The scientist’s investigation covers issues in Hyperalgesia, Pharmacology, Anesthesia, Nociception and Internal medicine. His Hyperalgesia research includes elements of Protein kinase A, Prostaglandin E2, Nociceptor, Prostaglandin E and Protein kinase C. His Pharmacology study combines topics from a wide range of disciplines, such as Inflammation, Agonist, Biochemistry, -Naloxone and Transient receptor potential channel.
His research integrates issues of Peripheral neuropathy and Dose–response relationship in his study of Anesthesia. The concepts of his Nociception study are interwoven with issues in Tonicity, Reflex, TRPV4 and Central nervous system. His Internal medicine study frequently draws parallels with other fields, such as Endocrinology.
Jon D. Levine focuses on Internal medicine, Hyperalgesia, Endocrinology, Anesthesia and Nociceptor. In general Internal medicine study, his work on Chemotherapy, Cancer and Breast cancer often relates to the realm of Context, thereby connecting several areas of interest. His Hyperalgesia research incorporates themes from Prostaglandin E2, Pharmacology and Prostaglandin E.
His Endocrinology study which covers Protein kinase A that intersects with Second messenger system and Protein kinase C. His Anesthesia study integrates concerns from other disciplines, such as Peripheral neuropathy and Placebo. His Nociceptor research integrates issues from Sensitization, Stimulation, Neuroscience, Chronic pain and Priming.
His primary areas of study are Internal medicine, Chemotherapy, Cancer, Hyperalgesia and Breast cancer. Jon D. Levine has researched Internal medicine in several fields, including Endocrinology and Sleep disorder. His Endocrinology research is multidisciplinary, relying on both Receptor, Glucocorticoid receptor, Estrogen receptor and Gene knockdown.
His work deals with themes such as DAMGO, Inflammation, TLR4, Prostaglandin E2 and Nociceptor, which intersect with Hyperalgesia. His Nociceptor study is concerned with the larger field of Nociception. His studies in Breast cancer integrate themes in fields like Peripheral neuropathy, Menopause, Surgery and Candidate gene.
Internal medicine, Hyperalgesia, Nociceptor, Context and Comorbidity are his primary areas of study. The various areas that Jon D. Levine examines in his Internal medicine study include Peripheral neuropathy and Physical therapy. His Hyperalgesia study deals with the bigger picture of Nociception.
His Nociception study combines topics in areas such as Signal transduction and Neuroscience. The study incorporates disciplines such as Anesthesia and Endocrinology in addition to Nociceptor. Within one scientific family, Jon D. Levine focuses on topics pertaining to Skeletal muscle under Anesthesia, and may sometimes address concerns connected to Receptor.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The capsaicin receptor: a heat-activated ion channel in the pain pathway
Michael J. Caterina;Mark A. Schumacher;Makoto Tominaga;Tobias A. Rosen.
Nature (1997)
Hyperalgesic agents increase a tetrodotoxin-resistant Na+ current in nociceptors
Michael S. Gold;David B. Reichling;Michael J. Shuster;Jon D. Levine.
Proceedings of the National Academy of Sciences of the United States of America (1996)
Expression of c‐fos protein in interneurons and projection neurons of the rat spinal cord in response to noxious somatic, articular, and visceral stimulation
D. Menétrey;A. Gannon;J. D. Levine;Allan I. Basbaum.
The Journal of Comparative Neurology (1989)
Intraneuronal substance P contributes to the severity of experimental arthritis
Jon D. Levine;Ron Clark;Marshall Devor;Clyde Helms.
Science (1984)
Systemic morphine suppresses noxious stimulus-evoked Fos protein-like immunoreactivity in the rat spinal cord.
RW Presley;D Menetrey;JD Levine;AI Basbaum.
The Journal of Neuroscience (1990)
Peptides and the primary afferent nociceptor
Jon D. Levine;Howard L. Fields;Allan I. Basbaum.
The Journal of Neuroscience (1993)
Kappa–opioids produce significantly greater analgesia in women than in men
Robert W. Gear;Christine Miaskowski;Newton C. Gordon;Steven M. Paul.
Nature Medicine (1996)
Modulation of TTX-R INa by PKC and PKA and Their Role in PGE2-Induced Sensitization of Rat Sensory Neurons In Vitro
Michael S. Gold;Jon D. Levine;Ana M. Correa.
The Journal of Neuroscience (1998)
A novel nociceptor signaling pathway revealed in protein kinase c ε mutant mice
Sachia G. Khasar;Yu Huei Lin;Annick Martin;Jahan Dadgar.
Neuron (1999)
Contribution of sensory afferents and sympathetic efferents to joint injury in experimental arthritis.
JD Levine;SJ Dardick;MF Roizen;C Helms.
The Journal of Neuroscience (1986)
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