H-Index & Metrics Best Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Medicine D-index 71 Citations 17,689 231 World Ranking 14628 National Ranking 7616

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Internal medicine

Kevin Shannon spends much of his time researching Cancer research, Leukemia, Neurofibromin 1, Juvenile myelomonocytic leukemia and Myeloid. His Cancer research study combines topics from a wide range of disciplines, such as Haematopoiesis, Stem cell, KRAS, PTPN11 and Signal transduction. His Leukemia research is within the category of Immunology.

His Neurofibromin 1 research includes elements of Tumor suppressor gene and Allele. In most of his Juvenile myelomonocytic leukemia studies, his work intersects topics such as Mutation. His Myeloid research includes themes of Myelogenous, Myeloproliferative Disorders and Bone marrow.

His most cited work include:

  • Hyperactive Ras in developmental disorders and cancer. (1235 citations)
  • Germline KRAS mutations cause Noonan syndrome (593 citations)
  • The Rate of Telomere Sequence Loss in Human Leukocytes Varies with Age (529 citations)

What are the main themes of his work throughout his whole career to date?

Kevin Shannon focuses on Cancer research, Myeloid, Leukemia, Immunology and Internal medicine. His Cancer research research incorporates elements of Haematopoiesis, Stem cell, KRAS, Neuroblastoma RAS viral oncogene homolog and Signal transduction. His Haematopoiesis research is multidisciplinary, relying on both Progenitor cell and MAPK/ERK pathway.

In Myeloid, Kevin Shannon works on issues like Bone marrow, which are connected to Somatic cell. Kevin Shannon has researched Leukemia in several fields, including Juvenile myelomonocytic leukemia, Myeloid leukemia, MEK inhibitor and Neurofibromin 1. His Immunology study frequently draws connections to adjacent fields such as Neurofibromatosis.

He most often published in these fields:

  • Cancer research (40.43%)
  • Myeloid (26.54%)
  • Leukemia (28.40%)

What were the highlights of his more recent work (between 2014-2021)?

  • Cancer research (40.43%)
  • Internal medicine (24.38%)
  • Cardiology (15.43%)

In recent papers he was focusing on the following fields of study:

Kevin Shannon mainly focuses on Cancer research, Internal medicine, Cardiology, Myeloid and Myeloid leukemia. The Cancer research study combines topics in areas such as Cancer, Leukemia, Immunology and KRAS, Neuroblastoma RAS viral oncogene homolog. His work in KRAS tackles topics such as Carcinogenesis which are related to areas like Allele.

As part of the same scientific family, Kevin Shannon usually focuses on Internal medicine, concentrating on Surgery and intersecting with Fontan procedure. His research in Myeloid focuses on subjects like Haematopoiesis, which are connected to Progenitor cell. His research on Myeloid leukemia also deals with topics like

  • Somatic cell that intertwine with fields like Chromosome 7,
  • Germline which is related to area like Germline mutation.

Between 2014 and 2021, his most popular works were:

  • KRAS Allelic Imbalance Enhances Fitness and Modulates MAP Kinase Dependence in Cancer. (92 citations)
  • AMPK/FIS1-Mediated Mitophagy Is Required for Self-Renewal of Human AML Stem Cells (82 citations)
  • Clinical Experience With the Subcutaneous Implantable Cardioverter–Defibrillator in Adults With Congenital Heart Disease (43 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Internal medicine

His scientific interests lie mostly in Cancer research, Internal medicine, Myeloid, Cardiology and Leukemia. His Cancer research research includes themes of T cell, KRAS, Combination therapy, Cytotoxic T cell and PI3K/AKT/mTOR pathway. His Myeloid study integrates concerns from other disciplines, such as Neurofibromin 1, Haematopoiesis, Myeloproliferative Disorders and Chromosome 7.

He interconnects Hematology, Bone marrow, Germline and Somatic cell in the investigation of issues within Chromosome 7. His Cardiology course of study focuses on Surgery and Heart disease. His biological study spans a wide range of topics, including AMPK, Myeloid leukemia, Stem cell and Janus kinase 2.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Hyperactive Ras in developmental disorders and cancer.

Suzanne Schubbert;Kevin Shannon;Gideon Bollag.
Nature Reviews Cancer (2007)

1694 Citations

Germline KRAS mutations cause Noonan syndrome

Suzanne Schubbert;Martin Zenker;Sara L . Rowe;Silke Böll.
Nature Genetics (2006)

743 Citations

The Rate of Telomere Sequence Loss in Human Leukocytes Varies with Age

Robert W. Frenck;Elizabeth H. Blackburn;Kevin M. Shannon.
Proceedings of the National Academy of Sciences of the United States of America (1998)

679 Citations

Loss of NF1 results in activation of the Ras signaling pathway and leads to aberrant growth in haematopoietic cells

G. Bollag;D. W. Clapp;S. Shih;F. Adler.
Nature Genetics (1996)

622 Citations

Differential effects of oncogenic K-Ras and N-Ras on proliferation, differentiation and tumor progression in the colon

Kevin M Haigis;Krystle R Kendall;Yufang Wang;Ann Cheung.
Nature Genetics (2008)

552 Citations

Loss of the normal NF1 allele from the bone marrow of children with type 1 neurofibromatosis and malignant myeloid disorders

Kevin M. Shannon;Peter O'Connell;George A. Martin;Dorothy Paderanga.
The New England Journal of Medicine (1994)

474 Citations

Mutations in PTPN11 implicate the SHP-2 phosphatase in leukemogenesis

Mignon L. Loh;Shashaank Vattikuti;Suzanne Schubbert;Melissa G. Reynolds.
Blood (2004)

446 Citations

Recombinant human erythropoietin stimulates erythropoiesis and reduces erythrocyte transfusions in very low birth weight preterm infants.

K M Shannon;J F Keith rd;W C Mentzer;R A Ehrenkranz.
Pediatrics (1995)

389 Citations

Somatic activation of oncogenic Kras in hematopoietic cells initiates a rapidly fatal myeloproliferative disorder.

Benjamin S. Braun;David A. Tuveson;Namie Kong;Doan T. Le.
Proceedings of the National Academy of Sciences of the United States of America (2004)

331 Citations

Homozygous Inactivation of the NF1 Gene in Bone Marrow Cells from Children with Neurofibromatosis Type 1 and Malignant Myeloid Disorders

Lucy Side;Brigit Taylor;Matthew Cayouette;Edward Conner.
The New England Journal of Medicine (1997)

316 Citations

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