2023 - Research.com Biology and Biochemistry in Japan Leader Award
2022 - Research.com Biology and Biochemistry in Japan Leader Award
2011 - Keio Medical Science Prize, Keio University, Tokyo, Japan
His primary areas of investigation include Cell biology, Ubiquitin, Biochemistry, Proteasome and Autophagy. Keiji Tanaka combines subjects such as Aggrephagy and Ubiquitin-conjugating enzyme with his study of Cell biology. His work deals with themes such as Parkin, DNA ligase and Phosphorylation, which intersect with Ubiquitin.
His study in Proteasome is interdisciplinary in nature, drawing from both Protease, Protein subunit, Proteolysis, Molecular biology and Proteasome assembly. The concepts of his Autophagy study are interwoven with issues in Phagosome and Neurodegeneration. His study looks at the intersection of Autophagosome and topics like Chaperone-mediated autophagy with Programmed cell death.
His main research concerns Cell biology, Biochemistry, Proteasome, Ubiquitin and Molecular biology. His research on Cell biology often connects related areas such as Autophagy. His Autophagy study focuses on Autophagy-Related Protein 7 in particular.
His study looks at the relationship between Proteasome and topics such as Protein subunit, which overlap with Complementary DNA. His Ubiquitin study incorporates themes from Parkin and DNA ligase. He interconnects Mitochondrion and Mitophagy in the investigation of issues within Parkin.
Keiji Tanaka focuses on Cell biology, Ubiquitin, Ubiquitin ligase, Proteasome and Biochemistry. His Cell biology research includes elements of Autophagy, Ribosome, Mutant and Monoubiquitination. His work carried out in the field of Ubiquitin brings together such families of science as Parkin, Signal transduction and DNA ligase.
His work is dedicated to discovering how Parkin, Mitochondrion are connected with Molecular biology and other disciplines. Keiji Tanaka specializes in Ubiquitin ligase, namely F-box protein. His research in Proteasome intersects with topics in Protein subunit, Chaperone, Function, Proteasome assembly and Proteostasis.
Keiji Tanaka mostly deals with Cell biology, Ubiquitin, Autophagy, Biochemistry and Ubiquitin ligase. The Cell biology study combines topics in areas such as Translation, Molecular biology and Ribosome, Ribosomal protein. His research integrates issues of Immune system, B cell, Enzyme, Function and Proteasome in his study of Ubiquitin.
The various areas that Keiji Tanaka examines in his Proteasome study include Gene duplication, Proteostasis, Mutant and Self Tolerance. Keiji Tanaka has included themes like Inclusion bodies, Cell and Signal transducing adaptor protein in his Autophagy study. His Ubiquitin ligase research is multidisciplinary, incorporating elements of Parkin, PINK1 and Deubiquitinating enzyme.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Loss of autophagy in the central nervous system causes neurodegeneration in mice
Masaaki Komatsu;Satoshi Waguri;Satoshi Waguri;Tomoki Chiba;Shigeo Murata.
Structure and Functions of the 20S and 26S Proteasomes
Olivier Coux;Keiji Tanaka;Alfred L. Goldberg.
Annual Review of Biochemistry (1996)
Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase
Hideki Shimura;Nobutaka Hattori;Shin-ichiro Kubo;Yoshikuni Mizuno.
Nature Genetics (2000)
Autophagy regulates lipid metabolism
Rajat Singh;Susmita Kaushik;Yongjun Wang;Youqing Xiang.
Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes
Daniel J. Klionsky;Hagai Abeliovich;Patrizia Agostinis;Devendra K. Agrawal.
Impairment of starvation-induced and constitutive autophagy in Atg7-deficient mice
Masaaki Komatsu;Satoshi Waguri;Takashi Ueno;Junichi Iwata.
Journal of Cell Biology (2005)
Homeostatic Levels of p62 Control Cytoplasmic Inclusion Body Formation in Autophagy-Deficient Mice
Masaaki Komatsu;Satoshi Waguri;Masato Koike;Yu shin Sou;Yu shin Sou.
Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1beta production.
Tatsuya Saitoh;Naonobu Fujita;Myoung Ho Jang;Satoshi Uematsu.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: