D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 84 Citations 18,788 192 World Ranking 7970 National Ranking 331

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Surgery

Abhijit Guha focuses on Cancer research, Pathology, Glioma, Growth factor and Epidermal growth factor receptor. Abhijit Guha does research in Cancer research, focusing on Vascular endothelial growth factor specifically. His Pathology research integrates issues from Cancer stem cell and Astrocytoma.

The various areas that Abhijit Guha examines in his Glioma study include Molecular biology and Stem cell. In Growth factor, he works on issues like Angiogenesis, which are connected to Cancer cell. He has included themes like Epidermal growth factor and Signal transduction in his Epidermal growth factor receptor study.

His most cited work include:

  • The Somatic Genomic Landscape of Glioblastoma (2550 citations)
  • Driver mutations in histone H3.3 and chromatin remodelling genes in paediatric glioblastoma (1542 citations)
  • Intercellular transfer of the oncogenic receptor EGFRvIII by microvesicles derived from tumour cells (1296 citations)

What are the main themes of his work throughout his whole career to date?

Abhijit Guha mainly focuses on Cancer research, Pathology, Glioma, Astrocytoma and Neurofibromatosis. His work deals with themes such as Genetically modified mouse, Signal transduction and Epidermal growth factor receptor, which intersect with Cancer research. His study in Pathology is interdisciplinary in nature, drawing from both Cancer and Central nervous system disease.

His Glioma research includes themes of Tumor suppressor gene and Molecular biology. He combines subjects such as Surgery and Gene with his study of Neurofibromatosis. His studies in Angiogenesis integrate themes in fields like Cell cycle and Growth factor.

He most often published in these fields:

  • Cancer research (62.70%)
  • Pathology (38.11%)
  • Glioma (18.03%)

What were the highlights of his more recent work (between 2007-2018)?

  • Cancer research (62.70%)
  • Pathology (38.11%)
  • Neurofibromatosis (18.44%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Cancer research, Pathology, Neurofibromatosis, Glioma and Genetics. His research in Cancer research is mostly focused on Angiogenesis. In his research on the topic of Pathology, Computational biology is strongly related with Gene expression profiling.

His Neurofibromatosis study incorporates themes from Presentation and Surgery. His biological study spans a wide range of topics, including Cancer cell, Progenitor cell, Molecular biology, Cell of origin and Neuroscience. His work carried out in the field of Epidermal growth factor receptor brings together such families of science as Cell culture, Receptor tyrosine kinase, Proximity ligation assay and PTEN.

Between 2007 and 2018, his most popular works were:

  • The Somatic Genomic Landscape of Glioblastoma (2550 citations)
  • Driver mutations in histone H3.3 and chromatin remodelling genes in paediatric glioblastoma (1542 citations)
  • Intercellular transfer of the oncogenic receptor EGFRvIII by microvesicles derived from tumour cells (1296 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Surgery

His primary scientific interests are in Cancer research, Cancer cell, Pathology, Glioma and Neurofibromatosis. His research integrates issues of Glycolysis, Kinase and DNA repair in his study of Cancer research. His studies deal with areas such as Microvesicles, Angiogenesis and Stem cell as well as Cancer cell.

His Angiogenesis research incorporates themes from Hexokinase and Vascular endothelial growth factor. Abhijit Guha combines subjects such as Carcinogenesis and Comparative genomic hybridization with his study of Pathology. His studies deal with areas such as Mutation, Germline mutation and Germline as well as Neurofibromatosis.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The Somatic Genomic Landscape of Glioblastoma

Cameron W. Brennan;Roel G W Verhaak;Aaron McKenna;Benito Campos.
Cell (2013)

4024 Citations

Intercellular transfer of the oncogenic receptor EGFRvIII by microvesicles derived from tumour cells

Khalid Al-Nedawi;Brian Meehan;Johann Micallef;Vladimir Lhotak.
Nature Cell Biology (2008)

2033 Citations

Driver mutations in histone H3.3 and chromatin remodelling genes in paediatric glioblastoma

Jeremy Schwartzentruber;Andrey Korshunov;Xiao Yang Liu;David T.W. Jones.
Nature (2012)

1923 Citations

Radial glia cells are candidate stem cells of ependymoma

Michael D. Taylor;Helen Poppleton;Christine Fuller;Xiaoping Su.
Cancer Cell (2005)

830 Citations

Hexokinase 2 is a key mediator of aerobic glycolysis and promotes tumor growth in human glioblastoma multiforme

Amparo Wolf;Sameer Agnihotri;Johann Micallef;Joydeep Mukherjee.
Journal of Experimental Medicine (2011)

632 Citations

Transforming fusions of FGFR and TACC genes in human glioblastoma

Devendra Singh;Joseph Minhow Chan;Pietro Zoppoli;Francesco Niola;Francesco Niola.
Science (2012)

611 Citations

Delineation of Two Clinically and Molecularly Distinct Subgroups of Posterior Fossa Ependymoma

Hendrik Witt;Stephen C. Mack;Marina Ryzhova;Sebastian Bender;Sebastian Bender.
Cancer Cell (2011)

420 Citations

Expression of PDGF and PDGF receptors in human astrocytoma operation specimens supports the existence of an autocrine loop

Abhijit Guha;Abhijit Guha;Kathleen Dashner;Peter L. Mc Black;John A. Wagner.
International Journal of Cancer (1995)

402 Citations

Proliferation of human malignant astrocytomas is dependent on Ras activation.

Abhijit Guha;Abhijit Guha;Matthias M Feldkamp;Nelson Lau;Gerry Boss.
Oncogene (1997)

391 Citations

Astrocyte-specific Expression of Activated p21-ras Results in Malignant Astrocytoma Formation in a Transgenic Mouse Model of Human Gliomas

Hao Ding;Luba Roncari;Luba Roncari;Patrick Shannon;Xiaoli Wu.
Cancer Research (2001)

332 Citations

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