The scientist’s investigation covers issues in Immunology, Antigen, Immune system, Inflammatory bowel disease and T cell. His biological study spans a wide range of topics, including Cytotoxic T cell and Homeostasis. His Antigen research incorporates elements of Antibody and Flagellin.
His study in Immune system is interdisciplinary in nature, drawing from both Inflammation, Gut flora and Bone marrow. In his study, Signal transduction, SOCS3, Paracrine signalling and Cell biology is inextricably linked to Cell growth, which falls within the broad field of T cell. Yingzi Cong combines subjects such as Molecular biology and IL-2 receptor with his study of Interleukin 21.
His primary scientific interests are in Immunology, Immune system, Cell biology, Colitis and Inflammatory bowel disease. Many of his studies on Immunology apply to Intestinal mucosa as well. His Immune system research is multidisciplinary, incorporating perspectives in Gut flora and Microbiology.
His work carried out in the field of Cell biology brings together such families of science as Cell, Epithelium, Cellular differentiation and FOXP3. Yingzi Cong focuses mostly in the field of Colitis, narrowing it down to matters related to Molecular biology and, in some cases, CD40, Interleukin 12, Interleukin 21, Antigen-presenting cell and Natural killer T cell. The various areas that Yingzi Cong examines in his Inflammatory bowel disease study include Tumor necrosis factor alpha, Microbiome, Cancer research, Inflammation and Ulcerative colitis.
His main research concerns Cell biology, Immune system, Cancer research, Inflammation and Inflammatory bowel disease. His research on Cell biology also deals with topics like
His Immunology study combines topics from a wide range of disciplines, such as Microbiome and Intestinal inflammation. His work deals with themes such as Oral administration, Intestinal mucosa, Cellular differentiation, Colorectal cancer and Flow cytometry, which intersect with Cancer research. Yingzi Cong combines subjects such as Proinflammatory cytokine, Endocrinology, Bromodomain and Ulcerative colitis with his study of Inflammatory bowel disease.
His scientific interests lie mostly in Cell biology, Immune system, Colitis, Gut flora and Inflammatory bowel disease. His Cell biology research focuses on subjects like Interleukin 10, which are linked to Antigen and Cell growth. His Immune system study results in a more complete grasp of Immunology.
His Colitis research incorporates themes from Proinflammatory cytokine, Inflammation and Intestinal mucosa. His Gut flora research includes themes of Receptor, Dendritic cell, Cholera toxin and Homeostasis. Yingzi Cong has researched Inflammatory bowel disease in several fields, including Cancer research and Intestinal epithelium.
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Bacterial flagellin is a dominant antigen in Crohn disease
Michael J. Lodes;Yingzi Cong;Charles O. Elson;Raodoh Mohamath.
Journal of Clinical Investigation (2004)
Experimental Models of Inflammatory Bowel Disease Reveal Innate, Adaptive, and Regulatory Mechanisms of Host Dialogue With the Microbiota
Charles O. Elson;Yingzi Cong;Vance J. McCracken;Reed A. Dimmitt.
Immunological Reviews (2005)
Monoclonal anti-interleukin 23 reverses active colitis in a T cell-mediated model in mice.
Charles O. Elson;Yingzi Cong;Casey T. Weaver;Trenton R. Schoeb.
Gastroenterology (2007)
Antibodies to CBir1 flagellin define a unique response that is associated independently with complicated Crohn's disease.
Stephan R. Targan;Carol J. Landers;Huiying Yang;Michael J. Lodes;Michael J. Lodes.
Gastroenterology (2005)
CD4+ T Cells Reactive to Enteric Bacterial Antigens in Spontaneously Colitic C3H/HeJBir Mice: Increased T Helper Cell Type 1 Response and Ability to Transfer Disease
Yingzi Cong;Steven L. Brandwein;Robert P. McCabe;A. Lazenby.
Journal of Experimental Medicine (1998)
Microbiota metabolite short chain fatty acids, GPCR, and inflammatory bowel diseases.
Mingming Sun;Mingming Sun;Wei Wu;Wei Wu;Zhanju Liu;Yingzi Cong.
Journal of Gastroenterology (2017)
Tumor Exosomes Inhibit Differentiation of Bone Marrow Dendritic Cells
Shaohua Yu;Cunren Liu;Kaihong Su;Jianhua Wang.
Journal of Immunology (2007)
A dominant, coordinated T regulatory cell-IgA response to the intestinal microbiota
Yingzi Cong;Ting Feng;Kohtaro Fujihashi;Trenton R. Schoeb.
Proceedings of the National Academy of Sciences of the United States of America (2009)
Hapten-induced model of murine inflammatory bowel disease: mucosa immune responses and protection by tolerance.
C O Elson;K W Beagley;A T Sharmanov;K Fujihashi.
Journal of Immunology (1996)
Treatment of murine CD5- B cells with anti-Ig, but not LPS, induces surface CD5: two B-cell activation pathways
Yingzi Cong;Evelyn Rabin;Henry H. Wortis.
International Immunology (1991)
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