What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His main research concerns Neuroscience, Microglia, Cell biology, Neuropathic pain and Receptor.
The various areas that he examines in his Neuroscience study include Glutamate receptor, Hyperalgesia, PPADS and Synaptic plasticity.
His Microglia study incorporates themes from Purinergic receptor, Signal transduction, MAPK/ERK pathway and Neuroglia.
His studies in Cell biology integrate themes in fields like Endocrinology, Biochemistry and Chemotaxis.
His research integrates issues of Pathological, Chronic pain and Nerve injury in his study of Neuropathic pain.
His work in Receptor tackles topics such as Pharmacology which are related to areas like Agonist and Anesthesia.
His most cited work include:
- BDNF from microglia causes the shift in neuronal anion gradient underlying neuropathic pain (1412 citations)
- P2x4 receptors induced in spinal microglia gate tactile allodynia after nerve injury (1187 citations)
- Neuropathic pain and spinal microglia: a big problem from molecules in "small" glia. (651 citations)
What are the main themes of his work throughout his whole career to date?
Kazuhide Inoue mostly deals with Neuroscience, Receptor, Microglia, Cell biology and Neuropathic pain.
His work deals with themes such as Glutamate receptor and Neurotransmission, which intersect with Neuroscience.
Kazuhide Inoue has researched Receptor in several fields, including Biophysics, Endocrinology and Extracellular.
His study focuses on the intersection of Microglia and fields such as Purinergic receptor with connections in the field of Purinergic signalling.
Cell biology is frequently linked to Chemotaxis in his study.
His Neuropathic pain study combines topics from a wide range of disciplines, such as Chronic pain, Nerve injury and Allodynia.
He most often published in these fields:
- Neuroscience (27.48%)
- Receptor (25.87%)
- Microglia (23.79%)
What were the highlights of his more recent work (between 2013-2021)?
- Neuropathic pain (20.09%)
- Microglia (23.79%)
- Neuroscience (27.48%)
In recent papers he was focusing on the following fields of study:
Kazuhide Inoue mainly investigates Neuropathic pain, Microglia, Neuroscience, Pharmacology and Receptor.
His biological study spans a wide range of topics, including Nerve injury, Allodynia, Chronic pain and Nociception.
His Microglia research is multidisciplinary, incorporating perspectives in Purinergic receptor, Cell biology, Downregulation and upregulation and Spinal cord.
His studies deal with areas such as Extracellular and Endocrinology, Stimulation, Internal medicine as well as Spinal cord.
Kazuhide Inoue interconnects CCR2 and Dorsum in the investigation of issues within Neuroscience.
His Receptor research is multidisciplinary, relying on both Bioinformatics, Biophysics, Molecular biology, Multiple sclerosis and Binding site.
Between 2013 and 2021, his most popular works were:
- Microglia in neuropathic pain: cellular and molecular mechanisms and therapeutic potential (196 citations)
- Transcription factor IRF5 drives P2X4R+-reactive microglia gating neuropathic pain. (107 citations)
- STAT3-dependent reactive astrogliosis in the spinal dorsal horn underlies chronic itch (93 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
His primary scientific interests are in Microglia, Neuropathic pain, Peripheral nerve injury, Chronic pain and Pharmacology.
His Microglia research incorporates themes from Purinergic receptor, Spinal cord, IRF8 and Nociception.
To a larger extent, Kazuhide Inoue studies Neuroscience with the aim of understanding Neuropathic pain.
The various areas that he examines in his Neuroscience study include P2Y receptor, Purinergic signalling and Homeostasis.
His Chronic pain research integrates issues from Defence system, Spinal Cord Dorsal Horn and Nervous system.
His work carried out in the field of Pharmacology brings together such families of science as Hot plate test, CCL3, Recombinant Chemokine and Hyperalgesia, Allodynia.
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