D-Index & Metrics Best Publications

Jochen Walter

University of Bonn
Germany

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name Citations Publications World Ranking National Ranking

Biology and Biochemistry D-index 65 Citations 38,853 166 World Ranking 5750 National Ranking 420

Overview

What is he best known for?

The fields of study Jochen Walter is best known for:

Gene
Enzyme
Alzheimer's disease

His Central nervous system study frequently draws connections between adjacent fields such as Endocrinology and Neuroscience. Jochen Walter conducts interdisciplinary study in the fields of Endocrinology and Pathology through his works. He combines topics linked to Neurodegeneration with his work on Pathology. Jochen Walter performs integrative study on Neurodegeneration and Biochemistry of Alzheimer's disease. His study in Central nervous system extends to Neuroscience with its themes. His work on Biochemistry is being expanded to include thematically relevant topics such as In vitro. Much of his study explores In vitro relationship to Biochemistry. As part of his studies on Disease, Jochen Walter frequently links adjacent subjects like P3 peptide. While working on this project, Jochen Walter studies both P3 peptide and Biochemistry of Alzheimer's disease.

His most cited work include:

Microglia-derived ASC specks cross-seed amyloid-β in Alzheimer’s disease (554 citations)
Constitutive Phosphorylation of the Parkinson's Disease Associated α-Synuclein (450 citations)
Structure of bovine pancreatic trypsin inhibitor (446 citations)

What are the main themes of his work throughout his whole career to date

His Amyloid precursor protein research is covered under the topics of Amyloid precursor protein secretase and P3 peptide. His research ties Apoptosis and Biochemistry together. In his articles, he combines various disciplines, including Cell biology and Enzyme. In his papers, he integrates diverse fields, such as Enzyme and Alzheimer's disease. He performs multidisciplinary study in the fields of Alzheimer's disease and Senile plaques via his papers. His Senile plaques study frequently draws connections to other fields, such as Pathology. His research on Pathology frequently links to adjacent areas such as Presenilin. His study on Disease is mostly dedicated to connecting different topics, such as Neurodegeneration. Jochen Walter regularly links together related areas like Disease in his Neurodegeneration studies.

Jochen Walter most often published in these fields:

Biochemistry (75.41%)
Cell biology (63.11%)
Disease (63.11%)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)

8964 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)

8302 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

7788 Citations

Constitutive Phosphorylation of the Parkinson's Disease Associated α-Synuclein *

Masayasu Okochi;Jochen Walter;Akihiko Koyama;Shigeo Nakajo.
Journal of Biological Chemistry (2000)

636 Citations

Inhibition of cyclin-dependent kinases, GSK-3β and CK1 by hymenialdisine, a marine sponge constituent

L. Meijer;A. M W H Thunnissen;A. W. White;M. Garnier.
Chemistry & Biology (2000)

562 Citations

Microglia-derived ASC specks cross-seed amyloid-β in Alzheimer’s disease

Carmen Venegas;Sathish Kumar;Bernardo S. Franklin;Tobias Dierkes.
Nature (2017)

527 Citations

The presenilin 2 mutation (N141I) linked to familial Alzheimer disease (Volga German families) increases the secretion of amyloid β protein ending at the 42nd (or 43rd) residue

Taisuke Tomita;Kei Maruyama;Takaomi C. Saido;Hideaki Kume.
Proceedings of the National Academy of Sciences of the United States of America (1997)

493 Citations

Nonsteroidal anti-inflammatory drugs repress β-secretase gene promoter activity by the activation of PPARγ

Magdalena Sastre;Ilse Dewachter;Steffen Rossner;Nenad Bogdanovic.
Proceedings of the National Academy of Sciences of the United States of America (2006)

454 Citations

Locus ceruleus controls Alzheimer's disease pathology by modulating microglial functions through norepinephrine

Michael T. Heneka;Fabian Nadrigny;Tommy Regen;Ana Martinez-Hernandez.
Proceedings of the National Academy of Sciences of the United States of America (2010)

453 Citations

Phosphorylation Regulates Intracellular Trafficking of β-Secretase

Jochen Walter;Regina Fluhrer;Bianka Hartung;Michael Willem.
Journal of Biological Chemistry (2001)

366 Citations

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