His primary areas of study are Immunology, Tumor necrosis factor alpha, Intestinal mucosa, In situ hybridization and Inflammation. His Immunology study combines topics from a wide range of disciplines, such as Inflammatory bowel disease and Microbiology. His work carried out in the field of Tumor necrosis factor alpha brings together such families of science as Enzyme activator and Pathogenesis.
His research integrates issues of Hormone, Antigen and Intraepithelial lymphocyte in his study of Intestinal mucosa. His In situ hybridization research also works with subjects such as
Christoph Mueller focuses on Immunology, Inflammation, Tumor necrosis factor alpha, Molecular biology and Cytotoxic T cell. The Immunology study combines topics in areas such as Receptor and Intestinal mucosa. His study focuses on the intersection of Inflammation and fields such as Cytokine with connections in the field of Chemokine.
In his work, Cell biology is strongly intertwined with Apoptosis, which is a subfield of Tumor necrosis factor alpha. His Molecular biology study integrates concerns from other disciplines, such as Gene, Transgene, CTL* and Monoclonal antibody. As a part of the same scientific family, he mostly works in the field of Cytotoxic T cell, focusing on CD8 and, on occasion, Major histocompatibility complex.
Christoph Mueller mostly deals with Immunology, Inflammation, Colitis, Immune system and Innate immune system. His work deals with themes such as Receptor, Intestinal mucosa and Disease, which intersect with Immunology. The various areas that Christoph Mueller examines in his Intestinal mucosa study include Tumor necrosis factor alpha, Mucus layer, Gut flora, Genetic model and Effector.
His Inflammation study also includes
Cytokine and related Dendritic cell and Cell activation,
Monocyte differentiation most often made with reference to Cancer research. His study on Colitis also encompasses disciplines like
Genetically modified mouse which is related to area like Parkinson's disease and Inflammatory bowel disease,
Pathogenesis which is related to area like Interleukin 17. His Immune system research integrates issues from Proinflammatory cytokine, PI3K/AKT/mTOR pathway and Rheumatoid arthritis.
His scientific interests lie mostly in Immunology, Innate immune system, Immune system, T cell and Inflammation. His research on Immunology often connects related topics like Mucus layer. His biological study spans a wide range of topics, including Acquired immune system, Gut flora, Intestinal mucosa, Genetic model and Gastrointestinal tract.
His T cell study combines topics in areas such as Receptor, Colitis and Sepsis. Christoph Mueller interconnects Stroke, Spleen, Integrin alpha M and Ischemia in the investigation of issues within Proinflammatory cytokine. His Cytokine research includes elements of Myeloid, Foam cell, Monocytosis, Monocyte and Monocyte differentiation.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
TREM-1–expressing intestinal macrophages crucially amplify chronic inflammation in experimental colitis and inflammatory bowel diseases
Mirjam Schenk;Axel Bouchon;Frank Seibold;Christoph Mueller.
Journal of Clinical Investigation (2007)
Expression of interleukin-8 gene in inflammatory bowel disease is related to the histological grade of active inflammation.
L Mazzucchelli;C Hauser;K Zgraggen;H Wagner.
American Journal of Pathology (1994)
EARLY EVENTS IN T-CELL MATURATION
B. Adkins;C. Mueller;C. Y. Okada;R. A. Reichert.
Annual Review of Immunology (1987)
Localized expression of mRNA for phagocyte-specific chemotactic cytokines in human periodontal infections
M S Tonetti;M A Imboden;L Gerber;N P Lang.
Infection and Immunity (1994)
Expression of perforin and granzymes in vivo: potential diagnostic markers for activated cytotoxic cells
Gillian M Griffiths;Christoph Mueller.
Immunology Today (1991)
Inflammation-associated Cell Cycle–independent Block of Apoptosis by Survivin in Terminally Differentiated Neutrophils
Frank Altznauer;Sibylla Martinelli;Shida Yousefi;Christine Thürig.
Journal of Experimental Medicine (2004)
Differential in situ expression of the genes encoding the chemokines MCP-1 and RANTES in human inflammatory bowel disease
Luca Mazzucchelli;Chantal Hauser;Kaspar Zgraggen;Hans E. Wagner.
The Journal of Pathology (1996)
Genes encoding tumor necrosis factor alpha and granzyme A are expressed during development of autoimmune diabetes.
W Held;H R MacDonald;I L Weissman;M W Hess.
Proceedings of the National Academy of Sciences of the United States of America (1990)
Nonlymphocyte-Derived Tumor Necrosis Factor Is Required for Induction of Colitis in Recombination Activating Gene (Rag)2−/− Mice upon Transfer of Cd4+Cd45rbhi T Cells
Nadia Corazza;Susanne Eichenberger;Hans-Pietro Eugster;Christoph Mueller.
Journal of Experimental Medicine (1999)
A high proportion of T lymphocytes that infiltrate H-2-incompatible heart allografts in vivo express genes encoding cytotoxic cell-specific serine proteases, but do not express the MEL-14-defined lymph node homing receptor.
C Mueller;H K Gershenfeld;C G Lobe;C Y Okada.
Journal of Experimental Medicine (1988)
Profile was last updated on December 6th, 2021.
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