Theodore J. Price focuses on Neuroscience, Allodynia, Endocrinology, Internal medicine and Hyperalgesia. His work carried out in the field of Neuroscience brings together such families of science as Nociceptor and Nociception. The concepts of his Nociceptor study are interwoven with issues in Memory consolidation and Nervous system.
His Allodynia research incorporates elements of Neuropathic pain and Pharmacology. His Endocrinology research is multidisciplinary, incorporating elements of Trigeminal ganglion, Calcitonin gene-related peptide and TRPV1. As a part of the same scientific study, Theodore J. Price usually deals with the Hyperalgesia, concentrating on AMPK and frequently concerns with MAPK/ERK pathway, PI3K/AKT/mTOR pathway and Resveratrol.
Theodore J. Price mostly deals with Neuroscience, Nociceptor, Chronic pain, Pharmacology and Dorsal root ganglion. Many of his studies on Neuroscience involve topics that are commonly interrelated, such as Nociception. Theodore J. Price combines subjects such as Endocrinology, Signal transduction and Nervous system with his study of Nociceptor.
His Endocrinology study which covers Calcitonin gene-related peptide that intersects with TRPV1 and Trigeminal ganglion. The various areas that Theodore J. Price examines in his Chronic pain study include Neuropathic pain, Disease and Bioinformatics. The study incorporates disciplines such as Metformin, AMPK, Hyperalgesia and MAPK/ERK pathway in addition to Pharmacology.
Theodore J. Price spends much of his time researching Dorsal root ganglion, Nociceptor, Neuroscience, Receptor and Sensory system. His work carried out in the field of Dorsal root ganglion brings together such families of science as Nociception, Transcriptome, Nervous system, Cell biology and Cell type. His Nociceptor study is concerned with the field of Internal medicine as a whole.
His studies deal with areas such as Downregulation and upregulation and Chronic pain as well as Internal medicine. His Neuroscience research includes elements of Cell and Disease. In the field of Receptor, his study on G protein-coupled receptor overlaps with subjects such as Population.
Theodore J. Price mainly focuses on Dorsal root ganglion, Nociceptor, Receptor, Sensory system and Neuroscience. His Nociceptor study integrates concerns from other disciplines, such as Glycolipid, Cell signaling, Signal transduction and Tuberculosis. His Receptor study incorporates themes from Neuropathic pain and Downregulation and upregulation.
His work in Neuroscience addresses subjects such as Interactome, which are connected to disciplines such as Growth factor. His research investigates the connection with Cell biology and areas like Transient receptor potential channel which intersect with concerns in Calcitonin gene-related peptide. The Nociception study combines topics in areas such as Endocrinology, Pharmacology, HMGB1 and TLR4.
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Cation-chloride cotransporters in neuronal development, plasticity and disease
Kai Kaila;Theodore J. Price;John A. Payne;Martin Puskarjov.
Nature Reviews Neuroscience (2014)
Chloride Regulation in the Pain Pathway
Theodore J. Price;Fernando Cervero;Michael S. Gold;Donna L. Hammond.
Brain Research Reviews (2009)
Critical evaluation of the colocalization between calcitonin gene-related peptide, substance P, transient receptor potential vanilloid subfamily type 1 immunoreactivities and isolectin B4 binding in primary afferent neurons of the rat and mouse
Theodore J. Price;Theodore J. Price;Christopher M. Flores.
The Journal of Pain (2007)
Role of cation-chloride-cotransporters (CCC) in pain and hyperalgesia.
Theodore J. Price;Fernando Cervero;Yves de Koninck.
Current Topics in Medicinal Chemistry (2005)
3D shape and 2D surface textures of human faces: the role of “averages” in attractiveness and age
Alice J. O'Toole;Theodore J. Price;Thomas Vetter;James C. Bartlett.
Image and Vision Computing (1999)
Engagement of descending inhibition from the rostral ventromedial medulla protects against chronic neuropathic pain
Milena De Felice;Raul Sanoja;Ruizhong Wang;Louis Vera-Portocarrero.
Targeting adenosine monophosphate-activated protein kinase (AMPK) in preclinical models reveals a potential mechanism for the treatment of neuropathic pain
Ohannes K Melemedjian;Marina N Asiedu;Dipti V Tillu;Raul Sanoja.
Molecular Pain (2011)
Decreased Nociceptive Sensitization in Mice Lacking the Fragile X Mental Retardation Protein: Role of mGluR1/5 and mTOR
Theodore J. Price;Harunor Rashid;Magali Millecamps;Raul Sanoja.
The Journal of Neuroscience (2007)
The cannabinoid WIN 55,212-2 inhibits transient receptor potential vanilloid 1 (TRPV1) and evokes peripheral antihyperalgesia via calcineurin
Amol M. Patwardhan;Nathaniel A. Jeske;Theodore J. Price;Nikita Gamper.
Proceedings of the National Academy of Sciences of the United States of America (2006)
IL-6- and NGF-induced rapid control of protein synthesis and nociceptive plasticity via convergent signaling to the eIF4F complex.
Ohannes K. Melemedjian;Marina N. Asiedu;Dipti V. Tillu;Katherine A. Peebles.
The Journal of Neuroscience (2010)
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