What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His primary areas of investigation include Cell biology, Ubiquitin, Biochemistry, Proteasome and Autophagy.
Keiji Tanaka combines subjects such as Aggrephagy and Ubiquitin-conjugating enzyme with his study of Cell biology.
His work deals with themes such as Parkin, DNA ligase and Phosphorylation, which intersect with Ubiquitin.
His study in Proteasome is interdisciplinary in nature, drawing from both Protease, Protein subunit, Proteolysis, Molecular biology and Proteasome assembly.
The concepts of his Autophagy study are interwoven with issues in Phagosome and Neurodegeneration.
His study looks at the intersection of Autophagosome and topics like Chaperone-mediated autophagy with Programmed cell death.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- Loss of autophagy in the central nervous system causes neurodegeneration in mice (2692 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Cell biology, Biochemistry, Proteasome, Ubiquitin and Molecular biology.
His research on Cell biology often connects related areas such as Autophagy.
His Autophagy study focuses on Autophagy-Related Protein 7 in particular.
His study looks at the relationship between Proteasome and topics such as Protein subunit, which overlap with Complementary DNA.
His Ubiquitin study incorporates themes from Parkin and DNA ligase.
He interconnects Mitochondrion and Mitophagy in the investigation of issues within Parkin.
He most often published in these fields:
- Cell biology (37.23%)
- Biochemistry (33.43%)
- Proteasome (31.68%)
What were the highlights of his more recent work (between 2012-2021)?
- Cell biology (37.23%)
- Ubiquitin (28.18%)
- Ubiquitin ligase (15.47%)
In recent papers he was focusing on the following fields of study:
Keiji Tanaka focuses on Cell biology, Ubiquitin, Ubiquitin ligase, Proteasome and Biochemistry.
His Cell biology research includes elements of Autophagy, Ribosome, Mutant and Monoubiquitination.
His work carried out in the field of Ubiquitin brings together such families of science as Parkin, Signal transduction and DNA ligase.
His work is dedicated to discovering how Parkin, Mitochondrion are connected with Molecular biology and other disciplines.
Keiji Tanaka specializes in Ubiquitin ligase, namely F-box protein.
His research in Proteasome intersects with topics in Protein subunit, Chaperone, Function, Proteasome assembly and Proteostasis.
Between 2012 and 2021, his most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Ubiquitin is phosphorylated by PINK1 to activate parkin (800 citations)
- Phosphorylation of p62 activates the Keap1-Nrf2 pathway during selective autophagy. (541 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
Keiji Tanaka mostly deals with Cell biology, Ubiquitin, Autophagy, Biochemistry and Ubiquitin ligase.
The Cell biology study combines topics in areas such as Translation, Molecular biology and Ribosome, Ribosomal protein.
His research integrates issues of Immune system, B cell, Enzyme, Function and Proteasome in his study of Ubiquitin.
The various areas that Keiji Tanaka examines in his Proteasome study include Gene duplication, Proteostasis, Mutant and Self Tolerance.
Keiji Tanaka has included themes like Inclusion bodies, Cell and Signal transducing adaptor protein in his Autophagy study.
His Ubiquitin ligase research is multidisciplinary, incorporating elements of Parkin, PINK1 and Deubiquitinating enzyme.
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