His main research concerns Molecular biology, Apoptosis, Fas ligand, Cytotoxic T cell and Immunology. His Molecular biology research integrates issues from Interleukin 2, Downregulation and upregulation, Interferon gamma and Cell biology. His Phagocyte study in the realm of Cell biology interacts with subjects such as Phagoptosis.
His study in Apoptosis is interdisciplinary in nature, drawing from both Cancer research, Transcription factor, Kinase and Phosphorylation. He has included themes like Tumor necrosis factor alpha, Fas receptor, Antibody, Monoclonal antibody and Aldesleukin in his Fas ligand study. As a member of one scientific family, Masato Tanaka mostly works in the field of Immunology, focusing on Macrophage and, on occasion, Polycythemia vera, Hemolytic anemia, Anemia, Homeostasis and Erythropoiesis.
His scientific interests lie mostly in Apoptosis, Cell biology, Immunology, Molecular biology and Fas ligand. His Apoptosis research incorporates themes from In vitro, Phagocytosis and Autoimmune disease. Masato Tanaka combines subjects such as Epidermal growth factor, Gene, Bone marrow and Apoptotic cell clearance with his study of Cell biology.
His Immunology research focuses on Macrophage and how it connects with Colitis. While the research belongs to areas of Molecular biology, he spends his time largely on the problem of Downregulation and upregulation, intersecting his research to questions surrounding Jurkat cells. His study in Fas ligand is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha, Cytotoxic T cell, Metalloproteinase and Fas receptor.
His primary scientific interests are in Cell biology, Immunology, Macrophage, Inflammation and Immune system. His Cell biology study integrates concerns from other disciplines, such as Cellular differentiation and Programmed cell death. His research integrates issues of Cancer research, Pathogenesis and Hepatocyte in his study of Macrophage.
He has researched Immune system in several fields, including Apoptosis and Gastrointestinal tract. His Apoptosis research is multidisciplinary, incorporating elements of Cancer cell, In vitro and MEDLINE. Masato Tanaka performs integrative study on Ubiquitin binding and Molecular biology in his works.
Cell biology, Immunology, Immune system, Innate immune system and Inflammation are his primary areas of study. The concepts of his Cell biology study are interwoven with issues in Spleen, Stromal cell, Lymphatic system and Lymph node. His work in the fields of Antigen, Colitis and Pattern recognition receptor overlaps with other areas such as Adipose tissue macrophages.
His research on Immune system often connects related areas such as Apoptosis. His Apoptosis study frequently links to adjacent areas such as Chemotaxis. His Inflammation research is multidisciplinary, relying on both Fibrosis, Monoblast and Hepatology.
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Generalized lymphoproliferative disease in mice, caused by a point mutation in the fas ligand
Tomohiro Takahashi;Masato Tanaka;Camllynn I. Brannan;Nancy A. Jenkins.
Tissue-Resident Macrophages Self-Maintain Locally throughout Adult Life with Minimal Contribution from Circulating Monocytes
Daigo Hashimoto;Andrew Chow;Andrew Chow;Clara Noizat;Clara Noizat;Pearline Teo.
Identification of a factor that links apoptotic cells to phagocytes
Rikinari Hanayama;Masato Tanaka;Keiko Miwa;Azusa Shinohara.
Fas ligand in human serum
Masato Tanaka;Masato Tanaka;Takashi Suda;Kyosuke Haze;Norio Nakamura.
Nature Medicine (1996)
Downregulation of Fas ligand by shedding
Masato Tanaka;Masato Tanaka;Toshimitsu Itai;Masashi Adachi;Shigekazu Nagata;Shigekazu Nagata.
Nature Medicine (1998)
Autoimmune disease and impaired uptake of apoptotic cells in MFG-E8-deficient mice.
Rikinari Hanayama;Masato Tanaka;Kay Miyasaka;Katsuyuki Aozasa.
Expression of the functional soluble form of human fas ligand in activated lymphocytes.
M. Tanaka;T. Suda;T. Takahashi;Shigekazu Nagata.
The EMBO Journal (1995)
Embryonic Lethality, Liver Degeneration, and Impaired NF-κB Activation in IKK-β-Deficient Mice
Masato Tanaka;Maria Elena Fuentes;Kyoko Yamaguchi;Michael H Durnin.
Endothelin-1 is an autocrine/paracrine factor in the mechanism of angiotensin II-induced hypertrophy in cultured rat cardiomyocytes.
H Ito;Y Hirata;S Adachi;M Tanaka.
Journal of Clinical Investigation (1993)
Hypoxia induces apoptosis with enhanced expression of Fas antigen messenger RNA in cultured neonatal rat cardiomyocytes.
M Tanaka;H Ito;S Adachi;H Akimoto.
Circulation Research (1994)
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