Hagai Ginsburg mostly deals with Plasmodium falciparum, Biochemistry, Chloroquine, Food vacuole and Host cell cytosol. In the field of Plasmodium falciparum, his study on Plasmodium overlaps with subjects such as Transcriptome. His Biochemistry study is mostly concerned with Red blood cell, Glutathione, Membrane, Oxidative stress and Amino acid.
His work deals with themes such as Quinine, Ion homeostasis, Pharmacology and Vacuole, which intersect with Chloroquine. His research in Food vacuole intersects with topics in Weak base, Ammonium chloride, IC50, Molecular biology and Distribution. His Host cell cytosol research focuses on Hemozoin and how it relates to Cell growth, Phenothiazine, Plasmodium vinckei and Stereochemistry.
Plasmodium falciparum, Biochemistry, Chloroquine, Pharmacology and Malaria are his primary areas of study. His work carried out in the field of Plasmodium falciparum brings together such families of science as Host cell membrane, Mechanism of action, Host cell cytosol, Microbiology and Cell biology. His Red blood cell, Food vacuole, In vitro, Glutathione and Membrane investigations are all subjects of Biochemistry research.
In his research on the topic of Chloroquine, Efflux is strongly related with Drug resistance. His Pharmacology research is multidisciplinary, incorporating elements of Plasmodium vinckei, Mefloquine and In vivo. The concepts of his Malaria study are interwoven with issues in Computational biology and Drug.
Hagai Ginsburg spends much of his time researching Plasmodium falciparum, Biochemistry, Malaria, Pharmacology and Chloroquine. He combines subjects such as Genetics, Hemoglobin, Hemolysis, Microbiology and Lysis with his study of Plasmodium falciparum. The Lysis study combines topics in areas such as Red blood cell and Homeostasis.
His Malaria study incorporates themes from Drug resistance, Drug and Virology. The study incorporates disciplines such as Plasmodium vinckei, In vivo, Mefloquine and Drug discovery in addition to Pharmacology. His Chloroquine research integrates issues from Mechanism of action, Glutathione and Plasmodium berghei.
His scientific interests lie mostly in Plasmodium falciparum, Biochemistry, Malaria, Hemoglobin and Cell biology. His Plasmodium falciparum research includes themes of Genetics, Lysis, Host cell cytosol and Pharmacology. His Biochemistry research is multidisciplinary, incorporating perspectives in Hemolysis and Microbiology.
His work on Plasmodium as part of general Malaria study is frequently linked to Modern medicine, therefore connecting diverse disciplines of science. His Hemoglobin study integrates concerns from other disciplines, such as Amodiaquine, Chloroquine, Mefloquine, 4-Aminoquinoline and Quinine. As part of one scientific family, he deals mainly with the area of Cell biology, narrowing it down to issues related to the Red Cell, and often Osmotic pressure and Red blood cell.
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Oxidative stress in malaria parasite-infected erythrocytes: Host-parasite interactions
Katja Becker;Leann Tilley;Jonathan L. Vennerstrom;David Roberts.
International Journal for Parasitology (2004)
Inhibition of glutathione-dependent degradation of heme by chloroquine and amodiaquine as a possible basis for their antimalarial mode of action.
Hagai Ginsburg;Oleg Famin;Jianmin Zhang;Miriam Krugliak.
Biochemical Pharmacology (1998)
Excess hemoglobin digestion and the osmotic stability ofPlasmodium falciparum–infected red blood cells
Virgilio L. Lew;Teresa Tiffert;Hagai Ginsburg.
Blood (2003)
Intraerythrocytic Plasmodium falciparum utilizes only a fraction of the amino acids derived from the digestion of host cell cytosol for the biosynthesis of its proteins.
Miriam Krugliak;Jianmin Zhang;Hagai Ginsburg.
Molecular and Biochemical Parasitology (2002)
The transcriptome of Plasmodium vivax reveals divergence and diversity of transcriptional regulation in malaria parasites
Zbynek Bozdech;Sachel Mok;Guangan Hu;Mallika Imwong.
Proceedings of the National Academy of Sciences of the United States of America (2008)
Origin of reactive oxygen species in erythrocytes infected with Plasmodium falciparum.
Hani Atamna;Hagai Ginsburg.
Molecular and Biochemical Parasitology (1993)
Heme degradation in the presence of glutathione. A proposed mechanism to account for the high levels of non-heme iron found in the membranes of hemoglobinopathic red blood cells.
Hani Atamna;Hagai Ginsburg.
Journal of Biological Chemistry (1995)
Susceptibility of human malaria parasites to chloroquine is pH dependent.
A Yayon;Z I Cabantchik;H Ginsburg.
Proceedings of the National Academy of Sciences of the United States of America (1985)
Characterization of permeation pathways appearing in the host membrane of Plasmodium falciparum infected red blood cells.
Hagai Ginsburg;Shirley Kutner;Miriam Krugliak;Z. Ioav Cabantchik.
Molecular and Biochemical Parasitology (1985)
New permeability pathways induced in membranes of Plasmodium falciparum infected erythrocytes
Hagai Ginsburg;Miriam Krugliak;Ofer Eidelman;Z. Ioav Cabantchik.
Molecular and Biochemical Parasitology (1983)
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