His primary areas of study are Cell biology, Endothelium, Molecular biology, Inflammation and Biochemistry. The concepts of his Cell biology study are interwoven with issues in Endothelial stem cell and Cell adhesion. His studies deal with areas such as Blood vessel, Blood flow, Pathology, Mechanotransduction and Vascular disease as well as Endothelium.
His biological study deals with issues like Protein kinase B, which deal with fields such as Basal, Serine and ATP synthase. His Inflammation research incorporates elements of microRNA, Downregulation and upregulation and Endothelial dysfunction. His study looks at the relationship between Biochemistry and fields such as Nitric oxide, as well as how they intersect with chemical problems.
His primary scientific interests are in Cell biology, Endothelium, Internal medicine, Inflammation and Endothelial stem cell. His work on Signal transduction as part of general Cell biology research is often related to Mechanosensitive channels, thus linking different fields of science. His biological study spans a wide range of topics, including Tunica intima, Endothelial dysfunction, KLF2, Pathology and In vivo.
His Internal medicine study integrates concerns from other disciplines, such as Endocrinology, Bone morphogenetic protein and Cardiology. His Inflammation research incorporates themes from Cancer research and Downregulation and upregulation. His microRNA research integrates issues from Microarray, Molecular biology, Gene silencing and Regulation of gene expression.
Hanjoong Jo mainly focuses on Cell biology, Inflammation, Endothelium, Cancer research and microRNA. Hanjoong Jo studies Mesenchymal stem cell, a branch of Cell biology. Hanjoong Jo conducts interdisciplinary study in the fields of Inflammation and Aortic valve calcification through his research.
Much of his study explores Endothelium relationship to In vivo. As part of the same scientific family, Hanjoong Jo usually focuses on Cancer research, concentrating on Endothelial dysfunction and intersecting with Regulation of gene expression, Ligation and Angiogenesis. His microRNA research is multidisciplinary, incorporating perspectives in Gene expression, Disease, RNA interference and Bioinformatics.
Endothelium, Endothelial dysfunction, Cancer research, Cell biology and Regulation of gene expression are his primary areas of study. He interconnects Spheroid, In vivo and Histology in the investigation of issues within Endothelium. His Endothelial dysfunction research includes elements of Coronary artery disease, Knockout mouse, Atheroma and CYR61, CTGF.
His research in Cancer research intersects with topics in Inflammation, Pathology, Matrix metalloproteinase and Candidate gene. He works in the field of Cell biology, namely Barrier function. His Regulation of gene expression study combines topics from a wide range of disciplines, such as MEG3, KLF4, NODAL and Gene expression.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
ROLE OF P47(PHOX) IN VASCULAR OXIDATIVE STRESS AND HYPERTENSION CAUSED BY ANGIOTENSIN II
Ulf Landmesser;Hua Cai;Sergey Dikalov;Louise McCann.
Biological aspects of reactive nitrogen species.
Rakesh P. Patel;Joanne McAndrew;Hassan Sellak;C.Roger White.
Biochimica et Biophysica Acta (1999)
Shear Stress Stimulates Phosphorylation of Endothelial Nitric-oxide Synthase at Ser1179 by Akt-independent Mechanisms ROLE OF PROTEIN KINASE A
Yong Chool Boo;George Sorescu;Nolan Boyd;Ichiro Shiojima.
Journal of Biological Chemistry (2002)
Role of xanthine oxidoreductase and NAD(P)H oxidase in endothelial superoxide production in response to oscillatory shear stress.
J. Scott McNally;Michael E. Davis;Don P. Giddens;Aniket Saha.
American Journal of Physiology-heart and Circulatory Physiology (2003)
Flow-dependent regulation of endothelial nitric oxide synthase: role of protein kinases
Yong Chool Boo;Hanjoong Jo.
American Journal of Physiology-cell Physiology (2003)
Caveolin-mediated regulation of signaling along the p42/44 MAP kinase cascade in vivo. A role for the caveolin-scaffolding domain.
Jeffrey A. Engelman;Caryn Chu;Anning Lin;Hanjoong Jo.
FEBS Letters (1998)
Bone Morphogenic Protein 4 Produced in Endothelial Cells by Oscillatory Shear Stress Induces Monocyte Adhesion by Stimulating Reactive Oxygen Species Production From a Nox1-Based NADPH Oxidase
George P. Sorescu;Hannah Song;Sarah L. Tressel;Jinah Hwang.
Circulation Research (2004)
Biomechanical factors in atherosclerosis: mechanisms and clinical implications.
Brenda R. Kwak;Magnus Bäck;Marie-Luce Bochaton-Piallat;Giuseppina Caligiuri.
European Heart Journal (2014)
Partial carotid ligation is a model of acutely induced disturbed flow, leading to rapid endothelial dysfunction and atherosclerosis.
Douglas Nam;Chih Wen Ni;Amir Rezvan;Jin Suo.
American Journal of Physiology-heart and Circulatory Physiology (2009)
Differential effect of shear stress on extracellular signal-regulated kinase and N-terminal Jun kinase in endothelial cells. Gi2- and Gbeta/gamma-dependent signaling pathways.
Hanjoong Jo;Katalin Sipos;Young-Mi Go;Robert Law.
Journal of Biological Chemistry (1997)
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