D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 104 Citations 40,863 246 World Ranking 389 National Ranking 237
Medicine D-index 104 Citations 40,931 253 World Ranking 4166 National Ranking 2360

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Neuron
  • Internal medicine

His primary scientific interests are in Neuroscience, Microglia, Neuropathic pain, Spinal cord and Chronic pain. His work carried out in the field of Neuroscience brings together such families of science as Hyperalgesia, Nociceptor, Neurotransmission and MAPK/ERK pathway. His Microglia study combines topics in areas such as Proinflammatory cytokine, Central nervous system, Neuroglia and Pathology.

His research in Neuropathic pain intersects with topics in Mitogen-activated protein kinase, Signal transduction, Astrocyte and Nervous system. His study in Spinal cord is interdisciplinary in nature, drawing from both c-Fos, Downregulation and upregulation and Nociception. Ru-Rong Ji combines subjects such as Neuralgia, Neuroinflammation and Immunology with his study of Chronic pain.

His most cited work include:

  • Central sensitization and LTP: do pain and memory share similar mechanisms? (1127 citations)
  • p38 MAPK activation by NGF in primary sensory neurons after inflammation increases TRPV1 levels and maintains heat hyperalgesia. (1011 citations)
  • Cytokine Mechanisms of Central Sensitization: Distinct and Overlapping Role of Interleukin-1β, Interleukin-6, and Tumor Necrosis Factor-α in Regulating Synaptic and Neuronal Activity in the Superficial Spinal Cord (758 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Neuroscience, Neuropathic pain, Spinal cord, Pharmacology and Chronic pain. His biological study spans a wide range of topics, including Hyperalgesia, Nociceptor and Nociception. His Neuropathic pain research is multidisciplinary, incorporating perspectives in Immunology, Nerve injury, Microglia and Neurotransmission.

His Spinal cord study integrates concerns from other disciplines, such as Endocrinology, Excitatory postsynaptic potential, Long-term potentiation, Internal medicine and Spinal Cord Dorsal Horn. Ru-Rong Ji has included themes like Inflammation, Lipid signaling, TRPV1 and Opioid in his Pharmacology study. His Chronic pain study incorporates themes from Neuroinflammation, Sensory system and Pathology.

He most often published in these fields:

  • Neuroscience (46.00%)
  • Neuropathic pain (42.80%)
  • Spinal cord (32.00%)

What were the highlights of his more recent work (between 2017-2021)?

  • Neuroscience (46.00%)
  • Chronic pain (24.00%)
  • Pharmacology (25.20%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Neuroscience, Chronic pain, Pharmacology, Neuropathic pain and Spinal cord. His Neuroscience research incorporates elements of Microglia and Nociception. His studies in Chronic pain integrate themes in fields like Neuroinflammation, MEDLINE, Astrocyte and Pathogenesis.

His Pharmacology research is multidisciplinary, relying on both Tumor necrosis factor alpha, In vitro, TRPV1 and Opioid receptor, Opioid. The various areas that Ru-Rong Ji examines in his Neuropathic pain study include Inflammation, Peripheral neuropathy and Neurotransmission. Ru-Rong Ji has researched Spinal cord in several fields, including Long-term potentiation and Excitatory postsynaptic potential.

Between 2017 and 2021, his most popular works were:

  • Neuroinflammation and Central Sensitization in Chronic and Widespread Pain (202 citations)
  • Microglia in Pain: Detrimental and Protective Roles in Pathogenesis and Resolution of Pain. (153 citations)
  • Sex-Dependent Glial Signaling in Pathological Pain: Distinct Roles of Spinal Microglia and Astrocytes. (77 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Neuron
  • Internal medicine

Neuropathic pain, Neuroscience, Chronic pain, Inflammation and Pharmacology are his primary areas of study. His Neuropathic pain research integrates issues from Peripheral neuropathy, TRPV1 and Monoclonal antibody. His Neuroscience study combines topics from a wide range of disciplines, such as Synaptic plasticity, Neuroinflammation, Hyperalgesia and Microglia.

His work deals with themes such as Disease progression, Neurotransmission, Pathogenesis and Intensive care medicine, which intersect with Chronic pain. He interconnects Analgesic and Nociceptor in the investigation of issues within Inflammation. His research investigates the connection with Pharmacology and areas like Spinal cord which intersect with concerns in Nerve injury.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Central sensitization and LTP: do pain and memory share similar mechanisms?

Ru-Rong Ji;Tatsuro Kohno;Kimberly A Moore;Clifford J Woolf.
Trends in Neurosciences (2003)

1677 Citations

p38 MAPK activation by NGF in primary sensory neurons after inflammation increases TRPV1 levels and maintains heat hyperalgesia.

Ru-Rong Ji;Tarek A. Samad;Shan-Xue Jin;Raymond Schmoll.
Neuron (2002)

1384 Citations

Cytokine Mechanisms of Central Sensitization: Distinct and Overlapping Role of Interleukin-1β, Interleukin-6, and Tumor Necrosis Factor-α in Regulating Synaptic and Neuronal Activity in the Superficial Spinal Cord

Yasuhiko Kawasaki;Ling Zhang;Jen-Kun Cheng;Ru-Rong Ji.
The Journal of Neuroscience (2008)

1331 Citations

p38 mitogen-activated protein kinase is activated after a spinal nerve ligation in spinal cord microglia and dorsal root ganglion neurons and contributes to the generation of neuropathic pain

Shan-Xue Jin;Zhi-Ye Zhuang;Clifford J. Woolf;Ru-Rong Ji.
The Journal of Neuroscience (2003)

1155 Citations

MAP kinase and pain

Ru-Rong Ji;Robert W. Gereau;Marzia Malcangio;Gary R. Strichartz.
Brain Research Reviews (2009)

1057 Citations

Glia and pain: Is chronic pain a gliopathy?

Ru-Rong Ji;Temugin Berta.
Pain (2013)

964 Citations

Different immune cells mediate mechanical pain hypersensitivity in male and female mice

Robert E. Sorge;Josiane C.S. Mapplebeck;Sarah Rosen;Simon Beggs.
Nature Neuroscience (2015)

933 Citations

Neuronal plasticity and signal transduction in nociceptive neurons: implications for the initiation and maintenance of pathological pain.

Ru-Rong Ji;Clifford J. Woolf.
Neurobiology of Disease (2001)

884 Citations

Nociceptive-specific activation of ERK in spinal neurons contributes to pain hypersensitivity.

Ru-Rong Ji;Hiroshi Baba;Gary J. Brenner;Clifford J. Woolf.
Nature Neuroscience (1999)

883 Citations

ERK is sequentially activated in neurons, microglia, and astrocytes by spinal nerve ligation and contributes to mechanical allodynia in this neuropathic pain model.

Zhi Ye Zhuang;Peter Gerner;Clifford J. Woolf;Ru-Rong Ji.
Pain (2005)

816 Citations

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