D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 106 Citations 37,590 434 World Ranking 2833 National Ranking 1640

Research.com Recognitions

Awards & Achievements

2020 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Internal medicine

The scientist’s investigation covers issues in Neurofibromatosis, Pathology, Cancer research, Neurofibromin 1 and Astrocyte. His research integrates issues of Endocrinology, Pediatrics, Internal medicine and Glioma in his study of Neurofibromatosis. His work deals with themes such as Transgene, Carcinogenesis, Tumor suppressor gene, Immunology and PTEN, which intersect with Cancer research.

His Tumor suppressor gene research is multidisciplinary, relying on both Neoplastic transformation, Merlin and Cell growth. His Neurofibromin 1 research focuses on subjects like Gene product, which are linked to Positional cloning. In his study, Amyotrophic lateral sclerosis is strongly linked to Glial fibrillary acidic protein, which falls under the umbrella field of Astrocyte.

His most cited work include:

  • The diagnostic evaluation and multidisciplinary management of neurofibromatosis 1 and neurofibromatosis 2. (1030 citations)
  • Molecular Profiling Reveals Biologically Discrete Subsets and Pathways of Progression in Diffuse Glioma. (965 citations)
  • Astrocytes as determinants of disease progression in inherited amyotrophic lateral sclerosis. (858 citations)

What are the main themes of his work throughout his whole career to date?

David H. Gutmann mostly deals with Neurofibromatosis, Pathology, Cancer research, Neurofibromin 1 and Glioma. His Neurofibromatosis research is multidisciplinary, incorporating perspectives in Internal medicine, Neuroscience and Pediatrics. His research integrates issues of Endocrinology and Oncology in his study of Internal medicine.

David H. Gutmann has researched Cancer research in several fields, including Tumor suppressor gene, Signal transduction, Immunology and Cell growth. His research in Neurofibromin 1 tackles topics such as Cell biology which are related to areas like Merlin and Astrocyte. His Glioma research includes elements of Tumor microenvironment and Microglia.

He most often published in these fields:

  • Neurofibromatosis (43.97%)
  • Pathology (27.20%)
  • Cancer research (25.57%)

What were the highlights of his more recent work (between 2014-2021)?

  • Neurofibromatosis (43.97%)
  • Cancer research (25.57%)
  • Pathology (27.20%)

In recent papers he was focusing on the following fields of study:

Neurofibromatosis, Cancer research, Pathology, Glioma and Neuroscience are his primary areas of study. His Neurofibromatosis study focuses on Neurofibromin 1 in particular. His Cancer research research is multidisciplinary, incorporating elements of Mutation and Brain tumor.

His Pathology study integrates concerns from other disciplines, such as Astrocytoma and Optic nerve. His studies deal with areas such as Cancer cell, Microglia, Stem cell and Immunology as well as Glioma. His work carried out in the field of Neuroscience brings together such families of science as Phenotype and Cancer.

Between 2014 and 2021, his most popular works were:

  • Molecular Profiling Reveals Biologically Discrete Subsets and Pathways of Progression in Diffuse Glioma. (965 citations)
  • The role of microglia and macrophages in glioma maintenance and progression (537 citations)
  • Comprehensive and Integrated Genomic Characterization of Adult Soft Tissue Sarcomas (327 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Internal medicine

Neurofibromatosis, Glioma, Cancer research, Pathology and Microglia are his primary areas of study. David H. Gutmann is interested in Neurofibromin 1, which is a branch of Neurofibromatosis. His work deals with themes such as Mutation and Neural stem cell, which intersect with Neurofibromin 1.

His biological study spans a wide range of topics, including Transcriptome, Gene and Immunology. His Cancer research research incorporates elements of Stem cell and ATRX. David H. Gutmann combines subjects such as Neuroscience, Immune system, Cell type and Monocyte with his study of Microglia.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The diagnostic evaluation and multidisciplinary management of neurofibromatosis 1 and neurofibromatosis 2.

David H. Gutmann;Arthur Aylsworth;John C. Carey;Bruce Korf.
JAMA (1997)

1620 Citations

Molecular Profiling Reveals Biologically Discrete Subsets and Pathways of Progression in Diffuse Glioma.

Michele Ceccarelli;Floris P Barthel;Tathiane M Malta;Thais S Sabedot.
Cell (2016)

1204 Citations

Astrocytes as determinants of disease progression in inherited amyotrophic lateral sclerosis.

Koji Yamanaka;Seung Joo Chun;Severine Boillee;Noriko Fujimori-Tonou.
Nature Neuroscience (2008)

1082 Citations

Prevalence of dementia and major subtypes in Europe: A collaborative study of population-based cohorts. Neurologic Diseases in the Elderly Research Group.

Allison King;David H. Gutmann.
Neurology (2000)

1027 Citations

Subtypes of medulloblastoma have distinct developmental origins

Paul Gibson;Yiai Tong;Giles Robinson;Margaret C. Thompson.
Nature (2010)

754 Citations

Aberrant regulation of ras proteins in malignant tumour cells from type 1 neurofibromatosis patients.

Tanya N. Basu;David H. Gutmann;Jonathan A. Fletcher;Thomas W. Glover.
Nature (1992)

754 Citations

Rapamycin prevents epilepsy in a mouse model of tuberous sclerosis complex.

Ling-Hui Zeng;Lin Xu;David H. Gutmann;Michael Wong.
Annals of Neurology (2008)

750 Citations

International Consensus Statement on Malignant Peripheral Nerve Sheath Tumors in Neurofibromatosis 1

Rosalie E. Ferner;David H. Gutmann.
Cancer Research (2002)

691 Citations

The role of microglia and macrophages in glioma maintenance and progression

Dolores Hambardzumyan;David H Gutmann;Helmut Kettenmann.
Nature Neuroscience (2016)

666 Citations

Neurofibromatosis type 1 revisited.

Virginia C Williams;John Lucas;Michael A Babcock;David H Gutmann.
Pediatrics (2009)

637 Citations

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