Eugene B. Chang mainly focuses on Immunology, Intestinal mucosa, Cell biology, Molecular biology and Gut flora. His research brings together the fields of Microbiome and Immunology. The Intestinal mucosa study combines topics in areas such as Autophagy, Sodium–hydrogen antiporter, Chemokine, Stem cell and Monocyte.
His biological study spans a wide range of topics, including Heat shock protein, Oxidative stress, Biochemistry, Proinflammatory cytokine and Programmed cell death. His studies in Molecular biology integrate themes in fields like Cell culture, Transfection, Gene expression, Ion transporter and Receptor. His study in Gut flora is interdisciplinary in nature, drawing from both Endocrinology, Disease, Internal medicine, Lipid metabolism and Bioinformatics.
Eugene B. Chang mostly deals with Internal medicine, Cell biology, Immunology, Endocrinology and Biochemistry. His work deals with themes such as Heat shock protein and Apical membrane, which intersect with Cell biology. His Heat shock protein study also includes fields such as
His work focuses on many connections between Immunology and other disciplines, such as Inflammatory bowel disease, that overlap with his field of interest in Colitis. He has researched Endocrinology in several fields, including Receptor and Ion transporter. His Gut flora research is multidisciplinary, incorporating perspectives in Microbiome and Disease.
His primary scientific interests are in Microbiome, Gut flora, Internal medicine, Immunology and Inflammation. His Microbiome study combines topics from a wide range of disciplines, such as Computational biology and Pathology. His Gut flora research includes elements of Disease, Cell biology, Physiology, Interleukin 10 and Metabolism.
Eugene B. Chang has included themes like Gastroenterology and Endocrinology in his Internal medicine study. His study explores the link between Immunology and topics such as Inflammatory bowel disease that cross with problems in Gut dysbiosis. When carried out as part of a general Inflammation research project, his work on Systemic inflammation and Myeloperoxidase is frequently linked to work in Neointimal hyperplasia, therefore connecting diverse disciplines of study.
Microbiome, Internal medicine, Disease, Inflammation and Gut flora are his primary areas of study. As a part of the same scientific study, Eugene B. Chang usually deals with the Microbiome, concentrating on Immunology and frequently concerns with Inflammatory bowel disease. His Internal medicine research integrates issues from Gastroenterology, Dietary fiber and Endocrinology.
His research integrates issues of Tumor necrosis factor alpha, Wound healing and Pathology in his study of Inflammation. Eugene B. Chang usually deals with Tumor necrosis factor alpha and limits it to topics linked to HMGB1 and Cell biology. Eugene B. Chang combines subjects such as Digestion, Feces, Small intestine and Physiology with his study of Gut flora.
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Commensal Bifidobacterium promotes antitumor immunity and facilitates anti–PD-L1 efficacy
Ayelet Sivan;Leticia Corrales;Nathaniel Hubert;Jason B. Williams.
Dietary-fat-induced taurocholic acid promotes pathobiont expansion and colitis in Il10 −/− mice
Suzanne Devkota;Yunwei Wang;Mark W. Musch;Vanessa Leone.
Commensal bacteria protect against food allergen sensitization
Andrew T Stefka;Taylor Feehley;Prabhanshu Tripathi;Ju Qiu.
Proceedings of the National Academy of Sciences of the United States of America (2014)
16S rRNA gene-based analysis of fecal microbiota from preterm infants with and without necrotizing enterocolitis
Yunwei Wang;Jeanette D. Hoenig;Kathryn J. Malin;Sanaa Qamar.
The ISME Journal (2009)
Effects of diurnal variation of gut microbes and high-fat feeding on host circadian clock function and metabolism.
Vanessa Leone;Sean M. Gibbons;Sean M. Gibbons;Kristina Martinez;Alan L. Hutchison.
Cell Host & Microbe (2015)
Exercise Prevents Weight Gain and Alters the Gut Microbiota in a Mouse Model of High Fat Diet-Induced Obesity
Christian C. Evans;Kathy J. LePard;Jeff W. Kwak;Mary C. Stancukas.
Intestinal Electrolyte Transport and Diarrheal Disease
Michael Field;Mrinalini C. Rao;Eugene B. Chang.
The New England Journal of Medicine (1989)
Antibiotic-induced perturbations in gut microbial diversity influences neuro-inflammation and amyloidosis in a murine model of Alzheimer’s disease
Myles R. Minter;Can Zhang;Vanessa Leone;Daina L. Ringus.
Scientific Reports (2016)
Probiotics inhibit nuclear factor-κB and induce heat shock proteins in colonic epithelial cells through proteasome inhibition
Elaine O. Petrof;Keishi Kojima;Mark J. Ropeleski;Mark W. Musch.
Soluble factors from Lactobacillus GG activate MAPKs and induce cytoprotective heat shock proteins in intestinal epithelial cells
Yun Tao;Kenneth A. Drabik;Tonya S. Waypa;Mark W. Musch.
American Journal of Physiology-cell Physiology (2006)
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