D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 58 Citations 9,654 210 World Ranking 2344 National Ranking 202

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Bacteria
  • DNA

Craig Winstanley focuses on Pseudomonas aeruginosa, Microbiology, Cystic fibrosis, Immunology and Virulence. The various areas that Craig Winstanley examines in his Pseudomonas aeruginosa study include Genotyping, Genotype, Respiratory infection, Antibiotic resistance and Genetic variation. His Microbiology study combines topics from a wide range of disciplines, such as Genetics, Gene, Genetic diversity, Biofilm and Tobramycin.

His research integrates issues of Chronic infection, Pyocyanin and Outpatient clinic in his study of Cystic fibrosis. His work on Spirometry is typically connected to Respiratory equipment as part of general Immunology study, connecting several disciplines of science. His research investigates the connection with Virulence and areas like Pathogen which intersect with concerns in Regulon, Frameshift mutation, Pilin and Keratitis.

His most cited work include:

  • Pseudomonas genomes: diverse and adaptable (487 citations)
  • Spread of β-lactam-resistant Pseudomonas aeruginosa in a cystic fibrosis clinic (340 citations)
  • Pseudomonas aeruginosa Evolutionary Adaptation and Diversification in Cystic Fibrosis Chronic Lung Infections (330 citations)

What are the main themes of his work throughout his whole career to date?

The scientist’s investigation covers issues in Microbiology, Pseudomonas aeruginosa, Cystic fibrosis, Genetics and Gene. His Microbiology study integrates concerns from other disciplines, such as Biofilm, Bacteria, Genotype and Virulence. His research investigates the connection between Pseudomonas aeruginosa and topics such as Antibiotics that intersect with problems in Tobramycin.

His biological study spans a wide range of topics, including Phenotype, Pyocyanin, Lung and Pathology. His work in Genome, Flagellin, Multilocus sequence typing, Plasmid and Restriction fragment length polymorphism are all subfields of Genetics research. His work deals with themes such as Marker gene and Recombinant DNA, which intersect with Plasmid.

He most often published in these fields:

  • Microbiology (65.50%)
  • Pseudomonas aeruginosa (55.81%)
  • Cystic fibrosis (38.37%)

What were the highlights of his more recent work (between 2017-2021)?

  • Pseudomonas aeruginosa (55.81%)
  • Microbiology (65.50%)
  • Cystic fibrosis (38.37%)

In recent papers he was focusing on the following fields of study:

Craig Winstanley mainly focuses on Pseudomonas aeruginosa, Microbiology, Cystic fibrosis, Genetics and Antibiotic resistance. The Pseudomonas aeruginosa study combines topics in areas such as Antibiotics and Antimicrobial. His Microbiology research includes themes of Biofilm and Type three secretion system, Virulence.

His Cystic fibrosis research is multidisciplinary, relying on both Gene expression and Lung infection. His Lung infection research integrates issues from National registry, Ivacaftor and Immunology. In his study, Genetic structure, Chronic infection, Lineage, Genetic diversity and Genotyping is strongly linked to Strain, which falls under the umbrella field of Antibiotic resistance.

Between 2017 and 2021, his most popular works were:

  • Genomic characterisation of an international Pseudomonas aeruginosa reference panel indicates that the two major groups draw upon distinct mobile gene pools (28 citations)
  • Ivacaftor Is Associated with Reduced Lung Infection by Key Cystic Fibrosis Pathogens. A Cohort Study Using National Registry Data. (25 citations)
  • A megaplasmid family driving dissemination of multidrug resistance in Pseudomonas (18 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Bacteria
  • DNA

His primary areas of investigation include Pseudomonas aeruginosa, Genetics, Antibiotic resistance, Antibiotics and Microbiology. His Pseudomonas aeruginosa research incorporates elements of Internal medicine and Cystic fibrosis. His study in Cystic fibrosis is interdisciplinary in nature, drawing from both National registry, Downregulation and upregulation, Cohort study and Lung infection.

The concepts of his Antibiotic resistance study are interwoven with issues in Mutation, Exotoxin, Tobramycin and Keratitis. His Antibiotics research incorporates themes from Phage therapy, Eye infection and Virulence. Microbiology is closely attributed to Biofilm in his work.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Pseudomonas genomes: diverse and adaptable

Mark W. Silby;Mark W. Silby;Craig Winstanley;Scott A.C. Godfrey;Stuart B. Levy.
Fems Microbiology Reviews (2011)

843 Citations

Pseudomonas aeruginosa Evolutionary Adaptation and Diversification in Cystic Fibrosis Chronic Lung Infections

Craig Winstanley;Siobhan O’Brien;Michael A. Brockhurst.
Trends in Microbiology (2016)

569 Citations

Spread of beta-lactam-resistant Pseudomonas aeruginosa in a cystic fibrosis clinic.

K. Cheng;R. L. Smyth;J. R. W. Govan;C. Doherty.
The Lancet (1996)

483 Citations

Newly introduced genomic prophage islands are critical determinants of in vivo competitiveness in the Liverpool Epidemic Strain of Pseudomonas aeruginosa.

Craig Winstanley;Morgan G.I. Langille;Joanne L. Fothergill;Irena Kukavica-Ibrulj.
Genome Research (2009)

385 Citations

Pseudomonas aeruginosa Population Diversity and Turnover in Cystic Fibrosis Chronic Infections

Eilidh Mowat;Steve Paterson;Joanne L. Fothergill;Joanne L. Fothergill;Elli A. Wright.
American Journal of Respiratory and Critical Care Medicine (2011)

281 Citations

A Cystic Fibrosis Epidemic Strain of Pseudomonas aeruginosa Displays Enhanced Virulence and Antimicrobial Resistance

Prabhakar Salunkhe;Catherine H. M. Smart;J. Alun W. Morgan;Stavroula Panagea.
Journal of Bacteriology (2005)

255 Citations

Colonic mucosa-associated diffusely adherent afaC+ Escherichia coli expressing lpfA and pks are increased in inflammatory bowel disease and colon cancer

Maelle Prorok-Hamon;Melissa K Friswell;Abdullah Alswied;Carol L Roberts.
Gut (2014)

220 Citations

The role of quorum sensing in chronic cystic fibrosis Pseudomonas aeruginosa infections.

Craig Winstanley;Craig Winstanley;Joanne L. Fothergill;Joanne L. Fothergill.
Fems Microbiology Letters (2009)

213 Citations

Increased morbidity associated with chronic infection by an epidemic Pseudomonas aeruginosa strain in CF patients

M Al-Aloul;J Crawley;C Winstanley;C A Hart.
Thorax (2004)

213 Citations

Distribution of type III secretion gene clusters in Burkholderia pseudomallei, B. thailandensis and B. mallei.

Lucille Rainbow;C. Anthony Hart;Craig Winstanley.
Journal of Medical Microbiology (2002)

186 Citations

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