His scientific interests lie mostly in Molecular biology, Herpes simplex virus, Virus, Virology and Cell nucleus. The Molecular biology study combines topics in areas such as Membrane protein, Epitope, Nuclear lamina and Inner membrane. His Inner membrane study integrates concerns from other disciplines, such as Nuclear membrane and Cytoplasm.
His Herpes simplex virus research is multidisciplinary, relying on both Mutant and Capsid. His Virus research includes themes of Extracellular and Recombinant virus. His Virology study combines topics in areas such as Open reading frame and Gene, Sequence.
His primary areas of investigation include Herpes simplex virus, Molecular biology, Virus, Capsid and Virology. His Herpes simplex virus research includes elements of Vero cell, Cell culture, Viral replication, Viral tegument and Gene. The study incorporates disciplines such as Recombinant virus, Mutant, DNA, Inner membrane and Gene product in addition to Molecular biology.
In his study, which falls under the umbrella issue of Virus, Endoglycosidase H is strongly linked to Glycoprotein. The concepts of his Capsid study are interwoven with issues in Cytoplasm, Plasma protein binding, Cell biology, Immunoprecipitation and Scaffold protein. Joel D. Baines works mostly in the field of Virology, limiting it down to topics relating to Fusion protein and, in certain cases, Antiserum.
His primary areas of study are Herpes simplex virus, Virology, Capsid, Viral replication and Molecular biology. In his study, Nuclear lamina, Messenger RNA and Gene product is strongly linked to Cell biology, which falls under the umbrella field of Herpes simplex virus. His Virus and Vero cell study in the realm of Virology interacts with subjects such as Raltegravir.
His Capsid study combines topics from a wide range of disciplines, such as Southern blot, Biophysics, Nuclear membrane and Enzyme. His research integrates issues of Viral tegument and Viral transformation in his study of Viral replication. His research in Molecular biology intersects with topics in Reverse transcriptase, DNA, DNA replication and Active site.
The scientist’s investigation covers issues in Herpes simplex virus, Viral replication, Virus, Capsid and Virology. Joel D. Baines combines subjects such as Nuclear membrane, C-terminus and Cas9 with his study of Herpes simplex virus. Joel D. Baines has included themes like Keratitis, Encephalitis and CRISPR in his Viral replication study.
His work in the fields of Virus, such as Vero cell, Neutralizing antibody and Virus Release, intersects with other areas such as Alphaherpesvirinae. His studies in Capsid integrate themes in fields like Immunogold labelling, Inner membrane, Molecular biology and Cell biology. His Virology study frequently links to adjacent areas such as Gene.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Herpesviruses remodel host membranes for virus egress
David C. Johnson;Joel D. Baines.
Nature Reviews Microbiology (2011)
Ultrastructural localization of the herpes simplex virus type 1 UL31, UL34, and US3 proteins suggests specific roles in primary envelopment and egress of nucleocapsids.
Ashley E. Reynolds;Elizabeth G. Wills;Richard J. Roller;Brent J. Ryckman.
Journal of Virology (2002)
U(L)31 and U(L)34 proteins of herpes simplex virus type 1 form a complex that accumulates at the nuclear rim and is required for envelopment of nucleocapsids.
Ashley E. Reynolds;Brent J. Ryckman;Joel D. Baines;Yuping Zhou.
Journal of Virology (2001)
Conformational changes in the nuclear lamina induced by herpes simplex virus type 1 require genes U(L)31 and U(L)34.
Ashley E. Reynolds;Li Liang;Joel D. Baines.
Journal of Virology (2004)
Herpes Simplex Virus Type 1 Infection Induces Activation and Recruitment of Protein Kinase C to the Nuclear Membrane and Increased Phosphorylation of Lamin B
Richard Park;Joel D. Baines.
Journal of Virology (2006)
THE OPEN READING FRAMES UL3, UL4, UL10, AND UL16 ARE DISPENSABLE FOR THE REPLICATION OF HERPES SIMPLEX VIRUS 1 IN CELL CULTURE
J D Baines;B Roizman.
Journal of Virology (1991)
The UL20 gene of herpes simplex virus 1 encodes a function necessary for viral egress.
J D Baines;P L Ward;G Campadelli-Fiume;B Roizman.
Journal of Virology (1991)
Monoclonal antibodies to the spike protein of feline infectious peritonitis virus mediate antibody-dependent enhancement of infection of feline macrophages.
C W Olsen;W V Corapi;C K Ngichabe;J D Baines.
Journal of Virology (1992)
Type I Interferon Production during Herpes Simplex Virus Infection Is Controlled by Cell-Type-Specific Viral Recognition through Toll-Like Receptor 9, the Mitochondrial Antiviral Signaling Protein Pathway, and Novel Recognition Systems
Simon Brandtoft Rasmussen;Louise Nørgaard Sørensen;Lene Malmgaard;Nina Ank.
Journal of Virology (2007)
US3 of Herpes Simplex Virus Type 1 Encodes a Promiscuous Protein Kinase That Phosphorylates and Alters Localization of Lamin A/C in Infected Cells
Fan Mou;Tom Forest;Joel D. Baines.
Journal of Virology (2007)
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