2018 - Fellow of the Royal Society of Edinburgh
2017 - Member of Academia Europaea
Immunology, Cell biology, Apoptosis, Inflammation and Signal transduction are her primary areas of study. Moira K. B. Whyte has included themes like Odds ratio, Disease and Allele in her Immunology study. Her work on TLR2 as part of general Cell biology research is frequently linked to Restriction point, bridging the gap between disciplines.
Her Apoptosis research is multidisciplinary, incorporating elements of Proteases, Phagocytosis and Lung injury. Her Inflammation research includes themes of Receptor, Zebrafish, In vivo and Granulocyte. Her Signal transduction research is multidisciplinary, relying on both NFKB1 and Innate immune system.
Her primary areas of investigation include Immunology, Inflammation, Cell biology, Apoptosis and Innate immune system. Her Immunology study incorporates themes from Lung injury and Lung. The Inflammation study combines topics in areas such as Receptor, Signal transduction, Zebrafish, Granulocyte and In vivo.
Her research in Cell biology intersects with topics in Cell, Neutrophil extracellular traps, Proteases, Transcription factor and HIF1A. She interconnects Cancer research, Phagocytosis, Microbiology and Macrophage in the investigation of issues within Apoptosis. She has included themes like Hypoxia and Hypoxia-inducible factors in her Innate immune system study.
Moira K. B. Whyte focuses on Immunology, Inflammation, Idiopathic pulmonary fibrosis, Lung injury and Cell biology. Her biological study spans a wide range of topics, including Hypoxia and Zebrafish. Her Inflammation research incorporates elements of Efferocytosis, Protein kinase A, Gene knockdown and In vivo.
In her work, Apoptosis is strongly intertwined with Kinase, which is a subfield of Efferocytosis. Her studies in Lung injury integrate themes in fields like Glycogen, ARDS and Downregulation and upregulation. Her study of Signal transduction is a part of Cell biology.
Her scientific interests lie mostly in Immunology, Idiopathic pulmonary fibrosis, Phagocytosis, Inflammation and COPD. Her studies deal with areas such as Glycolysis, Apoptosis and Efferocytosis as well as Immunology. Her work deals with themes such as Cytokine, Neutrophil extracellular traps, Cystic fibrosis, Cystic fibrosis transmembrane conductance regulator and Proinflammatory cytokine, which intersect with Apoptosis.
In her study, Intensive care medicine, Patient discharge and Pharmacology toxicology is inextricably linked to Interstitial lung disease, which falls within the broad field of Idiopathic pulmonary fibrosis. The concepts of her Phagocytosis study are interwoven with issues in Oxidative stress and Bronchoalveolar lavage. Her Inflammation research includes elements of Ivacaftor and Cell biology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Succinate is an inflammatory signal that induces IL-1β through HIF-1α
G. M. Tannahill;A. M. Curtis;J. Adamik;E. M. Palsson-McDermott.
Nature (2013)
The Identification of Markers of Macrophage Differentiation in PMA-Stimulated THP-1 Cells and Monocyte-Derived Macrophages
Marc Daigneault;Julie A. Preston;Helen M. Marriott;Moira K. B. Whyte.
PLOS ONE (2010)
A transgenic zebrafish model of neutrophilic inflammation.
Stephen A. Renshaw;Catherine A. Loynes;Daniel M.I. Trushell;Stone Elworthy.
Blood (2006)
Inhibition of apoptosis and prolongation of neutrophil functional longevity by inflammatory mediators.
Alison Lee;Moira K.B. Whyte;Christopher Haslett.
Journal of Leukocyte Biology (1993)
Inhibition of Ced-3/ICE-related Proteases Does Not Prevent Cell Death Induced by Oncogenes, DNA Damage, or the Bcl-2 Homologue Bak
Nicola J. McCarthy;Moira K.B. Whyte;Christopher S. Gilbert;Gerard I. Evan.
Journal of Cell Biology (1997)
Impairment of function in aging neutrophils is associated with apoptosis.
M. K. B. Whyte;L. C. Meagher;J. Macdermot;C. Haslett.
Journal of Immunology (1993)
Toll-Like Receptor (TLR)2 and TLR4 in Human Peripheral Blood Granulocytes: A Critical Role for Monocytes in Leukocyte Lipopolysaccharide Responses
Ian Sabroe;Elizabeth C. Jones;Lynne R. Usher;Moira K. B. Whyte.
Journal of Immunology (2002)
APOPTOSIS AND THE CELL CYCLE
Gerard I Evan;Lamorna Brown;Moira Whyte;Elizabeth Harrington.
Current Opinion in Cell Biology (1995)
Selective Roles for Toll-Like Receptor (TLR)2 and TLR4 in the Regulation of Neutrophil Activation and Life Span
Ian Sabroe;Lynne R. Prince;Elizabeth C. Jones;Malcolm J. Horsburgh.
Journal of Immunology (2003)
Toll-Like Receptors in Health and Disease: Complex Questions Remain
Ian Sabroe;Robert C. Read;Moira K. B. Whyte;David H. Dockrell.
Journal of Immunology (2003)
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