D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 96 Citations 48,562 300 World Ranking 5754 National Ranking 3177

Research.com Recognitions

Awards & Achievements

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • DNA

His primary areas of study are Cancer research, Genetics, Mutation, Gene and Colorectal cancer. He has included themes like Carcinogenesis, Cancer, DNA methylation, Molecular biology and Signal transduction in his Cancer research study. His Cancer research integrates issues from Cell, Cell signaling, Immunology, Germline and Pathology.

His Mutation study integrates concerns from other disciplines, such as Transforming growth factor, Cell culture, Microsatellite instability and DNA repair. His DNA mismatch repair, Coding region, Wild type and Gene mutation study in the realm of Gene connects with subjects such as P110δ. His work focuses on many connections between Colorectal cancer and other disciplines, such as Methylation, that overlap with his field of interest in Vimentin.

His most cited work include:

  • High frequency of mutations of the PIK3CA gene in human cancers (2829 citations)
  • Inactivation of the Type II TGF-β Receptor in Colon Cancer Cells with Microsatellite Instability (2137 citations)
  • Suppression of human colorectal carcinoma cell growth by wild-type p53. (1542 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Cancer research, Colorectal cancer, Internal medicine, Cancer and Genetics. His work deals with themes such as Carcinogenesis, Tumor suppressor gene, Mutation, Microsatellite instability and Pathology, which intersect with Cancer research. His Mutation research includes themes of DNA repair and Somatic cell.

His Colorectal cancer research incorporates themes from Biomarker, Molecular biology and Adenoma. He has included themes like Gastroenterology, Endocrinology and Oncology in his Internal medicine study. His work carried out in the field of Cancer brings together such families of science as Immunology and Germline.

He most often published in these fields:

  • Cancer research (56.00%)
  • Colorectal cancer (45.71%)
  • Internal medicine (33.14%)

What were the highlights of his more recent work (between 2017-2021)?

  • Colorectal cancer (45.71%)
  • Internal medicine (33.14%)
  • Cancer research (56.00%)

In recent papers he was focusing on the following fields of study:

Colorectal cancer, Internal medicine, Cancer research, Oncology and Cancer are his primary areas of study. His work in Colorectal cancer addresses subjects such as Genome-wide association study, which are connected to disciplines such as Genetic association, Primary tumor and Carcinogenesis. His study focuses on the intersection of Internal medicine and fields such as Gastroenterology with connections in the field of Prospective cohort study, Celecoxib and Methylation.

His Cancer research study which covers Spleen that intersects with Regeneration. His Oncology research is multidisciplinary, incorporating elements of Esophagus, Genetic risk and Early detection. The Barrett's esophagus study combines topics in areas such as Epithelium, Pathology, Genetically modified mouse and Molecular biology.

Between 2017 and 2021, his most popular works were:

  • Discovery of common and rare genetic risk variants for colorectal cancer (131 citations)
  • Identifying DNA methylation biomarkers for non-endoscopic detection of Barrett's esophagus. (58 citations)
  • Novel Common Genetic Susceptibility Loci for Colorectal Cancer (57 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • DNA

Sanford D. Markowitz focuses on Colorectal cancer, Internal medicine, Cancer research, Adenoma and Gastroenterology. His Colorectal cancer research includes themes of Genome-wide association study, Indel, Case-control study, Wild type and Genotype. The concepts of his Case-control study study are interwoven with issues in Genetics, Allele, Allele frequency and Genetic predisposition.

His work is dedicated to discovering how Internal medicine, Oncology are connected with Breast cancer, Confidence interval, Mendelian Randomization Analysis, Odds ratio and Epidemiology of cancer and other disciplines. Sanford D. Markowitz has researched Cancer research in several fields, including Cell growth, Gene knockdown, Transcriptome, Glutamine and In vivo. In his research, Clinical trial and Receiver operating characteristic is intimately related to Colonoscopy, which falls under the overarching field of Adenoma.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

High frequency of mutations of the PIK3CA gene in human cancers

Yardena Samuels;Zhenghe Wang;Alberto Bardelli;Natalie Silliman.
Science (2004)

3990 Citations

Inactivation of the Type II TGF-β Receptor in Colon Cancer Cells with Microsatellite Instability

Sanford D Markowitz;Jing Wang;Lois Myeroff;Ramon Parsons.
Science (1995)

3015 Citations

Suppression of human colorectal carcinoma cell growth by wild-type p53.

Suzanne J. Baker;Sanford D Markowitz;Eric R. Fearon;James K V Willson.
Science (1990)

2414 Citations

Molecular Basis of Colorectal Cancer

Sanford D. Markowitz;Monica M. Bertagnolli.
The New England Journal of Medicine (2009)

2248 Citations

Mutations of mitotic checkpoint genes in human cancers

Daniel P. Cahill;Christoph Lengauer;Jian Yu;Gregory J. Riggins.
Nature (1998)

1913 Citations

p53 gene mutations occur in combination with 17p allelic deletions as late events in colorectal tumorigenesis.

Suzanne J. Baker;Antonette C. Preisinger;J. M. Jessup;Christos Paraskeva.
Cancer Research (1990)

1464 Citations

KILLER/DR5 is a DNA damage–inducible p53–regulated death receptor gene

G. S. Wu;T. F. Burns;E. R. McDonald;W. Jiang.
Nature Genetics (1997)

1368 Citations

Epigenetic inactivation of SFRP genes allows constitutive WNT signaling in colorectal cancer

Hiromu Suzuki;Hiromu Suzuki;D. Neil Watkins;Kam Wing Jair;Kornel E. Schuebel.
Nature Genetics (2004)

1248 Citations

Somatic mutations of the mitochondrial genome in human colorectal tumours

Kornelia Polyak;Kornelia Polyak;Yunbo Li;Hong Zhu;Christoph Lengauer.
Nature Genetics (1998)

1022 Citations

Microsatellite instability and mutations of the transforming growth factor β type II receptor gene in colorectal cancer

Ramon Parsons;Lois L. Myeroff;Bo Liu;James K. V. Willson.
Cancer Research (1995)

1015 Citations

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