D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 68 Citations 16,701 161 World Ranking 1337 National Ranking 40

Overview

What is he best known for?

The fields of study he is best known for:

  • Bacteria
  • Gene
  • Internal medicine

David P. Speert mainly focuses on Microbiology, Burkholderia cepacia complex, Pseudomonas aeruginosa, Genomovar and Burkholderia. His Microbiology research incorporates themes from Bacterial genetics, Cystic fibrosis, Virulence and Bacteria. David P. Speert focuses mostly in the field of Burkholderia cepacia complex, narrowing it down to topics relating to Multilocus sequence typing and, in certain cases, Sequence analysis.

His research in Pseudomonas aeruginosa intersects with topics in Efflux, Antibiotic resistance and Immunology, Chronic granulomatous disease. His Immunology research includes elements of Staphylococcus aureus and Pseudomonadaceae. As a part of the same scientific family, David P. Speert mostly works in the field of Genomovar, focusing on Burkholderia multivorans and, on occasion, Burkholderia vietnamiensis, Burkholderia stabilis and Typing.

His most cited work include:

  • Cutting edge: NF-kappaB activating pattern recognition and cytokine receptors license NLRP3 inflammasome activation by regulating NLRP3 expression. (1500 citations)
  • Genetic adaptation by Pseudomonas aeruginosa to the airways of cystic fibrosis patients. (1065 citations)
  • The Cationic Antimicrobial Peptide LL-37 Modulates Dendritic Cell Differentiation and Dendritic Cell-Induced T Cell Polarization (347 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Microbiology, Cystic fibrosis, Immunology, Pseudomonas aeruginosa and Burkholderia cepacia complex. His research integrates issues of Burkholderia multivorans, Virulence and Burkholderia, Bacteria in his study of Microbiology. David P. Speert interconnects Phenotype, Chronic infection, Antibiotic resistance and Virology in the investigation of issues within Virulence.

His work deals with themes such as Dextran, Sputum, Lung and Biofilm, which intersect with Cystic fibrosis. In his research on the topic of Pseudomonas aeruginosa, Antimicrobial is strongly related with Antibiotics. His Burkholderia cepacia complex research is multidisciplinary, incorporating elements of Burkholderia cenocepacia and Multilocus sequence typing.

He most often published in these fields:

  • Microbiology (61.02%)
  • Cystic fibrosis (29.94%)
  • Immunology (29.94%)

What were the highlights of his more recent work (between 2009-2017)?

  • Microbiology (61.02%)
  • Immunology (29.94%)
  • Burkholderia cepacia complex (23.73%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Microbiology, Immunology, Burkholderia cepacia complex, Cystic fibrosis and Pseudomonas aeruginosa. His Microbiology study integrates concerns from other disciplines, such as Burkholderia multivorans, Burkholderia, Virulence and Biofilm. His studies deal with areas such as Tobramycin and Polymyxin as well as Burkholderia cepacia complex.

His biological study spans a wide range of topics, including Chronic infection, Lung and Gene. His Pseudomonas aeruginosa study incorporates themes from Internal medicine and Antibiotics. David P. Speert combines subjects such as Respiratory disease and Clinical significance with his study of Bacteria.

Between 2009 and 2017, his most popular works were:

  • Clinical Features and Outcome of Patients With IRAK-4 and MyD88 Deficiency (288 citations)
  • Parallel Evolution in Pseudomonas aeruginosa over 39,000 Generations In Vivo (96 citations)
  • Profound Lack of Interleukin (IL)-12/IL-23p40 in Neonates Born Early in Gestation Is Associated with an Increased Risk of Sepsis (75 citations)

In his most recent research, the most cited papers focused on:

  • Bacteria
  • Gene
  • Internal medicine

David P. Speert focuses on Microbiology, Cystic fibrosis, Burkholderia cepacia complex, Immunology and Bacteria. His study in Microbiology is interdisciplinary in nature, drawing from both Recombinant DNA, Escherichia coli, Molecular biology, Burkholderia multivorans and Chronic infection. David P. Speert has researched Cystic fibrosis in several fields, including Pseudomonas aeruginosa and Lung.

His Pseudomonas aeruginosa study combines topics in areas such as Infectious disease, Antibiotics and C-reactive protein. His Burkholderia cepacia complex research is multidisciplinary, incorporating perspectives in Epidemiology, Infection control, Mutant, Colistin and Polymyxin B. His research links Virulence with Bacteria.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Cutting edge: NF-kappaB activating pattern recognition and cytokine receptors license NLRP3 inflammasome activation by regulating NLRP3 expression.

Franz G. Bauernfeind;Gabor Horvath;Andrea Stutz;Emad S. Alnemri.
Journal of Immunology (2009)

2339 Citations

Genetic adaptation by Pseudomonas aeruginosa to the airways of cystic fibrosis patients.

Eric E. Smith;Danielle G. Buckley;Zaining Wu;Channakhone Saenphimmachak.
Proceedings of the National Academy of Sciences of the United States of America (2006)

1483 Citations

Antibiotic resistance in Pseudomonas aeruginosa: mechanisms and impact on treatment.

Robert E.W. Hancock;David P. Speert.
Drug Resistance Updates (2000)

557 Citations

The Cationic Antimicrobial Peptide LL-37 Modulates Dendritic Cell Differentiation and Dendritic Cell-Induced T Cell Polarization

Donald J. Davidson;Andrew J. Currie;Gregor S. D. Reid;Dawn M. E. Bowdish.
Journal of Immunology (2004)

532 Citations

Selective predisposition to bacterial infections in IRAK-4–deficient children: IRAK-4–dependent TLRs are otherwise redundant in protective immunity

Cheng Lung Ku;Horst Von Bernuth;Horst Von Bernuth;Capucine Picard;Shen Ying Zhang.
Journal of Experimental Medicine (2007)

436 Citations

Clinical Features and Outcome of Patients With IRAK-4 and MyD88 Deficiency

Capucine Picard;Capucine Picard;Horst Von Bernuth;Horst Von Bernuth;Horst Von Bernuth;Pegah Ghandil;Pegah Ghandil;Pegah Ghandil;Maya Chrabieh;Maya Chrabieh.
Medicine (2010)

425 Citations

Diagnostically and Experimentally Useful Panel of Strains from the Burkholderia cepacia Complex

Eshwar Mahenthiralingam;Tom Coenye;Jacqueline W. Chung;David P. Speert.
Journal of Clinical Microbiology (2000)

387 Citations

Taxon K, a complex within the Burkholderia cepacia complex, comprises at least two novel species, Burkholderia contaminans sp. nov. and Burkholderia lata sp. nov.

Elke Vanlaere;Adam Baldwin;Dirk Gevers;Deborah Henry.
International Journal of Systematic and Evolutionary Microbiology (2009)

342 Citations

Human TLR-7-, -8-, and -9-Mediated Induction of IFN-α/β and -λ Is IRAK-4 Dependent and Redundant for Protective Immunity to Viruses

Kun Yang;Kun Yang;Anne Puel;Shenying Zhang;Shenying Zhang;Céline Eidenschenk.
Immunity (2005)

338 Citations

Multidrug Efflux Systems Play an Important Role in the Invasiveness of Pseudomonas aeruginosa

Yoichi Hirakata;Ramakrishnan Srikumar;Keith Poole;Naomasa Gotoh.
Journal of Experimental Medicine (2002)

332 Citations

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