H-Index & Metrics Best Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 56 Citations 9,987 115 World Ranking 7127 National Ranking 3252

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Internal medicine

His primary scientific interests are in Cell biology, Kinase, Signal transduction, Internal medicine and Endocrinology. His Cell biology study frequently links to other fields, such as Inflammation. His Kinase study combines topics from a wide range of disciplines, such as Molecular biology and Phosphorylation.

His Signal transduction research is multidisciplinary, incorporating perspectives in Endothelial stem cell and Tumor necrosis factor alpha. His Internal medicine research integrates issues from Phosphodiesterase and Protein kinase A. His research integrates issues of Thrombin and Thromboplastin in his study of Endocrinology.

His most cited work include:

  • Big mitogen-activated protein kinase 1 (BMK1) is a redox-sensitive kinase. (396 citations)
  • Cyclophilin A enhances vascular oxidative stress and the development of angiotensin II-induced aortic aneurysms. (266 citations)
  • c-Src is required for oxidative stress-mediated activation of big mitogen-activated protein kinase 1 (BMK1) (253 citations)

What are the main themes of his work throughout his whole career to date?

The scientist’s investigation covers issues in Cell biology, Internal medicine, Signal transduction, Kinase and Endocrinology. Phosphorylation is the focus of his Cell biology research. His research investigates the connection between Internal medicine and topics such as Protein kinase A that intersect with problems in MAPK/ERK pathway.

Jun Ichi Abe has included themes like Endothelial stem cell and Molecular biology in his Kinase study. His research investigates the link between Endocrinology and topics such as Phosphodiesterase 3 that cross with problems in CAMP-Responsive Element Modulator. His Inflammation research incorporates themes from Tumor necrosis factor alpha, Oxidative stress and Cancer research.

He most often published in these fields:

  • Cell biology (45.56%)
  • Internal medicine (26.11%)
  • Signal transduction (23.89%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cell biology (45.56%)
  • Cancer research (15.00%)
  • Inflammation (16.67%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Cell biology, Cancer research, Inflammation, Phosphorylation and Senescence. Signal transduction and Kinase are the primary areas of interest in his Cell biology study. His research in Signal transduction intersects with topics in Fibrosis, Unfolded protein response and Immunology.

His Cancer research research includes elements of Monocytes macrophages, Cancer, In vitro and DNA damage. His work in Inflammation tackles topics such as Downregulation and upregulation which are related to areas like Macrophage, Genetically modified mouse, Cell signaling and Efferocytosis. His studies in Phosphorylation integrate themes in fields like Endothelial stem cell and Apoptosis.

Between 2015 and 2021, his most popular works were:

  • STING-IRF3 Triggers Endothelial Inflammation in Response to Free Fatty Acid-Induced Mitochondrial Damage in Diet-Induced Obesity. (65 citations)
  • Radiation-Induced Endothelial Vascular Injury: A Review of Possible Mechanisms (53 citations)
  • PDE1C deficiency antagonizes pathological cardiac remodeling and dysfunction (40 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Internal medicine

His primary scientific interests are in Cell biology, Disease, Signal transduction, Cancer and Inflammation. The Cell biology study combines topics in areas such as Pyroptosis and Inflammasome. His research on Disease also deals with topics like

  • Radiation therapy, which have a strong connection to Animal studies and Angiogenesis,
  • Senescence which is related to area like Reactive oxygen species, Oxidative stress, Efferocytosis and Stroke.

His Signal transduction research is multidisciplinary, relying on both SUMO protein, Unfolded protein response, DNA methylation and Phosphorylation. Jun Ichi Abe works mostly in the field of Cancer, limiting it down to topics relating to Coronary artery disease and, in certain cases, Proinflammatory cytokine, Survival rate and Relative risk. His Fatty acid study integrates concerns from other disciplines, such as Internal medicine and Endocrinology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Big mitogen-activated protein kinase 1 (BMK1) is a redox-sensitive kinase.

Jun Ichi Abe;Masatoshi Kusuhara;Richard J. Ulevitch;Bradford C. Berk.
Journal of Biological Chemistry (1996)

571 Citations

Cyclophilin A enhances vascular oxidative stress and the development of angiotensin II-induced aortic aneurysms.

Kimio Satoh;Patrizia Nigro;Tetsuya Matoba;Michael R. O'Dell.
Nature Medicine (2009)

406 Citations

c-Src is required for oxidative stress-mediated activation of big mitogen-activated protein kinase 1 (BMK1)

Jun Ichi Abe;Masafumi Takahashi;Mari Ishida;Jiing Dwan Lee.
Journal of Biological Chemistry (1997)

389 Citations

Reactive oxygen species as mediators of signal transduction in cardiovascular disease.

Jun Ichi Abe;Bradford C. Berk.
Trends in Cardiovascular Medicine (1998)

354 Citations

Src and Cas Mediate JNK Activation but Not ERK1/2 and p38 Kinases by Reactive Oxygen Species

Masanori Yoshizumi;Jun Ichi Abe;Judith Haendeler;Qunhua Huang.
Journal of Biological Chemistry (2000)

345 Citations

p90(RSK) is a serum-stimulated Na+/H+ exchanger isoform-1 kinase. Regulatory phosphorylation of serine 703 of Na+/H+ exchanger isoform-1.

Eiichi Takáhashi;Jun Ichi Abe;Byron Gallis;Ruedi Aebersold.
Journal of Biological Chemistry (1999)

332 Citations

Endothelial Atheroprotective and Anti-inflammatory Mechanisms

Bradford C. Berk;Jun Ichi Abe;Wang Min;James Surapisitchat.
Annals of the New York Academy of Sciences (2008)

291 Citations

Fyn and JAK2 mediate Ras activation by reactive oxygen species.

Jun Ichi Abe;Bradford C. Berk.
Journal of Biological Chemistry (1999)

275 Citations

Insulin-Like Growth Factor-1 Enhances Inflammatory Responses in Endothelial Cells: Role of Gab1 and MEKK3 in TNF-α–Induced c-Jun and NF-κB Activation and Adhesion Molecule Expression

Wenyi Che;Nicole Lerner-Marmarosh;Qunhua Huang;Masaki Osawa.
Circulation Research (2002)

244 Citations

Cyclophilin A Mediates Vascular Remodeling by Promoting Inflammation and Vascular Smooth Muscle Cell Proliferation

Kimio Satoh;Tetsuya Matoba;Jun Suzuki;Michael R. O'Dell.
Circulation (2008)

227 Citations

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