His scientific interests lie mostly in Cell biology, Apoptosis, Inflammation, Phagocytosis and Immunology. His studies in Cell biology integrate themes in fields like Receptor and Integrin. His study in the field of Programmed cell death is also linked to topics like CD16.
His study in Inflammation is interdisciplinary in nature, drawing from both Cell, Macrophage and Granulocyte. The study incorporates disciplines such as Efferocytosis, Vitronectin, Interleukin 10 and Monocyte in addition to Phagocytosis. His study explores the link between Immunology and topics such as Apoptotic cell clearance that cross with problems in Acquired immune system, Antigen presentation, Innate immune system, Lymphokine and Immune system.
Cell biology, Apoptosis, Immunology, Inflammation and Phagocytosis are his primary areas of study. His work deals with themes such as Receptor, Integrin and Macrophage, Apoptotic cell clearance, which intersect with Cell biology. His study on Apoptosis also encompasses disciplines like
His study on Immunology is mostly dedicated to connecting different topics, such as Idiopathic pulmonary fibrosis. His Proinflammatory cytokine study in the realm of Inflammation connects with subjects such as Eosinophil. In general Phagocytosis, his work in Phagocyte is often linked to CD32 linking many areas of study.
His primary scientific interests are in Cell biology, Inflammation, Apoptosis, Immunology and Programmed cell death. His Cell biology research is multidisciplinary, incorporating perspectives in Receptor and Macrophage, Apoptotic cell clearance. Ian Dransfield interconnects Efferocytosis, Immune system, Phagocytosis and Cytokine in the investigation of issues within Inflammation.
Ian Dransfield focuses mostly in the field of Apoptosis, narrowing it down to topics relating to Cancer research and, in certain cases, B-cell lymphoma, Granulocyte and Leukotriene B4. His work on Antibody, Virus and Influenza A virus as part of general Immunology study is frequently linked to Eosinophil, bridging the gap between disciplines. His Programmed cell death research is multidisciplinary, incorporating elements of In vitro and Peptide.
Ian Dransfield mostly deals with Cell biology, Inflammation, Phagocytosis, Receptor and Receptor tyrosine kinase. His studies deal with areas such as Cell and Angiogenesis as well as Cell biology. His research in Inflammation is mostly concerned with Proinflammatory cytokine.
His Phagocytosis research includes elements of Macrophage, Apoptotic cell clearance and Immune system. His Receptor Protein-Tyrosine Kinases, TYRO3, C-Mer Tyrosine Kinase and Enzyme-linked receptor study, which is part of a larger body of work in Receptor, is frequently linked to ROR1, bridging the gap between disciplines. His Innate immune system study integrates concerns from other disciplines, such as Efferocytosis, Apoptosis, Programmed cell death and Lung.
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A blast from the past: clearance of apoptotic cells regulates immune responses
John Savill;Ian Dransfield;Chris Gregory;Chris Haslett.
Nature Reviews Immunology (2002)
Vitronectin receptor-mediated phagocytosis of cells undergoing apoptosis.
John Savill;Ian Dransfield;Nancy Hogg;Christopher Haslett.
Extracellular matrix proteins protect small cell lung cancer cells against apoptosis: A mechanism for small cell lung cancer growth and drug resistance in vivo
Tariq Sethi;Robert C. Rintoul;Sarah M. Moore;Alison C. MacKinnon.
Nature Medicine (1999)
Divalent cation regulation of the function of the leukocyte integrin LFA-1.
I Dransfield;C Cabañas;A Craig;N Hogg.
Journal of Cell Biology (1992)
Cyclin-dependent kinase inhibitors enhance the resolution of inflammation by promoting inflammatory cell apoptosis.
Adriano G Rossi;Deborah A Sawatzky;Annemieke Walker;Carol Ward.
Nature Medicine (2006)
In vivo fate of the inflammatory macrophage during the resolution of inflammation: inflammatory macrophages do not die locally, but emigrate to the draining lymph nodes.
G. J. Bellingan;H. Caldwell;S. E. M. Howie;I. Dransfield.
Journal of Immunology (1996)
Increased Platelet Binding to Circulating Monocytes in Acute Coronary Syndromes
Jaydeep Sarma;Caterina A. Laan;Shirjel Alam;Ashwani Jha.
Neutrophil apoptosis is associated with a reduction in CD16 (Fc gamma RIII) expression.
I Dransfield;A M Buckle;J S Savill;A McDowall.
Journal of Immunology (1994)
Regulation of Neutrophil Apoptosis by Tumor Necrosis Factor-α: Requirement for TNFR55 and TNFR75 for Induction of Apoptosis In Vitro
Joanna Murray;Joanna Murray;Joanna Murray;Jeffrey A.J. Barbara;Jeffrey A.J. Barbara;Jeffrey A.J. Barbara;Sarah A. Dunkley;Sarah A. Dunkley;Sarah A. Dunkley;Angel F. Lopez;Angel F. Lopez;Angel F. Lopez.
Regulated expression of Mg2+ binding epitope on leukocyte integrin alpha subunits.
I. Dransfield;N. Hogg.
The EMBO Journal (1989)
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