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Genetics

D-Index
107
Citations
52850
World Ranking
576
National Ranking
288

Medicine

D-Index
111
Citations
56432
World Ranking
5264
National Ranking
2840

Overview

Mike Hutton is primarily affiliated with Eli Lilly in the United States. Their research spans multiple fields including medicine and environmental science, with a focus on subfields such as surgery, ecology, genetics, ecological modeling, and radiology, nuclear medicine, and imaging.

The scientist has contributed to topics including:

  • Environmental DNA in Biodiversity Studies
  • Genetic diversity and population structure
  • Species Distribution and Climate Change
  • Surgical site infection prevention
  • Orthopedic Infections and Treatments
  • Total Knee Arthroplasty Outcomes
  • Radiomics and Machine Learning in Medical Imaging

Mike Hutton's recent published papers include:

  • Building meaningful collaboration in conservation genetics and genomics, 2024, Conservation Genetics
  • Getting it right first time: national survey of surgical site infection 2019, 2022, Annals of The Royal College of Surgeons of England
  • PH-0531: Assessment of geometric distortion of eleven clinical MRI scanners across the UK, 2020, Radiotherapy and Oncology
  • Indigenous and cross-cultural wildlife research in Australia: editorial, 2025, Wildlife Research

Frequent co-authors working with Mike Hutton include:

  • Hannah B. Cliff
  • Emilie Ens
  • Robyn E. Shaw
  • Brittany Brockett
  • Jennifer C. Pierson

Mike Hutton's publications have appeared in venues such as:

  • Conservation Genetics
  • Annals of The Royal College of Surgeons of England
  • Radiotherapy and Oncology
  • Wildlife Research

Best Publications

  • Association of missense and 5′-splice-site mutations in tau with the inherited dementia FTDP-17

    M. Hutton;C. L. Lendon;P. Rizzu;M. Baker

  • Secreted amyloid beta-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease.

    D. Scheuner;C. Eckman;C. Eckman;M. Jensen;X. Song

  • Tau Suppression in a Neurodegenerative Mouse Model Improves Memory Function

    K. SantaCruz;J. Lewis;T. Spires;J. Paulson

  • Mutations in progranulin cause tau-negative frontotemporal dementia linked to chromosome 17

    Matt Baker;Ian R. Mackenzie;Stuart M. Pickering-Brown;Jennifer Gass

  • Increased amyloid-β42(43) in brains of mice expressing mutant presenilin 1

    Karen Duff;Karen Duff;Chris Eckman;Cindy Zehr;Cindy Zehr;Xin Yu

  • Enhanced neurofibrillary degeneration in transgenic mice expressing mutant tau and APP.

    Jada Lewis;Dennis W. Dickson;Wen-Lang Lin;Louise Chisholm

  • Neurofibrillary tangles, amyotrophy and progressive motor disturbance in mice expressing mutant (P301L) tau protein.

    Jada Lewis;Eileen McGowan;Julia Rockwood;Heather Melrose

  • Tauopathy in Drosophila: Neurodegeneration Without Neurofibrillary Tangles

    Curtis W. Wittmann;Matthew F. Wszolek;Joshua M. Shulman;Paul M. Salvaterra

  • Association of an extended haplotype in the tau gene with progressive supranuclear palsy.

    Matt Baker;Irene Litvan;Henry Houlden;Jennifer Adamson

  • TDP‐43 immunoreactivity in hippocampal sclerosis and Alzheimer's disease

    Catalina Amador-Ortiz;Wen Lang Lin;Zeshan Ahmed;David Personett

  • CHIP and Hsp70 regulate tau ubiquitination, degradation and aggregation

    Leonard Petrucelli;Dennis Dickson;Kathryn Kehoe;Julie Taylor

  • The high-affinity HSP90-CHIP complex recognizes and selectively degrades phosphorylated tau client proteins

    Chad A. Dickey;Chad A. Dickey;Adeela Kamal;Karen Lundgren;Natalia Klosak

  • Age-Dependent Neurofibrillary Tangle Formation, Neuron Loss, and Memory Impairment in a Mouse Model of Human Tauopathy (P301L)

    Martin Ramsden;Linda Kotilinek;Colleen Forster;Jennifer Paulson

  • Aβ42 Is Essential for Parenchymal and Vascular Amyloid Deposition in Mice

    Eileen McGowan;Fiona Pickford;Jungsu Kim;Luisa Onstead

  • Mutations in progranulin are a major cause of ubiquitin-positive frontotemporal lobar degeneration

    Jennifer Gass;Ashley Cannon;Ian R. Mackenzie;Bradley Boeve

  • Wild-Type Human TDP-43 Expression Causes TDP-43 Phosphorylation, Mitochondrial Aggregation, Motor Deficits, and Early Mortality in Transgenic Mice

    Ya Fei Xu;Tania F. Gendron;Yong Jie Zhang;Wen Lang Lin

  • GAB2 Alleles Modify Alzheimer's Risk in APOE ε4 Carriers

    Eric M. Reiman;Jennifer A. Webster;Amanda J. Myers;Amanda J. Myers;John Hardy;John Hardy

  • Accumulation of Pathological Tau Species and Memory Loss in a Conditional Model of Tauopathy

    Zdenek Berger;Hanno Roder;Amanda Hanna;Aaron Carlson

  • The Presenilins and Alzheimer's Disease

    Michael Hutton;John Hardy

  • Estimation of the Genetic Contribution of Presenilin-1 and -2 Mutations in a Population-Based Study of Presenile Alzheimer Disease

    Marc Cruts;Cornelia M. van Duijn;Hubert Backhovens;Marleen Van den Broeck

Frequent Co-Authors

John Hardy
John Hardy University College London
Jordi Pérez-Tur
Jordi Pérez-Tur Spanish National Research Council
Rosa Rademakers
Rosa Rademakers University of Antwerp
Bradley F. Boeve
Bradley F. Boeve Mayo Clinic
Ronald C. Petersen
Ronald C. Petersen University of Pennsylvania
Ryan J. Uitti
Ryan J. Uitti Mayo Clinic
Matthew J. Farrer
Matthew J. Farrer University of Florida

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