David T.W. Jones mainly focuses on Medulloblastoma, Cancer research, Pathology, Bioinformatics and DNA methylation. The concepts of his Medulloblastoma study are interwoven with issues in Cancer, Gene expression profiling, Immunology, Chromothripsis and Human genetics. His Cancer research study combines topics from a wide range of disciplines, such as Mutation, IDH1, Genetics and Exome.
His research in Pathology intersects with topics in Young adult, Internal medicine and Oncology. His Bioinformatics study integrates concerns from other disciplines, such as Survival rate, Disease, Gene and Genomics. His DNA methylation research is multidisciplinary, incorporating perspectives in Regulation of gene expression, Methylation, Epigenetics and Histopathology.
His primary areas of study are Pathology, Cancer research, DNA methylation, Internal medicine and Medulloblastoma. His Pathology research incorporates elements of Magnetic resonance imaging and Mutation. David T.W. Jones has researched Cancer research in several fields, including Mutation and Carcinogenesis.
Mutation is a primary field of his research addressed under Genetics. As a member of one scientific family, David T.W. Jones mostly works in the field of DNA methylation, focusing on Epigenetics and, on occasion, Epigenomics. The Medulloblastoma study combines topics in areas such as Cancer, Wnt signaling pathway and Bioinformatics.
Cancer research, Internal medicine, DNA methylation, Pathology and Oncology are his primary areas of study. His Cancer research research includes elements of Mutation, Gene mutation and Epigenetics. David T.W. Jones regularly ties together related areas like MEDLINE in his Internal medicine studies.
He interconnects Methylation, DNA, Sarcoma and Clear cell in the investigation of issues within DNA methylation. His Pathology research includes themes of Gene panel and Oligodendroglioma. In Disease, David T.W. Jones works on issues like Clinical trial, which are connected to Biomarker.
His scientific interests lie mostly in Pathology, Internal medicine, Frontotemporal dementia, DNA methylation and Cancer research. His work carried out in the field of Pathology brings together such families of science as Hippocampal sclerosis, Standardized uptake value and Magnetic resonance imaging. His Internal medicine study incorporates themes from Oncology, MEDLINE and Profiling.
In general Frontotemporal dementia study, his work on Frontotemporal lobar degeneration and C9orf72 often relates to the realm of Grossman, thereby connecting several areas of interest. His research integrates issues of Gene panel, Methylation, Cohort and Clear cell in his study of DNA methylation. His studies in Cancer research integrate themes in fields like Mutation, Gene mutation and Histone H3.
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Unruptured intracranial aneurysms - Risk of rupture and risks of surgical intervention
D. Wiebers;J. Whisnant;G. Forbes;I. Meissner.
The New England Journal of Medicine (1998)
Driver mutations in histone H3.3 and chromatin remodelling genes in paediatric glioblastoma
Jeremy Schwartzentruber;Andrey Korshunov;Xiao Yang Liu;David T.W. Jones.
Hotspot mutations in H3F3A and IDH1 define distinct epigenetic and biological subgroups of glioblastoma.
Dominik Sturm;Hendrik Witt;Hendrik Witt;Volker Hovestadt;Dong Anh Khuong-Quang.
Cancer Cell (2012)
Pan-cancer analysis of whole genomes
Peter J. Campbell;Gad Getz;Jan O. Korbel;Joshua M. Stuart.
DNA methylation-based classification of central nervous system tumours
David Capper;David Capper;David Capper;David T. W. Jones;Martin Sill;Volker Hovestadt.
Molecular subgroups of medulloblastoma: an international meta-analysis of transcriptome, genetic aberrations, and clinical data of WNT, SHH, Group 3, and Group 4 medulloblastomas
Marcel Kool;Andrey Korshunov;Marc Remke;Marc Remke;David T.W. Jones.
Acta Neuropathologica (2012)
Tandem duplication producing a novel oncogenic BRAF fusion gene defines the majority of pilocytic astrocytomas.
David T.W. Jones;Sylvia Kocialkowski;Lu Liu;Danita M. Pearson.
Cancer Research (2008)
Dissecting the genomic complexity underlying medulloblastoma
David T. W. Jones;Natalie Jäger;Marcel Kool;Thomas Zichner.
Genome Sequencing of Pediatric Medulloblastoma Links Catastrophic DNA Rearrangements with TP53 Mutations
Tobias Rausch;David T.W. Jones;Marc Zapatka;Adrian M. Stütz.
Medulloblastoma exome sequencing uncovers subtype-specific somatic mutations
Trevor J Pugh;Trevor J Pugh;Shyamal Dilhan Weeraratne;Shyamal Dilhan Weeraratne;Tenley C. Archer;Tenley C. Archer;Daniel A. Pomeranz Krummel.
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