D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 56 Citations 15,432 95 World Ranking 2707 National Ranking 229

Research.com Recognitions

Awards & Achievements

2018 - Fellow of the Royal Society of Edinburgh

2017 - Member of Academia Europaea

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Internal medicine
  • Enzyme

Immunology, Cell biology, Apoptosis, Inflammation and Signal transduction are her primary areas of study. Moira K. B. Whyte has included themes like Odds ratio, Disease and Allele in her Immunology study. Her work on TLR2 as part of general Cell biology research is frequently linked to Restriction point, bridging the gap between disciplines.

Her Apoptosis research is multidisciplinary, incorporating elements of Proteases, Phagocytosis and Lung injury. Her Inflammation research includes themes of Receptor, Zebrafish, In vivo and Granulocyte. Her Signal transduction research is multidisciplinary, relying on both NFKB1 and Innate immune system.

Her most cited work include:

  • Succinate is an inflammatory signal that induces IL-1β through HIF-1α (1591 citations)
  • The Identification of Markers of Macrophage Differentiation in PMA-Stimulated THP-1 Cells and Monocyte-Derived Macrophages (691 citations)
  • A transgenic zebrafish model of neutrophilic inflammation. (670 citations)

What are the main themes of her work throughout her whole career to date?

Her primary areas of investigation include Immunology, Inflammation, Cell biology, Apoptosis and Innate immune system. Her Immunology study incorporates themes from Lung injury and Lung. The Inflammation study combines topics in areas such as Receptor, Signal transduction, Zebrafish, Granulocyte and In vivo.

Her research in Cell biology intersects with topics in Cell, Neutrophil extracellular traps, Proteases, Transcription factor and HIF1A. She interconnects Cancer research, Phagocytosis, Microbiology and Macrophage in the investigation of issues within Apoptosis. She has included themes like Hypoxia and Hypoxia-inducible factors in her Innate immune system study.

She most often published in these fields:

  • Immunology (50.00%)
  • Inflammation (36.70%)
  • Cell biology (27.98%)

What were the highlights of her more recent work (between 2014-2021)?

  • Immunology (50.00%)
  • Inflammation (36.70%)
  • Idiopathic pulmonary fibrosis (8.26%)

In recent papers she was focusing on the following fields of study:

Moira K. B. Whyte focuses on Immunology, Inflammation, Idiopathic pulmonary fibrosis, Lung injury and Cell biology. Her biological study spans a wide range of topics, including Hypoxia and Zebrafish. Her Inflammation research incorporates elements of Efferocytosis, Protein kinase A, Gene knockdown and In vivo.

In her work, Apoptosis is strongly intertwined with Kinase, which is a subfield of Efferocytosis. Her studies in Lung injury integrate themes in fields like Glycogen, ARDS and Downregulation and upregulation. Her study of Signal transduction is a part of Cell biology.

Between 2014 and 2021, her most popular works were:

  • Genetic variants associated with susceptibility to idiopathic pulmonary fibrosis in people of European ancestry: a genome-wide association study (102 citations)
  • PGE2 production at sites of tissue injury promotes an anti-inflammatory neutrophil phenotype and determines the outcome of inflammation resolution in vivo (77 citations)
  • Delayed neutrophil apoptosis enhances NET formation in cystic fibrosis. (73 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Enzyme

Her scientific interests lie mostly in Immunology, Idiopathic pulmonary fibrosis, Phagocytosis, Inflammation and COPD. Her studies deal with areas such as Glycolysis, Apoptosis and Efferocytosis as well as Immunology. Her work deals with themes such as Cytokine, Neutrophil extracellular traps, Cystic fibrosis, Cystic fibrosis transmembrane conductance regulator and Proinflammatory cytokine, which intersect with Apoptosis.

In her study, Intensive care medicine, Patient discharge and Pharmacology toxicology is inextricably linked to Interstitial lung disease, which falls within the broad field of Idiopathic pulmonary fibrosis. The concepts of her Phagocytosis study are interwoven with issues in Oxidative stress and Bronchoalveolar lavage. Her Inflammation research includes elements of Ivacaftor and Cell biology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Succinate is an inflammatory signal that induces IL-1β through HIF-1α

G. M. Tannahill;A. M. Curtis;J. Adamik;E. M. Palsson-McDermott.
Nature (2013)

2566 Citations

The Identification of Markers of Macrophage Differentiation in PMA-Stimulated THP-1 Cells and Monocyte-Derived Macrophages

Marc Daigneault;Julie A. Preston;Helen M. Marriott;Moira K. B. Whyte.
PLOS ONE (2010)

1141 Citations

A transgenic zebrafish model of neutrophilic inflammation.

Stephen A. Renshaw;Catherine A. Loynes;Daniel M.I. Trushell;Stone Elworthy.
Blood (2006)

941 Citations

Inhibition of apoptosis and prolongation of neutrophil functional longevity by inflammatory mediators.

Alison Lee;Moira K.B. Whyte;Christopher Haslett.
Journal of Leukocyte Biology (1993)

863 Citations

Inhibition of Ced-3/ICE-related Proteases Does Not Prevent Cell Death Induced by Oncogenes, DNA Damage, or the Bcl-2 Homologue Bak

Nicola J. McCarthy;Moira K.B. Whyte;Christopher S. Gilbert;Gerard I. Evan.
Journal of Cell Biology (1997)

700 Citations

Impairment of function in aging neutrophils is associated with apoptosis.

M. K. B. Whyte;L. C. Meagher;J. Macdermot;C. Haslett.
Journal of Immunology (1993)

585 Citations

Toll-Like Receptor (TLR)2 and TLR4 in Human Peripheral Blood Granulocytes: A Critical Role for Monocytes in Leukocyte Lipopolysaccharide Responses

Ian Sabroe;Elizabeth C. Jones;Lynne R. Usher;Moira K. B. Whyte.
Journal of Immunology (2002)

469 Citations

APOPTOSIS AND THE CELL CYCLE

Gerard I Evan;Lamorna Brown;Moira Whyte;Elizabeth Harrington.
Current Opinion in Cell Biology (1995)

442 Citations

Selective Roles for Toll-Like Receptor (TLR)2 and TLR4 in the Regulation of Neutrophil Activation and Life Span

Ian Sabroe;Lynne R. Prince;Elizabeth C. Jones;Malcolm J. Horsburgh.
Journal of Immunology (2003)

433 Citations

Toll-Like Receptors in Health and Disease: Complex Questions Remain

Ian Sabroe;Robert C. Read;Moira K. B. Whyte;David H. Dockrell.
Journal of Immunology (2003)

314 Citations

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