Fazlul H. Sarkar mainly focuses on Cancer research, Cancer, Cell growth, Internal medicine and Cancer cell. His Cancer research research includes elements of Genistein, Pancreatic cancer, Apoptosis, Cancer stem cell and microRNA. His Cancer research is multidisciplinary, relying on both Curcumin and Immunology.
His Cell growth study incorporates themes from Cell culture, Protein kinase B, Signal transduction, Cell biology and Notch 1. His work carried out in the field of Internal medicine brings together such families of science as Endocrinology and Oncology. His Cancer cell research focuses on Pharmacology and how it connects with Cancer therapy and Radiation therapy.
Cancer research, Cancer, Internal medicine, Pancreatic cancer and Cell growth are his primary areas of study. The study incorporates disciplines such as Cancer cell, Apoptosis, Immunology, Metastasis and Prostate cancer in addition to Cancer research. Fazlul H. Sarkar combines subjects such as microRNA and Pharmacology with his study of Cancer.
His research integrates issues of Endocrinology, Oncology and In vivo in his study of Internal medicine. Fazlul H. Sarkar focuses mostly in the field of Pancreatic cancer, narrowing it down to matters related to Pancreas and, in some cases, Pathology. His Cell growth study combines topics from a wide range of disciplines, such as Cell cycle, Signal transduction, Cell biology and Notch 1.
Fazlul H. Sarkar mainly focuses on Cancer research, Cancer, microRNA, Pancreatic cancer and Cancer stem cell. His research in Cancer research intersects with topics in Epithelial–mesenchymal transition, CD44, Cell growth, Cancer cell and Immunology. His Cell growth research is multidisciplinary, incorporating perspectives in Apoptosis and Cell biology.
His research on Cancer concerns the broader Internal medicine. His microRNA research incorporates themes from Carcinogenesis, Downregulation and upregulation, Epigenetics and Bioinformatics. His Pancreatic cancer course of study focuses on Cell culture and Stereochemistry.
Cancer research, Cancer, microRNA, Pancreatic cancer and Cancer stem cell are his primary areas of study. The various areas that Fazlul H. Sarkar examines in his Cancer research study include Epithelial–mesenchymal transition, CD44, Cancer cell, Immunology and Signal transduction. His Cancer research incorporates elements of Angiogenesis and Ubiquitin ligase.
His microRNA research integrates issues from Oncology, Bioinformatics, Carcinogenesis, Internal medicine and Regulation of gene expression. The concepts of his Pancreatic cancer study are interwoven with issues in Cell growth, Apoptosis, Molecular biology, Curcumin and Drug. His biological study spans a wide range of topics, including Downregulation and upregulation and Pathology.
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Exosomes in Cancer Development, Metastasis and Drug Resistance: A Comprehensive Review
Asfar S. Azmi;Bin Bao;Fazlul H. Sarkar.
Cancer and Metastasis Reviews (2013)
Antiintegrin alpha v beta 3 blocks human breast cancer growth and angiogenesis in human skin.
Peter C. Brooks;Staffan Strömblad;Richard Klemke;Daniel Visscher.
Journal of Clinical Investigation (1995)
Up-regulation of miR-200 and let-7 by Natural Agents Leads to the Reversal of Epithelial-to-Mesenchymal Transition in Gemcitabine-Resistant Pancreatic Cancer Cells
Yiwei Li;Timothy G. VandenBoom;Dejuan Kong;Zhiwei Wang.
Cancer Research (2009)
Multi-targeted therapy of cancer by genistein
Sanjeev Banerjee;Yiwei Li;Zhiwei Wang;Fazlul H. Sarkar.
Cancer Letters (2008)
Acquisition of Epithelial-Mesenchymal Transition Phenotype of Gemcitabine-Resistant Pancreatic Cancer Cells Is Linked with Activation of the Notch Signaling Pathway
Zhiwei Wang;Yiwei Li;Dejuan Kong;Sanjeev Banerjee.
Cancer Research (2009)
Phase II randomized clinical trial of lycopene supplementation before radical prostatectomy
Omer Kucuk;Fazlul H. Sarkar;Wael Sakr;Zora Djuric.
Cancer Epidemiology, Biomarkers & Prevention (2001)
Antioxidant effect of zinc in humans.
Ananda S. Prasad;Bin Bao;Frances W.J. Beck;Omer Kucuk.
Free Radical Biology and Medicine (2004)
Gemcitabine sensitivity can be induced in pancreatic cancer cells through modulation of miR-200 and miR-21 expression by curcumin or its analogue CDF.
Shadan Ali;Aamir Ahmad;Sanjeev Banerjee;Subhash Padhye.
Cancer Research (2010)
miR-146a suppresses invasion of pancreatic cancer cells
Yiwei Li;Timothy G. VandenBoom;Zhiwei Wang;Dejuan Kong.
Cancer Research (2010)
Evolving role of uPA/uPAR system in human cancers
Kathleen Dass;Aamir Ahmad;Asfar S. Azmi;Sarah H. Sarkar.
Cancer Treatment Reviews (2008)
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