What is he best known for?
The fields of study he is best known for:
His primary scientific interests are in Molecular biology, Cell culture, Cancer research, Internal medicine and Endocrinology.
His Molecular biology research incorporates themes from In vitro, Cancer cell, Frameshift mutation, Trichostatin A and Colchicine.
His studies in Cell culture integrate themes in fields like Carcinoembryonic antigen, Cell growth, Pathology, Transforming growth factor and Cell type.
As a part of the same scientific family, Michael G. Brattain mostly works in the field of Transforming growth factor, focusing on Growth inhibition and, on occasion, Secretion.
His Cancer research research is multidisciplinary, relying on both Cancer, Colorectal cancer, Protein kinase B, Immunology and Transforming growth factor beta.
Michael G. Brattain works mostly in the field of Internal medicine, limiting it down to concerns involving Cellular differentiation and, occasionally, Plasminogen activator, Urokinase receptor, Urokinase and Cell.
His most cited work include:
- Inactivation of the Type II TGF-β Receptor in Colon Cancer Cells with Microsatellite Instability (2137 citations)
- Heterogeneity of Malignant Cells from a Human Colonic Carcinoma (391 citations)
- Mutational Inactivation of Transforming Growth Factor β Receptor Type II in Microsatellite Stable Colon Cancers (376 citations)
What are the main themes of his work throughout his whole career to date?
Michael G. Brattain spends much of his time researching Cancer research, Molecular biology, Transforming growth factor, Cell culture and Internal medicine.
The Cancer research study combines topics in areas such as Cancer, Protein kinase B, Survivin, Apoptosis and Colorectal cancer.
Michael G. Brattain studied Molecular biology and Fibronectin that intersect with Laminin and Retinoic acid.
His biological study deals with issues like Autocrine signalling, which deal with fields such as Cell cycle.
The concepts of his Cell culture study are interwoven with issues in Receptor, Carcinoembryonic antigen, Cellular differentiation and Cell growth.
His Internal medicine study incorporates themes from Endocrinology and Oncology.
He most often published in these fields:
- Cancer research (41.06%)
- Molecular biology (43.90%)
- Transforming growth factor (39.02%)
What were the highlights of his more recent work (between 2007-2021)?
- Cancer research (41.06%)
- Colorectal cancer (13.82%)
- Metastasis (10.57%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Cancer research, Colorectal cancer, Metastasis, XIAP and Apoptosis.
His work carried out in the field of Cancer research brings together such families of science as Protein kinase B, Survivin, Histone deacetylase, Transforming growth factor beta and Kinase.
Michael G. Brattain has researched Histone deacetylase in several fields, including Molecular biology and Pancreatic cancer.
He works mostly in the field of Molecular biology, limiting it down to topics relating to Transfection and, in certain cases, Proto-Oncogene Proteins c-akt, as a part of the same area of interest.
As part of the same scientific family, Michael G. Brattain usually focuses on In vivo, concentrating on Cell culture and intersecting with TUNEL assay.
The study of Internal medicine is intertwined with the study of Endocrinology in a number of ways.
Between 2007 and 2021, his most popular works were:
- Cell survival and metastasis regulation by Akt signaling in colorectal cancer (104 citations)
- MicroRNA-192 suppresses liver metastasis of colon cancer. (97 citations)
- Characterization of HCT116 human colon cancer cells in an orthotopic model. (94 citations)
In his most recent research, the most cited papers focused on:
Michael G. Brattain mostly deals with Cancer research, PI3K/AKT/mTOR pathway, Kinase, Survivin and Metastasis.
His Cancer research study combines topics from a wide range of disciplines, such as Endocrinology, Apoptosis, Insulin-like growth factor 1 receptor, Internal medicine and Transforming growth factor beta.
His research brings together the fields of Cell culture and Apoptosis.
His PI3K/AKT/mTOR pathway study combines topics in areas such as Protein kinase B and Cell growth.
The various areas that Michael G. Brattain examines in his Survivin study include Cell, Downregulation and upregulation, Transfection and Cell biology.
Michael G. Brattain interconnects Colorectal cancer and Immunology in the investigation of issues within Metastasis.
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