What is he best known for?
The fields of study he is best known for:
His primary areas of study are Microbiology, Antimicrobial, Antimicrobial peptides, Pseudomonas aeruginosa and Biochemistry.
The various areas that he examines in his Microbiology study include Bacteria, Biofilm, Bacterial outer membrane and Innate immune system.
He combines subjects such as Cationic polymerization, Host, Peptide library and Host defence with his study of Antimicrobial.
His research on Antimicrobial peptides concerns the broader Peptide.
His work in Peptide tackles topics such as Peptide sequence which are related to areas like Amino acid.
His Pseudomonas aeruginosa study also includes fields such as
- Mutant together with Molecular biology,
- Polymyxin B that connect with fields like Indolicidin.
His most cited work include:
- Complete genome sequence of Pseudomonas aeruginosa PAO1, an opportunistic pathogen. (3575 citations)
- Antimicrobial and host-defense peptides as new anti-infective therapeutic strategies. (2716 citations)
- Agar and broth dilution methods to determine the minimal inhibitory concentration (MIC) of antimicrobial substances (2533 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Microbiology, Biochemistry, Pseudomonas aeruginosa, Bacterial outer membrane and Peptide.
His study in Microbiology is interdisciplinary in nature, drawing from both Bacteria, Biofilm and Mutant.
His Pseudomonas aeruginosa study incorporates themes from Polymyxin, Polymyxin B, Swarming motility and Virulence.
Robert E. W. Hancock has researched Bacterial outer membrane in several fields, including Molecular biology, Periplasmic space, Lipopolysaccharide and Membrane protein.
The study incorporates disciplines such as Cationic polymerization and Stereochemistry, Circular dichroism in addition to Peptide.
His research in Antimicrobial is mostly focused on Antimicrobial peptides.
He most often published in these fields:
- Microbiology (36.83%)
- Biochemistry (25.17%)
- Pseudomonas aeruginosa (20.98%)
What were the highlights of his more recent work (between 2014-2021)?
- Microbiology (36.83%)
- Biofilm (8.97%)
- Pseudomonas aeruginosa (20.98%)
In recent papers he was focusing on the following fields of study:
Robert E. W. Hancock focuses on Microbiology, Biofilm, Pseudomonas aeruginosa, Antimicrobial and Peptide.
The Microbiology study combines topics in areas such as Bacteria, Staphylococcus aureus and In vivo.
His Pseudomonas aeruginosa study combines topics from a wide range of disciplines, such as Gene, Mutant, Virulence, Swarming motility and Pseudomonas.
His Innate immune system research extends to Antimicrobial, which is thematically connected.
In Innate immune system, he works on issues like Cystic fibrosis, which are connected to Immunology.
His Peptide research is included under the broader classification of Biochemistry.
Between 2014 and 2021, his most popular works were:
- Alternatives to antibiotics—a pipeline portfolio review (396 citations)
- NetworkAnalyst for statistical, visual and network-based meta-analysis of gene expression data (374 citations)
- The immunology of host defence peptides: beyond antimicrobial activity (367 citations)
In his most recent research, the most cited papers focused on:
Robert E. W. Hancock mainly focuses on Microbiology, Biofilm, Antimicrobial, Antibiotics and Peptide.
His studies deal with areas such as Bacteria, Pseudomonas aeruginosa, Staphylococcus aureus, Gram-negative bacteria and In vivo as well as Microbiology.
His biological study spans a wide range of topics, including Membrane and Cell membrane.
Robert E. W. Hancock is interested in Antimicrobial peptides, which is a branch of Antimicrobial.
He has included themes like Biofilm growth, Structure–activity relationship and Biotechnology in his Antibiotics study.
His Peptide study deals with the bigger picture of Biochemistry.
This overview was generated by a machine learning system which analysed the scientist’s
body of work. If you have any feedback, you can
contact us
here.
