What is he best known for?
The fields of study he is best known for:
- Gene
- Immune system
- Internal medicine
His main research concerns Molecular biology, Apoptosis, Fas ligand, Cytotoxic T cell and Immunology.
His Molecular biology research integrates issues from Interleukin 2, Downregulation and upregulation, Interferon gamma and Cell biology.
His Phagocyte study in the realm of Cell biology interacts with subjects such as Phagoptosis.
His study in Apoptosis is interdisciplinary in nature, drawing from both Cancer research, Transcription factor, Kinase and Phosphorylation.
He has included themes like Tumor necrosis factor alpha, Fas receptor, Antibody, Monoclonal antibody and Aldesleukin in his Fas ligand study.
As a member of one scientific family, Masato Tanaka mostly works in the field of Immunology, focusing on Macrophage and, on occasion, Polycythemia vera, Hemolytic anemia, Anemia, Homeostasis and Erythropoiesis.
His most cited work include:
- Generalized lymphoproliferative disease in mice, caused by a point mutation in the fas ligand (1432 citations)
- Tissue-Resident Macrophages Self-Maintain Locally throughout Adult Life with Minimal Contribution from Circulating Monocytes (1207 citations)
- Identification of a factor that links apoptotic cells to phagocytes (1061 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Apoptosis, Cell biology, Immunology, Molecular biology and Fas ligand.
His Apoptosis research incorporates themes from In vitro, Phagocytosis and Autoimmune disease.
Masato Tanaka combines subjects such as Epidermal growth factor, Gene, Bone marrow and Apoptotic cell clearance with his study of Cell biology.
His Immunology research focuses on Macrophage and how it connects with Colitis.
While the research belongs to areas of Molecular biology, he spends his time largely on the problem of Downregulation and upregulation, intersecting his research to questions surrounding Jurkat cells.
His study in Fas ligand is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha, Cytotoxic T cell, Metalloproteinase and Fas receptor.
He most often published in these fields:
- Apoptosis (33.33%)
- Cell biology (28.47%)
- Immunology (26.39%)
What were the highlights of his more recent work (between 2013-2021)?
- Cell biology (28.47%)
- Immunology (26.39%)
- Macrophage (11.81%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in Cell biology, Immunology, Macrophage, Inflammation and Immune system.
His Cell biology study integrates concerns from other disciplines, such as Cellular differentiation and Programmed cell death.
His research integrates issues of Cancer research, Pathogenesis and Hepatocyte in his study of Macrophage.
He has researched Immune system in several fields, including Apoptosis and Gastrointestinal tract.
His Apoptosis research is multidisciplinary, incorporating elements of Cancer cell, In vitro and MEDLINE.
Masato Tanaka performs integrative study on Ubiquitin binding and Molecular biology in his works.
Between 2013 and 2021, his most popular works were:
- Dectin-2 Is a Direct Receptor for Mannose-Capped Lipoarabinomannan of Mycobacteria (152 citations)
- Programmed cell death and the immune system. (151 citations)
- Intestinal CD169+ macrophages initiate mucosal inflammation by secreting CCL8 that recruits inflammatory monocytes (96 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Immune system
- Internal medicine
Cell biology, Immunology, Immune system, Innate immune system and Inflammation are his primary areas of study.
The concepts of his Cell biology study are interwoven with issues in Spleen, Stromal cell, Lymphatic system and Lymph node.
His work in the fields of Antigen, Colitis and Pattern recognition receptor overlaps with other areas such as Adipose tissue macrophages.
His research on Immune system often connects related areas such as Apoptosis.
His Apoptosis study frequently links to adjacent areas such as Chemotaxis.
His Inflammation research is multidisciplinary, relying on both Fibrosis, Monoblast and Hepatology.
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