within Virology. As a part of the same scientific study, Mark Harris usually deals with the Hepatitis C virus, concentrating on In vitro and frequently concerns with Amantadine.

His biological study spans a wide range of topics, including Cancer research, NS5B, PI3K/AKT/mTOR pathway, Phosphoinositide 3-kinase and Molecular biology. Mark Harris has included themes like RNA, Signal transduction and AP-1 transcription factor in his Molecular biology study. His research in Virus tackles topics such as Replicon which are related to areas like Intracellular, Bafilomycin and Interferon.

His most cited work include:

  • The p7 protein of hepatitis C virus forms an ion channel that is blocked by the antiviral drug, Amantadine. (429 citations)
  • Hepatitis C virus NS5A: tales of a promiscuous protein. (336 citations)
  • The Hepatitis C Virus NS5A Protein Activates a Phosphoinositide 3-Kinase-dependent Survival Signaling Cascade (193 citations)

What are the main themes of his work throughout his whole career to date?

Virology, Virus, Hepatitis C virus, Viral replication and NS5A are his primary areas of study. His Virology research incorporates themes from Molecular biology, Replicon and Subgenomic mRNA. The concepts of his Virus study are interwoven with issues in Gene and Cell biology.

His research on Hepatitis C virus also deals with topics like

  • In vitro which connect with In vivo,
  • Ion channel which is related to area like Amantadine. Mark Harris does research in Viral replication, focusing on Viral life cycle specifically. His work in NS5A covers topics such as Phosphorylation which are related to areas like Kinase and Mutation.

He most often published in these fields:

  • Virology (63.69%)
  • Virus (33.33%)
  • Hepatitis C virus (30.36%)

What were the highlights of his more recent work (between 2017-2021)?

  • Virology (63.69%)
  • Virus (33.33%)
  • Replicon (18.45%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Virology, Virus, Replicon, Hepatitis C virus and Viral replication. In the subject of general Virology, his work in Viral entry is often linked to Context, thereby combining diverse domains of study. His Virus research is multidisciplinary, relying on both RNA and Gene.

The Replicon study combines topics in areas such as RNA-dependent RNA polymerase and Foot-and-mouth disease virus. His work on NS5A as part of general Hepatitis C virus study is frequently linked to Antiviral therapy, therefore connecting diverse disciplines of science. His Viral replication study deals with In vitro intersecting with Anthranilic acid, Zika virus and RNA silencing.

Between 2017 and 2021, his most popular works were:

  • A role for domain I of the hepatitis C virus NS5A protein in virus assembly. (23 citations)
  • Technical and Imaging Outcomes from the UK Registry of Prostate Artery Embolization (UK-ROPE) Study: Focusing on Predictors of Clinical Success (22 citations)
  • Factors Predicting a Good Symptomatic Outcome After Prostate Artery Embolisation (PAE). (22 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • DNA

Mark Harris spends much of his time researching Virology, Virus, Replicon, Embolization and Viral replication. His Virology research includes elements of Luciferase and Pathogen. His research in Replicon intersects with topics in Internal ribosome entry site and Foot-and-mouth disease virus, Aphthovirus.

His study in Viral replication is interdisciplinary in nature, drawing from both RNA and Hepatitis C virus. While the research belongs to areas of RNA, Mark Harris spends his time largely on the problem of Cell biology, intersecting his research to questions surrounding NS5A. His work carried out in the field of Hepatitis C virus brings together such families of science as Phenotype and Molecular biology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The p7 protein of hepatitis C virus forms an ion channel that is blocked by the antiviral drug, Amantadine.

Stephen D.C. Griffin;Lucy P. Beales;Dean S. Clarke;Oliver Worsfold.
FEBS Letters (2003)

666 Citations

Hepatitis C virus NS5A: tales of a promiscuous protein.

Andrew Macdonald;Mark Harris.
Journal of General Virology (2004)

487 Citations

The Hepatitis C Virus NS5A Protein Activates a Phosphoinositide 3-Kinase-dependent Survival Signaling Cascade

Andrew Street;Andrew Macdonald;Katherine Crowder;Mark Harris.
Journal of Biological Chemistry (2004)

265 Citations

A conserved basic loop in hepatitis C virus p7 protein is required for amantadine-sensitive ion channel activity in mammalian cells but is dispensable for localization to mitochondria.

Stephen D. C. Griffin;Ruth Harvey;Dean S. Clarke;Wendy S. Barclay.
Journal of General Virology (2004)

225 Citations

Intracellular Proton Conductance of the Hepatitis C Virus p7 Protein and Its Contribution to Infectious Virus Production

Ann L. Wozniak;Stephen Griffin;Stephen Griffin;David Rowlands;Mark Harris.
PLOS Pathogens (2010)

212 Citations

Physical and Functional Interaction of Nef with Lck HIV-1 Nef-INDUCED T-CELL SIGNALING DEFECTS

Y. Collette;H. Dutartre;A. Benziane;F. Ramos-Morales.
Journal of Biological Chemistry (1996)

187 Citations

Identification of the Nef-associated kinase as p21-activated kinase 2.

G.Herma Renkema;Aki Manninen;Derek A. Mann;Mark Harris.
Current Biology (1999)

185 Citations

Construction of genetically engineered baculovirus insecticides containing the Bacillus thuringiensis subsp. kurstaki HD-73 delta endotoxin.

Alison T. Merryweather;Ulrike Weyer;Mark P. G. Harris;Mark Hirst.
Journal of General Virology (1990)

184 Citations

The hepatitis C virus non-structural NS5A protein inhibits activating protein-1 function by perturbing ras-ERK pathway signaling

Andrew Macdonald;Katherine Crowder;Andrew Street;Christopher McCormick.
Journal of Biological Chemistry (2003)

184 Citations

HIV-1 Nef Induces the Release of Inflammatory Factors from Human Monocyte/Macrophages: Involvement of Nef Endocytotic Signals and NF-κB Activation

Eleonora Olivetta;Zulema Percario;Gianna Fiorucci;Gianfranco Mattia;Gianfranco Mattia.
Journal of Immunology (2003)

176 Citations

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