D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 56 Citations 15,500 153 World Ranking 9758 National Ranking 708

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Apoptosis

Gerhard Müller-Newen focuses on Cell biology, Immunology, Signal transduction, Cytokine and Cancer research. His studies deal with areas such as Transcription factor and Pathology as well as Cell biology. His Janus kinase study combines topics in areas such as STAT3, SOCS3 and JAK-STAT signaling pathway.

His research integrates issues of Janus kinase 1, Glycoprotein 130 and STAT protein in his study of JAK-STAT signaling pathway. His biological study deals with issues like Leukemia inhibitory factor, which deal with fields such as Suppressor of cytokine signalling and Tyrosine kinase 2. Gerhard Müller-Newen works mostly in the field of Suppressor of cytokine signalling, limiting it down to topics relating to Receptor tyrosine kinase and, in certain cases, Molecular biology, as a part of the same area of interest.

His most cited work include:

  • Principles of interleukin (IL)-6-type cytokine signalling and its regulation. (2470 citations)
  • INTERLEUKIN-6-TYPE CYTOKINE SIGNALLING THROUGH THE GP130/JAK/STAT PATHWAY (1788 citations)
  • Mutations in CLCN2 encoding a voltage-gated chloride channel are associated with idiopathic generalized epilepsies. (314 citations)

What are the main themes of his work throughout his whole career to date?

Gerhard Müller-Newen spends much of his time researching Cell biology, Glycoprotein 130, Molecular biology, Receptor and Signal transduction. As part of the same scientific family, he usually focuses on Cell biology, concentrating on Transcription factor and intersecting with Mutant, Nuclear export signal and Inflammation. His Glycoprotein 130 study combines topics from a wide range of disciplines, such as Oncostatin M and Leukemia inhibitory factor receptor, Leukemia inhibitory factor.

His work in Molecular biology covers topics such as Interleukin 6 which are related to areas like Tumor necrosis factor alpha. Gerhard Müller-Newen combines subjects such as Endocrinology, COS cells and Fusion protein with his study of Receptor. As a part of the same scientific study, Gerhard Müller-Newen usually deals with the Janus kinase, concentrating on JAK-STAT signaling pathway and frequently concerns with Tyrosine kinase 2 and SOCS3.

He most often published in these fields:

  • Cell biology (38.56%)
  • Glycoprotein 130 (28.10%)
  • Molecular biology (25.49%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cell biology (38.56%)
  • Cancer research (13.73%)
  • STAT3 (12.42%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Cell biology, Cancer research, STAT3, Transcription factor and Immunology. Gerhard Müller-Newen interconnects Ribosome and Induced pluripotent stem cell in the investigation of issues within Cell biology. His Cancer research study integrates concerns from other disciplines, such as Myeloid Progenitor Cells, Stem cell, Over expression, Kinase and GLI1.

The Transcription factor study combines topics in areas such as Molecular biology, SH2 domain, STAT5, Phosphorylation and STAT protein. His Molecular biology study incorporates themes from Interleukin 6 and Monoclonal antibody. His work on Proinflammatory cytokine and Cytokine as part of his general Immunology study is frequently connected to Hypereosinophilia and Ulcerative colitis, thereby bridging the divide between different branches of science.

Between 2015 and 2021, his most popular works were:

  • Oncostatin M drives intestinal inflammation and predicts response to tumor necrosis factor-neutralizing therapy in patients with inflammatory bowel disease (270 citations)
  • Gli1+ Mesenchymal Stromal Cells Are a Key Driver of Bone Marrow Fibrosis and an Important Cellular Therapeutic Target (113 citations)
  • Surface Topography Guides Morphology and Spatial Patterning of Induced Pluripotent Stem Cell Colonies (67 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Apoptosis

His primary areas of investigation include Cell biology, Cancer research, Immunology, Oncostatin M and Tumor necrosis factor alpha. His Cell biology research incorporates themes from RNA and RRNA processing. The various areas that Gerhard Müller-Newen examines in his Cancer research study include Haematopoiesis, Cell cycle, Cyclin E1, Cyclin and Kinase.

His Oncostatin M research includes elements of Colitis and Pathology. The concepts of his Tumor necrosis factor alpha study are interwoven with issues in Proinflammatory cytokine and Interleukin 6. His studies in Signal transduction integrate themes in fields like Inflammation, Molecular biology and Transcription factor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Principles of interleukin (IL)-6-type cytokine signalling and its regulation.

Peter C Heinrich;Iris Behrmann;Serge Haan;Heike M Hermanns.
Biochemical Journal (2003)

3801 Citations

INTERLEUKIN-6-TYPE CYTOKINE SIGNALLING THROUGH THE GP130/JAK/STAT PATHWAY

Peter C. Heinrich;Iris Behrmann;Gerhard Müller-Newen;Fred Schaper.
Biochemical Journal (1998)

2719 Citations

Oncostatin M drives intestinal inflammation and predicts response to tumor necrosis factor-neutralizing therapy in patients with inflammatory bowel disease

N R West;A N Hegazy;Owens Bmj.;S J Bullers.
Nature Medicine (2017)

472 Citations

Mutations in CLCN2 encoding a voltage-gated chloride channel are associated with idiopathic generalized epilepsies

Karsten Haug;Maike Warnstedt;Alexi K Alekov;Thomas Sander.
Nature Genetics (2003)

468 Citations

Activation of STAT3 by IL-6 and IL-10 in Primary Human Macrophages Is Differentially Modulated by Suppressor of Cytokine Signaling 3

Claudia Niemand;Ariane Nimmesgern;Serge Haan;Patrick Fischer.
Journal of Immunology (2003)

408 Citations

Transplanted mesenchymal stem cells accelerate glomerular healing in experimental glomerulonephritis.

Uta Kunter;Song Rong;Zivka Djuric;Peter Boor.
Journal of The American Society of Nephrology (2006)

385 Citations

Plasticity and cross-talk of interleukin 6-type cytokines.

Christoph Garbers;Heike M. Hermanns;Fred Schaper;Gerhard Müller-Newen.
Cytokine & Growth Factor Reviews (2012)

336 Citations

Mesenchymal Stem Cells Prevent Progressive Experimental Renal Failure but Maldifferentiate into Glomerular Adipocytes

Uta Kunter;Song Rong;Peter Boor;Frank Eitner.
Journal of The American Society of Nephrology (2007)

291 Citations

Identification and Functional Characterization of Dendritic Cells in the Healthy Murine Kidney and in Experimental Glomerulonephritis

Thilo Krüger;Dirk Benke;Frank Eitner;Andreas Lang.
Journal of The American Society of Nephrology (2004)

283 Citations

Soluble IL-6 Receptor Potentiates the Antagonistic Activity of Soluble gp130 on IL-6 Responses

Gerhard Müller-Newen;Andrea Küster;Ulrike Hemmann;Radovan Keul.
Journal of Immunology (1998)

223 Citations

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