D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 55 Citations 12,824 168 World Ranking 2827 National Ranking 1336

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Cytokine

The scientist’s investigation covers issues in Immunology, Antigen, Immune system, Inflammatory bowel disease and T cell. His biological study spans a wide range of topics, including Cytotoxic T cell and Homeostasis. His Antigen research incorporates elements of Antibody and Flagellin.

His study in Immune system is interdisciplinary in nature, drawing from both Inflammation, Gut flora and Bone marrow. In his study, Signal transduction, SOCS3, Paracrine signalling and Cell biology is inextricably linked to Cell growth, which falls within the broad field of T cell. Yingzi Cong combines subjects such as Molecular biology and IL-2 receptor with his study of Interleukin 21.

His most cited work include:

  • Bacterial flagellin is a dominant antigen in Crohn disease (669 citations)
  • Experimental Models of Inflammatory Bowel Disease Reveal Innate, Adaptive, and Regulatory Mechanisms of Host Dialogue With the Microbiota (423 citations)
  • Antibodies to CBir1 flagellin define a unique response that is associated independently with complicated Crohn's disease. (404 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Immunology, Immune system, Cell biology, Colitis and Inflammatory bowel disease. Many of his studies on Immunology apply to Intestinal mucosa as well. His Immune system research is multidisciplinary, incorporating perspectives in Gut flora and Microbiology.

His work carried out in the field of Cell biology brings together such families of science as Cell, Epithelium, Cellular differentiation and FOXP3. Yingzi Cong focuses mostly in the field of Colitis, narrowing it down to matters related to Molecular biology and, in some cases, CD40, Interleukin 12, Interleukin 21, Antigen-presenting cell and Natural killer T cell. The various areas that Yingzi Cong examines in his Inflammatory bowel disease study include Tumor necrosis factor alpha, Microbiome, Cancer research, Inflammation and Ulcerative colitis.

He most often published in these fields:

  • Immunology (51.09%)
  • Immune system (28.26%)
  • Cell biology (22.83%)

What were the highlights of his more recent work (between 2017-2021)?

  • Cell biology (22.83%)
  • Immune system (28.26%)
  • Cancer research (10.33%)

In recent papers he was focusing on the following fields of study:

His main research concerns Cell biology, Immune system, Cancer research, Inflammation and Inflammatory bowel disease. His research on Cell biology also deals with topics like

  • Colitis, Cd4 t cell, Antigen and Cell growth most often made with reference to Interleukin 10,
  • Epithelium which connect with Carcinogenesis, Stromal cell, Transcription factor, Hippo signaling and Wound healing. Immune system is a subfield of Immunology that Yingzi Cong explores.

His Immunology study combines topics from a wide range of disciplines, such as Microbiome and Intestinal inflammation. His work deals with themes such as Oral administration, Intestinal mucosa, Cellular differentiation, Colorectal cancer and Flow cytometry, which intersect with Cancer research. Yingzi Cong combines subjects such as Proinflammatory cytokine, Endocrinology, Bromodomain and Ulcerative colitis with his study of Inflammatory bowel disease.

Between 2017 and 2021, his most popular works were:

  • Microbiota-derived short-chain fatty acids promote Th1 cell IL-10 production to maintain intestinal homeostasis (108 citations)
  • GPR43 mediates microbiota metabolite SCFA regulation of antimicrobial peptide expression in intestinal epithelial cells via activation of mTOR and STAT3 (91 citations)
  • CD177+ neutrophils as functionally activated neutrophils negatively regulate IBD. (59 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Cytokine

His scientific interests lie mostly in Cell biology, Immune system, Colitis, Gut flora and Inflammatory bowel disease. His Cell biology research focuses on subjects like Interleukin 10, which are linked to Antigen and Cell growth. His Immune system study results in a more complete grasp of Immunology.

His Colitis research incorporates themes from Proinflammatory cytokine, Inflammation and Intestinal mucosa. His Gut flora research includes themes of Receptor, Dendritic cell, Cholera toxin and Homeostasis. Yingzi Cong has researched Inflammatory bowel disease in several fields, including Cancer research and Intestinal epithelium.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Bacterial flagellin is a dominant antigen in Crohn disease

Michael J. Lodes;Yingzi Cong;Charles O. Elson;Raodoh Mohamath.
Journal of Clinical Investigation (2004)

864 Citations

Experimental Models of Inflammatory Bowel Disease Reveal Innate, Adaptive, and Regulatory Mechanisms of Host Dialogue With the Microbiota

Charles O. Elson;Yingzi Cong;Vance J. McCracken;Reed A. Dimmitt.
Immunological Reviews (2005)

625 Citations

Monoclonal anti-interleukin 23 reverses active colitis in a T cell-mediated model in mice.

Charles O. Elson;Yingzi Cong;Casey T. Weaver;Trenton R. Schoeb.
Gastroenterology (2007)

583 Citations

Antibodies to CBir1 flagellin define a unique response that is associated independently with complicated Crohn's disease.

Stephan R. Targan;Carol J. Landers;Huiying Yang;Michael J. Lodes;Michael J. Lodes.
Gastroenterology (2005)

559 Citations

CD4+ T Cells Reactive to Enteric Bacterial Antigens in Spontaneously Colitic C3H/HeJBir Mice: Increased T Helper Cell Type 1 Response and Ability to Transfer Disease

Yingzi Cong;Steven L. Brandwein;Robert P. McCabe;A. Lazenby.
Journal of Experimental Medicine (1998)

550 Citations

Microbiota metabolite short chain fatty acids, GPCR, and inflammatory bowel diseases.

Mingming Sun;Mingming Sun;Wei Wu;Wei Wu;Zhanju Liu;Yingzi Cong.
Journal of Gastroenterology (2017)

521 Citations

Tumor Exosomes Inhibit Differentiation of Bone Marrow Dendritic Cells

Shaohua Yu;Cunren Liu;Kaihong Su;Jianhua Wang.
Journal of Immunology (2007)

437 Citations

A dominant, coordinated T regulatory cell-IgA response to the intestinal microbiota

Yingzi Cong;Ting Feng;Kohtaro Fujihashi;Trenton R. Schoeb.
Proceedings of the National Academy of Sciences of the United States of America (2009)

410 Citations

Hapten-induced model of murine inflammatory bowel disease: mucosa immune responses and protection by tolerance.

C O Elson;K W Beagley;A T Sharmanov;K Fujihashi.
Journal of Immunology (1996)

308 Citations

Treatment of murine CD5- B cells with anti-Ig, but not LPS, induces surface CD5: two B-cell activation pathways

Yingzi Cong;Evelyn Rabin;Henry H. Wortis.
International Immunology (1991)

299 Citations

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