What is he best known for?
The fields of study he is best known for:
Immunology, Calcitriol receptor, Cell biology, Inflammation and Intestinal mucosa are his primary areas of study.
His Immunology study combines topics in areas such as Microbiome, vitamin D deficiency and Inflammatory bowel disease.
Calcitriol receptor is closely attributed to Immune system in his research.
His research investigates the connection between Cell biology and topics such as Cell culture that intersect with issues in NFKB1.
His Autophagy research is multidisciplinary, relying on both Programmed cell death and Physiology.
His study in Computational biology is interdisciplinary in nature, drawing from both Autophagosome, Autolysosome, Chaperone-mediated autophagy, MAP1LC3B and Sequestosome 1.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Novel role of the vitamin D receptor in maintaining the integrity of the intestinal mucosal barrier (436 citations)
- 16S rRNA gene-based analysis of fecal microbiota from preterm infants with and without necrotizing enterocolitis (436 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Calcitriol receptor, Immunology, Cell biology, Inflammation and Cancer research.
Jun Sun has included themes like Microbiome, Colorectal cancer and Inflammatory bowel disease in his Immunology study.
His research in Cell biology intersects with topics in Autophagy and Cell culture.
His Autophagy research incorporates themes from AMPK and Programmed cell death.
He interconnects Cytokine, Innate immune system, Immune system and Microbiology in the investigation of issues within Inflammation.
His Cancer research research is multidisciplinary, incorporating perspectives in Carcinogenesis, Barrier function, Signal transduction and Necrotizing enterocolitis.
He most often published in these fields:
- Calcitriol receptor (33.62%)
- Immunology (28.82%)
- Cell biology (26.64%)
What were the highlights of his more recent work (between 2019-2021)?
- Calcitriol receptor (33.62%)
- Microbiome (15.72%)
- Dysbiosis (11.79%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Calcitriol receptor, Microbiome, Dysbiosis, Cancer research and Gut flora.
The various areas that he examines in his Calcitriol receptor study include Signal transduction and Colitis.
His studies deal with areas such as Transmission and Horizontal transmission as well as Microbiome.
His Cancer research study combines topics in areas such as Carcinogenesis, STAT3, stat and Claudin.
His Immunology research is multidisciplinary, incorporating elements of Vomiting, Gastrointestinal tract and Disease, Inflammatory bowel disease.
He focuses mostly in the field of Inflammation, narrowing it down to matters related to Downregulation and upregulation and, in some cases, Tight junction.
Between 2019 and 2021, his most popular works were:
- Influence of the Gut Microbiome, Diet, and Environment on Risk of Colorectal Cancer (64 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition) (38 citations)
- Vitamin D receptor promotes healthy microbial metabolites and microbiome. (11 citations)
In his most recent research, the most cited papers focused on:
Jun Sun mainly focuses on Calcitriol receptor, Microbiome, Immunology, Dysbiosis and Inflammatory bowel disease.
His Calcitriol receptor study is concerned with the field of Internal medicine as a whole.
His work deals with themes such as Cell culture, Cell growth, Queuosine, Transfer RNA and Colorectal cancer, which intersect with Microbiome.
In his study, Vitamin D and neurology, Gut flora, Colitis, Inflammation and Receptor is inextricably linked to Crohn's disease, which falls within the broad field of Immunology.
His Dysbiosis research is multidisciplinary, relying on both STAT3, Cancer research, Janus kinase and stat.
As a part of the same scientific family, Jun Sun mostly works in the field of ATG16L1, focusing on Cathelicidin and, on occasion, Autophagy.
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