What is he best known for?
The fields of study he is best known for:
- Immune system
- Gene
- Antibody
The scientist’s investigation covers issues in Immunology, Cell biology, T cell, Mycobacterium tuberculosis and Antigen.
His Immunology study integrates concerns from other disciplines, such as Immune control and Clinical trial.
His Cell biology research focuses on Apoptosis and how it connects with Necrosis, Macrophage, Efferocytosis and Phagocytosis.
His T cell study typically links adjacent topics like Cytotoxic T cell.
His work carried out in the field of Mycobacterium tuberculosis brings together such families of science as Immune system, Interferon gamma and Immunity.
His work is dedicated to discovering how Antigen, Antigen presentation are connected with Major histocompatibility complex, Molecular biology and Endosome and other disciplines.
His most cited work include:
- Recognition of a lipid antigen by CD1-restricted αβ+ T cells (868 citations)
- Natural killer T cells recognize diacylglycerol antigens from pathogenic bacteria. (541 citations)
- Murine CD1d-Restricted T Cell Recognition of Cellular Lipids (446 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Immunology, Mycobacterium tuberculosis, T cell, Cytotoxic T cell and Antigen.
His Mycobacterium tuberculosis study combines topics in areas such as Genotype, Lung, Microbiology and Virology.
His research integrates issues of Necrosis and Apoptosis in his study of Microbiology.
His T cell course of study focuses on Cell biology and Antigen presentation and CD1.
The Cytotoxic T cell study combines topics in areas such as Epitope and CD8.
His Antigen research incorporates elements of Glycolipid, Biochemistry and Cellular differentiation.
He most often published in these fields:
- Immunology (56.67%)
- Mycobacterium tuberculosis (38.00%)
- T cell (38.00%)
What were the highlights of his more recent work (between 2016-2021)?
- Mycobacterium tuberculosis (38.00%)
- Immunology (56.67%)
- Tuberculosis (28.00%)
In recent papers he was focusing on the following fields of study:
Mycobacterium tuberculosis, Immunology, Tuberculosis, T cell and Cytotoxic T cell are his primary areas of study.
His Mycobacterium tuberculosis research integrates issues from Flow cytometry, Innate immune system, Lung and Effector.
Samuel M. Behar interconnects Disease, Immunity and T-cell receptor in the investigation of issues within Tuberculosis.
His T cell research includes themes of Quantitative trait locus, Genotype, Mutation and Microbiology.
His studies in Cytotoxic T cell integrate themes in fields like CD8, Immune system and Antigen.
The various areas that Samuel M. Behar examines in his Immune system study include Antibody and Virology.
Between 2016 and 2021, his most popular works were:
- Role of Granulocyte-Macrophage Colony-Stimulating Factor Production by T Cells during Mycobacterium tuberculosis Infection. (35 citations)
- Mycobacterium tuberculosis-specific CD4+ and CD8+ T cells differ in their capacity to recognize infected macrophages. (33 citations)
- TRAV1-2+ CD8+ T-cells including oligoconal expansions of MAIT cells are enriched in the airways in human tuberculosis (32 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Immune system
- Antibody
His primary scientific interests are in Immunology, Mycobacterium tuberculosis, Tuberculosis, T cell and Immune system.
Samuel M. Behar has researched Tuberculosis in several fields, including Acquired immune system, Disease, Cell growth and T-cell receptor.
Samuel M. Behar has included themes like Immunogenicity, Immunity and Virology in his Disease study.
The study incorporates disciplines such as Immunological memory, Lung, CD8 and Microbiology in addition to T cell.
The concepts of his Microbiology study are interwoven with issues in Cell culture, Antigen-presenting cell, Granulocyte macrophage colony-stimulating factor, Epitope and Macrophage.
His Immune system research incorporates themes from Cytotoxic T cell, Antibody and Antigen.
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