D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Genetics and Molecular Biology D-index 49 Citations 7,259 130 World Ranking 3924 National Ranking 1857

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

J. Justin McCormick spends much of his time researching Molecular biology, Xeroderma pigmentosum, Nucleotide excision repair, Cell culture and DNA repair. His Molecular biology study combines topics from a wide range of disciplines, such as Genetics, Biochemistry, DNA, Gene and DNA replication. His research in Xeroderma pigmentosum intersects with topics in Mutation, Cytotoxic T cell, DNA excision and Carcinogen.

His Cell culture research is multidisciplinary, incorporating perspectives in Ploidy, Cell, Fibrosarcoma and Oncogene. His work is dedicated to discovering how DNA repair, DNA damage are connected with Gene mutation and other disciplines. His Fibroblast study incorporates themes from Malignant transformation and Cancer research.

His most cited work include:

  • Frequency of ultraviolet light-induced mutations is higher in xeroderma pigmentosum variant cells than in normal human cells (297 citations)
  • Hydrogen ion buffers for biological research (278 citations)
  • Overview of matrix metalloproteinase expression in cultured human cells. (226 citations)

What are the main themes of his work throughout his whole career to date?

J. Justin McCormick mainly investigates Molecular biology, DNA, Cell culture, Biochemistry and Nucleotide excision repair. His Molecular biology research is multidisciplinary, incorporating elements of Mutation, Genetics, Gene, Transfection and Fibroblast. His Fibroblast research integrates issues from Malignant transformation and Transformation.

He has included themes like Cell and Fibrosarcoma in his Cell culture study. His Nucleotide excision repair research incorporates themes from Xeroderma pigmentosum and DNA synthesis. His study focuses on the intersection of Xeroderma pigmentosum and fields such as Cytotoxic T cell with connections in the field of Cytotoxicity.

He most often published in these fields:

  • Molecular biology (59.28%)
  • DNA (26.95%)
  • Cell culture (25.75%)

What were the highlights of his more recent work (between 2004-2020)?

  • Molecular biology (59.28%)
  • Cell biology (10.18%)
  • Cancer research (10.78%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Molecular biology, Cell biology, Cancer research, Malignant transformation and Cell culture. His Molecular biology research is multidisciplinary, relying on both Genetics, DNA, DNA polymerase, Reporter gene and Cell cycle. DNA is a subfield of Biochemistry that J. Justin McCormick studies.

His research on Malignant transformation also deals with topics like

  • Fibroblast, which have a strong connection to Oncogene, Cell, Transformation, Cdc42 GTP-Binding Protein and Mutant,
  • Transcription factor which intersects with area such as Hypoxia-inducible factors, Regulation of gene expression and Sp1 transcription factor. His research in Cell culture focuses on subjects like Fibrosarcoma, which are connected to MAPK/ERK pathway and Receptor. J. Justin McCormick regularly ties together related areas like Nucleotide excision repair in his Xeroderma pigmentosum studies.

Between 2004 and 2020, his most popular works were:

  • Down-regulation of overexpressed Sp1 protein in human fibrosarcoma cell lines inhibits tumor formation (110 citations)
  • Evidence that in Xeroderma Pigmentosum Variant Cells, which Lack DNA Polymerase η, DNA Polymerase ι Causes the Very High Frequency and Unique Spectrum of UV-Induced Mutations (96 citations)
  • The Biphasic Role of the Hypoxia-Inducible Factor Prolyl-4-Hydroxylase, PHD2, in Modulating Tumor-Forming Potential (52 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Enzyme

The scientist’s investigation covers issues in Molecular biology, Cancer research, Cell culture, Cell and Mutation. J. Justin McCormick combines Molecular biology and DNA Polymerase Iota in his studies. His Cancer research research includes elements of Ovarian tumor, Ovarian cancer, Distribution and In vivo.

His study in Cell culture is interdisciplinary in nature, drawing from both Fibrosarcoma and Oncogene. His work focuses on many connections between Cell and other disciplines, such as Mutagenesis, that overlap with his field of interest in Fibroblast. His studies deal with areas such as Cell cycle, Protein subunit and DNA, DNA polymerase as well as Mutation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Frequency of ultraviolet light-induced mutations is higher in xeroderma pigmentosum variant cells than in normal human cells

Veronica M. Maher;Louis M. Ouellette;Rodger D. Curren;J. Justin Mccormick.
Nature (1976)

452 Citations

Hydrogen ion buffers for biological research

Wilfred J. Ferguson;K.I. Braunschweiger;W.R. Braunschweiger;James R. Smith.
Analytical Biochemistry (1980)

428 Citations

DNA excision-repair processes in human cells can eliminate the cytotoxic and mutagenic consequences of ultraviolet irradiation.

Veronica M. Maher;Delia J. Dorney;Alan L. Mendrala;Beate Konze-Thomas.
Mutation Research (1979)

327 Citations

Overview of matrix metalloproteinase expression in cultured human cells.

Troy A. Giambernardi;George M. Grant;Gail P. Taylor;Robert J. Hay.
Matrix Biology (1998)

308 Citations

Evidence from mutation spectra that the UV hypermutability of xeroderma pigmentosum variant cells reflects abnormal, error-prone replication on a template containing photoproducts.

Yi Ching Wang;Veronica M. Maher;David L. Mitchell;J. Justin Mccormick.
Molecular and Cellular Biology (1993)

224 Citations

Cell cycle-dependent strand bias for UV-induced mutations in the transcribed strand of excision repair-proficient human fibroblasts but not in repair-deficient cells.

W. G. Mcgregor;Ruey-Hwa Chen;L. Lukash;V. M. Maher.
Molecular and Cellular Biology (1991)

194 Citations

Amplification and direct nucleotide sequencing of cDNA from the lysate of low numbers of diploid human cells.

Jia-Ling Yang;Veronica M. Maher;J.Justin McCormick.
Gene (1989)

179 Citations

Preferential repair and strand-specific repair of benzo[a]pyrene diol epoxide adducts in the HPRT gene of diploid human fibroblasts

Ruey-Hwa Chen;V. M. Maher;J. Brouwer;P. Van De Putte.
Proceedings of the National Academy of Sciences of the United States of America (1992)

171 Citations

Down-regulation of overexpressed Sp1 protein in human fibrosarcoma cell lines inhibits tumor formation

Zhenjun Lou;Sandra O'Reilly;Hongyan Liang;Veronica M. Maher.
Cancer Research (2005)

169 Citations

Correlation between O6-methylguanine-DNA-methyltransferase activity and resistance of human cells to the cytotoxic and mutagenic effect of N-methyl-N'-nitro-N-nitrosoguanidine.

Jeanne Domoradzki;Anthony E. Pegg;M. Eileen Dolan;Veronica M. Maher.
Carcinogenesis (1984)

165 Citations

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