D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 110 Citations 54,354 247 World Ranking 429 National Ranking 282
Medicine D-index 91 Citations 38,132 217 World Ranking 5540 National Ranking 3089

Research.com Recognitions

Awards & Achievements

2017 - Fellow of the American Association for the Advancement of Science (AAAS)

2016 - Fellow, National Academy of Inventors

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Internal medicine

Transforming growth factor, Cell biology, Transforming growth factor beta, Molecular biology and Cancer research are his primary areas of study. His Transforming growth factor research incorporates themes from Cell culture, Epidermal growth factor, Biochemistry, Cell growth and Growth factor. His Cell biology study combines topics in areas such as Endocrinology, Internal medicine, Platelet-derived growth factor receptor and Cellular differentiation.

The various areas that he examines in his Transforming growth factor beta study include Epithelial–mesenchymal transition, Tumor microenvironment, Fibronectin, Transforming growth factor, beta 3 and Adenocarcinoma. His work deals with themes such as Trypsin, Keratinocyte, TGF beta signaling pathway, Receptor and Platelet, which intersect with Molecular biology. The Cancer research study combines topics in areas such as Mammary tumor, Carcinogenesis, Immunology, Metastasis and Tumor progression.

His most cited work include:

  • Stromal fibroblasts in cancer initiation and progression (1820 citations)
  • TGF-beta signaling in fibroblasts modulates the oncogenic potential of adjacent epithelia. (1170 citations)
  • Depletion of Carcinoma-Associated Fibroblasts and Fibrosis Induces Immunosuppression and Accelerates Pancreas Cancer with Reduced Survival. (1155 citations)

What are the main themes of his work throughout his whole career to date?

Harold L. Moses mostly deals with Transforming growth factor, Cancer research, Cell biology, Transforming growth factor beta and Internal medicine. His Transforming growth factor research integrates issues from Cell culture, Epidermal growth factor, Cell growth, Molecular biology and Transforming growth factor, beta 3. His work carried out in the field of Cancer research brings together such families of science as Tumor progression, Cancer and Metastasis.

His study looks at the relationship between Cell biology and fields such as Autocrine signalling, as well as how they intersect with chemical problems. His studies link Cellular differentiation with Transforming growth factor beta. His Internal medicine research includes elements of Endocrinology and Oncology.

He most often published in these fields:

  • Transforming growth factor (38.27%)
  • Cancer research (33.42%)
  • Cell biology (28.83%)

What were the highlights of his more recent work (between 2009-2020)?

  • Cancer research (33.42%)
  • Tumor microenvironment (9.95%)
  • Internal medicine (20.41%)

In recent papers he was focusing on the following fields of study:

Harold L. Moses focuses on Cancer research, Tumor microenvironment, Internal medicine, Cancer and Pathology. His research in Cancer research intersects with topics in Tumor progression, Metastasis, Mammary tumor and Transforming growth factor. He studies Transforming growth factor beta, a branch of Transforming growth factor.

Harold L. Moses interconnects Gastroenterology, Endocrinology, Physical therapy and Oncology in the investigation of issues within Internal medicine. His studies in Cancer integrate themes in fields like Antibody and Immunology. His Pathology research is multidisciplinary, incorporating perspectives in Cancer cell, Colorectal cancer, Laser capture microdissection and Cell growth.

Between 2009 and 2020, his most popular works were:

  • Depletion of Carcinoma-Associated Fibroblasts and Fibrosis Induces Immunosuppression and Accelerates Pancreas Cancer with Reduced Survival. (1155 citations)
  • TGF-β and immune cells: an important regulatory axis in the tumor microenvironment and progression (575 citations)
  • The roles of TGFβ in the tumour microenvironment (523 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Internal medicine

Harold L. Moses mainly focuses on Cancer research, Tumor microenvironment, Stromal cell, Transforming growth factor and Tumor progression. His Cancer research study incorporates themes from Cancer, Metastasis, Endocrinology, Immunology and Transforming growth factor beta. His research investigates the connection with Endocrinology and areas like Cellular differentiation which intersect with concerns in Internal medicine, Prostate, Lewis lung carcinoma and Molecular biology.

His Transforming growth factor beta research includes themes of Epithelial–mesenchymal transition, Cell migration and Carcinogenesis. In his study, Receptor and Cancer cell is inextricably linked to Cell, which falls within the broad field of Transforming growth factor. His studies deal with areas such as TGF beta signaling pathway and Cell type as well as Signal transduction.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Stromal fibroblasts in cancer initiation and progression

Neil A. Bhowmick;Eric G. Neilson;Harold L. Moses.
Nature (2004)

2884 Citations

TGF-beta signaling in fibroblasts modulates the oncogenic potential of adjacent epithelia.

Neil A. Bhowmick;Anna Chytil;David Plieth;Agnieszka E. Gorska.
Science (2004)

1694 Citations

Tumour microenvironment: TGFβ: the molecular Jekyll and Hyde of cancer

Brian Bierie;Harold L. Moses.
Nature Reviews Cancer (2006)

1619 Citations

Depletion of Carcinoma-Associated Fibroblasts and Fibrosis Induces Immunosuppression and Accelerates Pancreas Cancer with Reduced Survival.

Berna C. Özdemir;Berna C. Özdemir;Tsvetelina Pentcheva-Hoang;Julienne L. Carstens;Xiaofeng Zheng.
Cancer Cell (2014)

1455 Citations

TGF-β stimulation and inhibition of cell proliferation: New mechanistic insights

Harold L. Moses;Edmund Y. Yang;Jennifer A. Pietenpol.
Cell (1990)

1404 Citations

Proteolytic activation of latent transforming growth factor-beta from fibroblast-conditioned medium.

R M Lyons;J Keski-Oja;H L Moses.
Journal of Cell Biology (1988)

1331 Citations

Transforming Growth Factor-β1 Mediates Epithelial to Mesenchymal Transdifferentiation through a RhoA-dependent Mechanism

Neil A. Bhowmick;Mayshan Ghiassi;Andrei Bakin;Mary Aakre.
Molecular Biology of the Cell (2001)

1258 Citations

Growth Factors and Cancer

Anton Scott Goustin;Edward B. Leof;Gary D. Shipley;Harold L. Moses.
Cancer Research (1986)

1187 Citations

Phosphatidylinositol 3-Kinase Function Is Required for Transforming Growth Factor β-mediated Epithelial to Mesenchymal Transition and Cell Migration

Andrei V. Bakin;Anne K. Tomlinson;Neil A. Bhowmick;Harold L. Moses.
Journal of Biological Chemistry (2000)

1101 Citations

Stimulation of the chemotactic migration of human fibroblasts by transforming growth factor beta.

A E Postlethwaite;J Keski-Oja;H L Moses;A H Kang.
Journal of Experimental Medicine (1987)

1099 Citations

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