J. Justin McCormick spends much of his time researching Molecular biology, Xeroderma pigmentosum, Nucleotide excision repair, Cell culture and DNA repair. His Molecular biology study combines topics from a wide range of disciplines, such as Genetics, Biochemistry, DNA, Gene and DNA replication. His research in Xeroderma pigmentosum intersects with topics in Mutation, Cytotoxic T cell, DNA excision and Carcinogen.
His Cell culture research is multidisciplinary, incorporating perspectives in Ploidy, Cell, Fibrosarcoma and Oncogene. His work is dedicated to discovering how DNA repair, DNA damage are connected with Gene mutation and other disciplines. His Fibroblast study incorporates themes from Malignant transformation and Cancer research.
J. Justin McCormick mainly investigates Molecular biology, DNA, Cell culture, Biochemistry and Nucleotide excision repair. His Molecular biology research is multidisciplinary, incorporating elements of Mutation, Genetics, Gene, Transfection and Fibroblast. His Fibroblast research integrates issues from Malignant transformation and Transformation.
He has included themes like Cell and Fibrosarcoma in his Cell culture study. His Nucleotide excision repair research incorporates themes from Xeroderma pigmentosum and DNA synthesis. His study focuses on the intersection of Xeroderma pigmentosum and fields such as Cytotoxic T cell with connections in the field of Cytotoxicity.
His primary scientific interests are in Molecular biology, Cell biology, Cancer research, Malignant transformation and Cell culture. His Molecular biology research is multidisciplinary, relying on both Genetics, DNA, DNA polymerase, Reporter gene and Cell cycle. DNA is a subfield of Biochemistry that J. Justin McCormick studies.
His research on Malignant transformation also deals with topics like
The scientist’s investigation covers issues in Molecular biology, Cancer research, Cell culture, Cell and Mutation. J. Justin McCormick combines Molecular biology and DNA Polymerase Iota in his studies. His Cancer research research includes elements of Ovarian tumor, Ovarian cancer, Distribution and In vivo.
His study in Cell culture is interdisciplinary in nature, drawing from both Fibrosarcoma and Oncogene. His work focuses on many connections between Cell and other disciplines, such as Mutagenesis, that overlap with his field of interest in Fibroblast. His studies deal with areas such as Cell cycle, Protein subunit and DNA, DNA polymerase as well as Mutation.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Frequency of ultraviolet light-induced mutations is higher in xeroderma pigmentosum variant cells than in normal human cells
Veronica M. Maher;Louis M. Ouellette;Rodger D. Curren;J. Justin Mccormick.
Nature (1976)
Hydrogen ion buffers for biological research
Wilfred J. Ferguson;K.I. Braunschweiger;W.R. Braunschweiger;James R. Smith.
Analytical Biochemistry (1980)
DNA excision-repair processes in human cells can eliminate the cytotoxic and mutagenic consequences of ultraviolet irradiation.
Veronica M. Maher;Delia J. Dorney;Alan L. Mendrala;Beate Konze-Thomas.
Mutation Research (1979)
Overview of matrix metalloproteinase expression in cultured human cells.
Troy A. Giambernardi;George M. Grant;Gail P. Taylor;Robert J. Hay.
Matrix Biology (1998)
Evidence from mutation spectra that the UV hypermutability of xeroderma pigmentosum variant cells reflects abnormal, error-prone replication on a template containing photoproducts.
Yi Ching Wang;Veronica M. Maher;David L. Mitchell;J. Justin Mccormick.
Molecular and Cellular Biology (1993)
Cell cycle-dependent strand bias for UV-induced mutations in the transcribed strand of excision repair-proficient human fibroblasts but not in repair-deficient cells.
W. G. Mcgregor;Ruey-Hwa Chen;L. Lukash;V. M. Maher.
Molecular and Cellular Biology (1991)
Amplification and direct nucleotide sequencing of cDNA from the lysate of low numbers of diploid human cells.
Jia-Ling Yang;Veronica M. Maher;J.Justin McCormick.
Gene (1989)
Preferential repair and strand-specific repair of benzo[a]pyrene diol epoxide adducts in the HPRT gene of diploid human fibroblasts
Ruey-Hwa Chen;V. M. Maher;J. Brouwer;P. Van De Putte.
Proceedings of the National Academy of Sciences of the United States of America (1992)
Down-regulation of overexpressed Sp1 protein in human fibrosarcoma cell lines inhibits tumor formation
Zhenjun Lou;Sandra O'Reilly;Hongyan Liang;Veronica M. Maher.
Cancer Research (2005)
Correlation between O6-methylguanine-DNA-methyltransferase activity and resistance of human cells to the cytotoxic and mutagenic effect of N-methyl-N'-nitro-N-nitrosoguanidine.
Jeanne Domoradzki;Anthony E. Pegg;M. Eileen Dolan;Veronica M. Maher.
Carcinogenesis (1984)
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