Michael Lanzer mainly focuses on Plasmodium falciparum, Genetics, Chloroquine, Biochemistry and Cytoplasm. Plasmodium falciparum is a subfield of Malaria that Michael Lanzer investigates. His studies deal with areas such as Phenotype, Amiloride, Efflux, Vacuole and Pharmacology as well as Chloroquine.
His Biochemistry research incorporates themes from Molecular biology and Stimulation. His biological study deals with issues like Organelle, which deal with fields such as Maurer's cleft. His study explores the link between Cell biology and topics such as Protein targeting that cross with problems in Apicomplexa, Host cell cytoplasm and Ultrastructure.
The scientist’s investigation covers issues in Plasmodium falciparum, Malaria, Biochemistry, Genetics and Cell biology. Michael Lanzer interconnects Chloroquine, Pharmacology, Drug resistance and Cytoplasm in the investigation of issues within Plasmodium falciparum. In Chloroquine, Michael Lanzer works on issues like Transporter, which are connected to Xenopus.
His work carried out in the field of Malaria brings together such families of science as Drug and Virology. His study in Gene, Yeast artificial chromosome, Subtelomere, Chromosome and Chromatin is carried out as part of his studies in Genetics. His work investigates the relationship between Immunology and topics such as Cell adhesion that intersect with problems in Receptor.
His primary areas of investigation include Plasmodium falciparum, Malaria, Cell biology, Immunology and Drug resistance. His research integrates issues of Biochemistry, Wild type, Bacterial adhesin, Chloroquine and Pharmacology in his study of Plasmodium falciparum. The Chloroquine study combines topics in areas such as Quinine, Genetics and Transporter.
His study on Quantitative trait locus and Gene is often connected to Peroxiredoxin as part of broader study in Genetics. In his research, Transport Pathway, Secretion, Infected erythrocyte and Mutation is intimately related to Virology, which falls under the overarching field of Malaria. His Cell biology research includes elements of Adhesion, Ubiquitin and Gene expression.
Plasmodium falciparum, Immunology, Malaria, Chloroquine and Genetics are his primary areas of study. Michael Lanzer has included themes like Transport protein, Cell biology, Cell adhesion, Bacterial adhesin and Drug resistance in his Plasmodium falciparum study. Michael Lanzer has researched Cell biology in several fields, including Cytoskeleton, Biophysical Phenomena, Catch bond and Host cell surface.
In his study, which falls under the umbrella issue of Drug resistance, Multiple drug resistance, Membrane transport and Pharmacology is strongly linked to Transporter. His research in Malaria focuses on subjects like Virology, which are connected to Xenopus and Mutation. His Chloroquine course of study focuses on Quinine and Candidate gene, Genetic screen, Ubiquitin ligase, Quantitative trait locus and Quinidine.
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Antigenic variation in malaria: in situ switching, relaxed and mutually exclusive transcription of var genes during intra‐erythrocytic development in Plasmodium falciparum
A. Scherf;R. Hernandez‐Rivas;P. Buffet;E. Bottius.
The EMBO Journal (1998)
Promoters largely determine the efficiency of repressor action
Michael Lanzer;Hermann Bujard.
Proceedings of the National Academy of Sciences of the United States of America (1988)
A T5 promoter-based transcription-translation system for the analysis of proteins in vitro and in vivo
Hermann Bujard;Reiner Gentz;Michael Lanzer;Dietrich Stueber.
Methods in Enzymology (1987)
Drug-resistant malaria: molecular mechanisms and implications for public health.
Ines Petersen;Richard Eastman;Michael Lanzer.
FEBS Letters (2011)
stevor and rif are Plasmodium falciparum multicopy gene families which potentially encode variant antigens.
Qin Cheng;Nicole Cloonan;Katja Fischer;Jenny Thompson.
Molecular and Biochemical Parasitology (1998)
A superfamily of variant genes encoded in the subtelomeric region of Plasmodium vivax
Hernando A. del Portillo;Carmen Fernandez-Becerra;Sharen Bowman;Karen Oliver.
Nature (2001)
Hemoglobins S and C interfere with actin remodeling in Plasmodium falciparum-infected erythrocytes.
Marek Cyrklaff;Cecilia P. Sanchez;Nicole Kilian;Cyrille Bisseye.
Science (2011)
A single member of the Plasmodium falciparum var multigene family determines cytoadhesion to the placental receptor chondroitin sulphate A
Nicola K Viebig;Benoit Gamain;Christine Scheidig;Catherine Lépolard.
EMBO Reports (2005)
Maurer's clefts: A novel multi-functional organelle in the cytoplasm of Plasmodium falciparum-infected erythrocytes
Michael Lanzer;Hannes Wickert;Georg Krohne;Laetitia Vincensini.
International Journal for Parasitology (2006)
Genetic linkage of pfmdr1 with food vacuolar solute import in Plasmodium falciparum
Petra Rohrbach;Cecilia P Sanchez;Karen Hayton;Oliver Friedrich.
The EMBO Journal (2006)
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