His scientific interests lie mostly in Microglia, Neuroscience, Neuroglia, Innate immune system and Inflammation. His Microglia research incorporates themes from Proinflammatory cytokine, Phagocytosis, Cell biology and Immune system. The Neuroscience study which covers Signal transducing adaptor protein that intersects with Bioinformatics, Disease, Human brain and Regulator.
His Neuroglia study combines topics in areas such as Tumor necrosis factor alpha and Neuroinflammation. His Innate immune system research is multidisciplinary, relying on both TREM2 and Neurodegeneration. He works mostly in the field of Inflammation, limiting it down to concerns involving Multiple sclerosis and, occasionally, Cytotoxicity, Parenchyma, Apoptosis and Cytotoxic T cell.
Harald Neumann spends much of his time researching Microglia, Cell biology, Immunology, Receptor and Neuroscience. Harald Neumann has researched Microglia in several fields, including Neurodegeneration, Molecular biology, Neuroinflammation, Proinflammatory cytokine and Innate immune system. The concepts of his Cell biology study are interwoven with issues in Tumor necrosis factor alpha, Biochemistry and Complement system, Immune system.
His study in Immunology is interdisciplinary in nature, drawing from both Cancer research and Neurotrophin. His work on TREM2 and Signal transducing adaptor protein as part of general Receptor study is frequently linked to Immunoreceptor Tyrosine-Based Inhibition Motif, therefore connecting diverse disciplines of science. His Central nervous system study which covers Neuron that intersects with Major histocompatibility complex and Premovement neuronal activity.
His primary areas of investigation include Cell biology, Microglia, Complement system, Polysialic acid and Neurodegeneration. His Cell biology study incorporates themes from Receptor, Innate immune system, Sialic acid and Neuroinflammation. His Innate immune system course of study focuses on SIGLEC and Factor H.
His Microglia research is classified as research in Immunology. In his work, Complement receptor 1, Macrophage-1 antigen, Integrin alpha M and Neuroscience is strongly intertwined with Opsonin, which is a subfield of Complement system. His Neurodegeneration research incorporates themes from Reactive nitrogen species, Molecular biology and TREM2.
The scientist’s investigation covers issues in Microglia, Cell biology, Molecular biology, Complement system and TREM2. Microglia is a subfield of Immunology that Harald Neumann explores. His Cell biology study integrates concerns from other disciplines, such as Sialic acid and Mononuclear phagocyte system.
His Molecular biology research is multidisciplinary, relying on both Mutation, SURF1, Protein subunit and Genetics. His Complement system research is multidisciplinary, incorporating perspectives in Cell sorting, Plasma protein binding, Antibody opsonization and Chinese hamster ovary cell. In TREM2, Harald Neumann works on issues like Neurodegeneration, which are connected to Receptor, Cytokine, Immune system, Cytokine secretion and Induced pluripotent stem cell.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Integrated Systems Approach Identifies Genetic Nodes and Networks in Late-Onset Alzheimer’s Disease
Bin Zhang;Chris Gaiteri;Liviu Gabriel Bodea;Zhi Wang.
Cell (2013)
Debris clearance by microglia: an essential link between degeneration and regeneration
H P H Neumann;M. R. Kotter;R J M Franklin.
Brain (2008)
Clearance of apoptotic neurons without inflammation by microglial triggering receptor expressed on myeloid cells-2.
Kazuya Takahashi;Christian D.P. Rochford;Harald Neumann.
Journal of Experimental Medicine (2005)
Neuronal ‘On’ and ‘Off’ signals control microglia
Knut Biber;Harald Neumann;Kazuhide Inoue;Hendrikus W.G.M. Boddeke.
Trends in Neurosciences (2007)
Cytotoxic T lymphocytes in autoimmune and degenerative CNS diseases.
Harald Neumann;Isabelle M Medana;Jan Bauer;Hans Lassmann.
Trends in Neurosciences (2002)
Induction of MHC class I genes in neurons
Harald Neumann;Adolfo Cavalié;Dieter E. Jenne;Hartmut Wekerle.
Science (1995)
Constitutive NF-kappa B activity in neurons.
Christian Kaltschmidt;Barbara Kaltschmidt;H. Neumann;H. Wekerle.
Molecular and Cellular Biology (1994)
Janus-faced microglia: beneficial and detrimental consequences of microglial phagocytosis
Amanda Sierra;Oihane Abiega;Anahita Shahraz;Harald Neumann.
Frontiers in Cellular Neuroscience (2013)
TREM2-Transduced Myeloid Precursors Mediate Nervous Tissue Debris Clearance and Facilitate Recovery in an Animal Model of Multiple Sclerosis
Kazuya Takahashi;Marco Prinz;Massimiliano Stagi;Massimiliano Stagi;Olga Chechneva.
PLOS Medicine (2007)
LPS receptor (CD14): a receptor for phagocytosis of Alzheimer’s amyloid peptide
Yang Liu;Silke Walter;Massimiliano Stagi;Dmitry Cherny.
Brain (2005)
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